Academic Journal
Subacute combined degeneration due to nitrous oxide-induced vitamin B-12 deficiency
| العنوان: | Subacute combined degeneration due to nitrous oxide-induced vitamin B-12 deficiency |
|---|---|
| المؤلفون: | Nadal, Jorge, Hernandez, Daniela, Cantazaro, Brandon |
| المصدر: | Research Colloquium |
| بيانات النشر: | ScholarWorks @ UTRGV |
| سنة النشر: | 2023 |
| مصطلحات موضوعية: | Subacute combined degeneration, nitrous oxide recreational use, vitamin B-12 deficiency, Medicine and Health Sciences, Nervous System Diseases, Pathological Conditions, Signs and Symptoms, Substance Abuse and Addiction |
| الوصف: | Background: Nitrous oxide also known as “laughing gas†is a naturally occurring gas that is colorless, odorless, nonflammable, and nontoxic. It has been used as an inhalant anesthetic in the medical field for more than 150 years for dental and surgical procedures. Due to its wide availability and ability to cause euphoria, recreational use is on the rise.1 Here, I will present a case of subacute combined degeneration in the setting of nitrous oxide induced vitamin B-12 deficiency. Case presentation: 23-year-old right-handed gentleman with no past medical history presented to the ED complaining of 1 week of bilateral lower extremity symmetric paresthesia, weakness, and difficulty with ambulation. He initially experienced paresthesia in the lower extremities. Two days later, he experienced difficulty ambulating which led to multiple falls at home. The patient stated sensation in both legs was reduced in addition to experiencing difficulty with positioning during movement and balance difficulty. Strength in bilateral lower extremities was also markedly reduced as he eventually noticed he was unable to stand by himself and after his most recent fall which prompted his visit to the emergency department. The patient denied trauma, fever, chills, fatigue, weight loss, nausea, vomiting, diarrhea, dizziness, vision changes. He denied alcohol use, tobacco use, or drug use. On neurological examination, the patient was alert and oriented to place, time, and situation, normal 5-minute recollection, normal mood, no aphasia, cranial nerves II-XII intact. Motor strength was 4/5 hip flexion bilaterally with eyes open, decreased to 3/5 with eyes closed. Dorsiflexion 4/5 bilaterally with eyes open, decreased to 3/5 with eyes closed. Deep tendon reflexes were 2+ in bicep reflex bilaterally, 3+ in patellar reflex with cross adductor response bilaterally, 3+ in achilles reflex. No clonus. Absent Babinski. On sensory exam, there was profound impairment in proprioception and vibration in bilateral lower extremities and decreased ... |
| نوع الوثيقة: | text |
| وصف الملف: | application/pdf |
| اللغة: | unknown |
| Relation: | https://scholarworks.utrgv.edu/colloquium/2022/posters/20; https://scholarworks.utrgv.edu/context/colloquium/article/1103/viewcontent/SCD_Case_Report_.pdf |
| الاتاحة: | https://scholarworks.utrgv.edu/colloquium/2022/posters/20 https://scholarworks.utrgv.edu/context/colloquium/article/1103/viewcontent/SCD_Case_Report_.pdf |
| رقم الانضمام: | edsbas.4FC61CF6 |
| قاعدة البيانات: | BASE |
| الوصف غير متاح. |