V. dahliae VOS1 function is independently of microsclerotia development.

التفاصيل البيبلوغرافية
العنوان: V. dahliae VOS1 function is independently of microsclerotia development.
المؤلفون: Annalena M. Höfer (10305727), Rebekka Harting (9213632), Nils F. Aßmann (10305730), Jennifer Gerke (236089), Kerstin Schmitt (474567), Jessica Starke (4728282), Özgür Bayram (150583), Van-Tuan Tran (6706739), Oliver Valerius (150607), Susanna A. Braus-Stromeyer (9213635), Gerhard H. Braus (150631)
سنة النشر: 2021
المجموعة: Smithsonian Institution: Digital Repository
مصطلحات موضوعية: Biochemistry, Microbiology, Cell Biology, Genetics, Molecular Biology, Ecology, Developmental Biology, Cancer, Plant Biology, Biological Sciences not elsewhere classified, Vel 1-Vel Vel 2-Vos, plant root colonization, fungus Verticillium dahliae, Vel 3 control, dahliae velvet proteins, Vel 3-Vos heterodimers, Rel homology domain, disease symptom induction, plant pathogenicity experiments, life cycle, proteins link development, DNA, NF -κB transcription factor, velvet protein Vel 1 controls, velvet domain encoding genes, Vel 1
الوصف: Images give an overview of fungal colonies after spotting 5x10 4 spores of indicated V . dahliae strains on either simulated xylem medium (SXM) or Czapek-Dox-Medium (CDM) and subsequent incubation for 10 days at 25°C in constant light or darkness. Single colonies on SXM are shown from the back and colonies on CDM are shown from the top of the plate. Cross sections were made through the middle of the colony. (A) Growth and development of the VOS1 deletion (Δ VOS1 ) and the wild type (WT) strains are similar. Single colonies (left column) and colony cross sections (right column) are shown. (B) A VEL3 and VOS1 double deletion strain (Δ VEL3 /Δ VOS1 ) resembles the phenotype of the VEL3 (Δ VEL3 ) single deletion strain. (TIF)
نوع الوثيقة: still image
اللغة: unknown
Relation: https://figshare.com/articles/figure/_i_V_dahliae_VOS1_i_function_is_independently_of_microsclerotia_development_/14218888
DOI: 10.1371/journal.pgen.1009434.s008
الاتاحة: https://doi.org/10.1371/journal.pgen.1009434.s008
Rights: CC BY 4.0
رقم الانضمام: edsbas.25525583
قاعدة البيانات: BASE
الوصف
DOI:10.1371/journal.pgen.1009434.s008