Functional characterization of AATF transcriptome in human leukemic cells

التفاصيل البيبلوغرافية
العنوان: Functional characterization of AATF transcriptome in human leukemic cells
المؤلفون: Deepak Kaul, Aanchal Mehrotra
المصدر: Molecular and cellular biochemistry. 297(1-2)
سنة النشر: 2006
مصطلحات موضوعية: Programmed cell death, Transcription, Genetic, Cell Survival, Sp1 Transcription Factor, Clinical Biochemistry, Molecular Sequence Data, Apoptosis, HL-60 Cells, Biology, Response Elements, Jurkat cells, Models, Biological, Catechin, Transcriptome, Proto-Oncogene Proteins c-myc, Jurkat Cells, Downregulation and upregulation, Humans, Molecular Biology, Transcription factor, PI3K/AKT/mTOR pathway, Regulation of gene expression, Leukemia, Base Sequence, Cell growth, Cell Biology, General Medicine, Flow Cytometry, Cell biology, Androstadienes, Gene Expression Regulation, Neoplastic, Repressor Proteins, Cancer research, Apoptosis Regulatory Proteins, Wortmannin, Transcription Factors
الوصف: The study, addressed to explore the transcriptional expression and regulation of Apoptosis-antagonizing transcription factor (AATF) gene within various types of human leukemic cell lines, revealed that AATF gene was overexpressed ubiquitously in all the leukemic cell lines studied and this upregulation was accompanied by c-myc gene overamplification in these cells. Downregulation of AATF gene transcription within leukemic cells not only resulted in the downregulation of c-myc gene and vice-versa but also contributed to apoptosis leading to cell death. Further, the link between AATF expression and leukemic cellular apoptosis involved PI3K/Akt pathway. Based on these results we propose that AATF gene may be of crucial importance in maintaining the leukemic state of a cell compartment through its ability to initiate cell proliferation coupled with repression of cellular apoptosis.
تدمد: 0300-8177
URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::8e313848bbeb64294270e34e7faff3b1
https://pubmed.ncbi.nlm.nih.gov/17006618
Rights: CLOSED
رقم الانضمام: edsair.doi.dedup.....8e313848bbeb64294270e34e7faff3b1
قاعدة البيانات: OpenAIRE