Depression enhanced the production of autoantibodies against 16α‑hydroxyestrone-estrogen receptor adduct in breast cancer

التفاصيل البيبلوغرافية
العنوان: Depression enhanced the production of autoantibodies against 16α‑hydroxyestrone-estrogen receptor adduct in breast cancer
المؤلفون: Waseem Ahmad Siddiqui, Wahid Ali Khan, Subuhi Sherwani, Mohd Wajid Ali Khan
المصدر: International immunopharmacology. 66
سنة النشر: 2018
مصطلحات موضوعية: 0301 basic medicine, Oncology, medicine.medical_specialty, Hydroxyestrones, Immunology, Estrogen receptor, Autoimmunity, Breast Neoplasms, Proinflammatory cytokine, 03 medical and health sciences, 0302 clinical medicine, Breast cancer, Internal medicine, Immunology and Allergy, Medicine, Humans, Depression (differential diagnoses), Aged, Autoantibodies, Pharmacology, biology, business.industry, Depression, Interleukin-6, Tumor Necrosis Factor-alpha, Autoantibody, Cancer, Middle Aged, medicine.disease, Precipitin, 030104 developmental biology, Receptors, Estrogen, 030220 oncology & carcinogenesis, biology.protein, Female, Antibody, Inflammation Mediators, business, Protein Binding
الوصف: Mentally depressed breast cancer (MDBC) patients expressed estrogen receptor (ER) and 16α‑hydroxyestrone (16α‑OHE1) is directly responsible for causing breast cancer (BC). This study aimed to identify whether depression in breast cancer patients enhanced the production of autoantibodies against 16α‑OHE1-ER adduct in breast cancer patients. The antibodies in the serum of 65 breast cancer patients (including 35 MDBC) and 40 control subjects were screened by direct binding, inhibition enzyme-linked immunosorbent assay (ELISA), and quantitative precipitin titration. Competition ELISA was also utilized for the estimation of 16α‑OHE1 in the serum of 30 cancer patients. Autoantibodies from MDBC showed strong recognition to 16α‑OHE1-ER in comparison to overall breast cancer patients (p
تدمد: 1878-1705
URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::6add16d9edec3a7113a5b56c900cd6f5
https://pubmed.ncbi.nlm.nih.gov/30500622
Rights: CLOSED
رقم الانضمام: edsair.doi.dedup.....6add16d9edec3a7113a5b56c900cd6f5
قاعدة البيانات: OpenAIRE