QRS/T-wave and calcium alternans in a type I diabetic mouse model for spontaneous postmyocardial infarction ventricular tachycardia: A mechanism for the antiarrhythmic effect of statins

التفاصيل البيبلوغرافية
العنوان: QRS/T-wave and calcium alternans in a type I diabetic mouse model for spontaneous postmyocardial infarction ventricular tachycardia: A mechanism for the antiarrhythmic effect of statins
المؤلفون: Sami F. Noujaim, Bo Wang, Mohammad Rajab, Ho-Jin Park, Farzad Noubary, Alfred Albano, Hongwei Jin, Jonas B. Galper, Mark S. Link, Robert M. Blanton, Charles M. Welzig, Mark Aronovitz
المصدر: Heart Rhythm. 14:1406-1416
بيانات النشر: Elsevier BV, 2017.
سنة النشر: 2017
مصطلحات موضوعية: 0301 basic medicine, medicine.medical_specialty, Statin, medicine.drug_class, Heart Ventricles, Phosphatase, Myocardial Infarction, chemistry.chemical_element, Infarction, 030204 cardiovascular system & hematology, Calcium, Ventricular tachycardia, Article, Diabetes Mellitus, Experimental, Electrocardiography, Mice, 03 medical and health sciences, QRS complex, 0302 clinical medicine, Physiology (medical), Internal medicine, Animals, Medicine, Myocardial infarction, business.industry, medicine.disease, Mice, Inbred C57BL, Diabetes Mellitus, Type 1, 030104 developmental biology, chemistry, Tachycardia, Ventricular, Cardiology, Hydroxymethylglutaryl-CoA Reductase Inhibitors, Cardiology and Cardiovascular Medicine, business, Pravastatin, medicine.drug
الوصف: Background The incidence of sudden arrhythmic death is markedly increased in diabetics. Objective The purpose of this study was to develop a mouse model for postmyocardial infarction (post-MI) ventricular tachycardia (VT) in the diabetic heart and determine the mechanism of an antiarrhythmic effect of statins. Methods ECG transmitters were implanted in wild-type (WT), placebo, and pravastatin-treated type I diabetic Akita mice. MIs were induced by coronary ligation, and Ca 2+ transients were studied by optical mapping, and Ca 2+ transients and sparks in left ventricular myocytes (VM) by the Ionoptix system and confocal microscopy. Results Burst pacing of Akita mouse hearts resulted in rate-related QRS/T-wave alternans, which was attenuated in pravastatin-treated mice. Post-MI Akita mice developed QRS/T-wave alternans and VT at 2820 ± 879 beats per mouse, which decreased to 343 ± 115 in pravastatin-treated mice (n = 13, P 2+ alternans, both attenuated in hearts of statin-treated mice. Akita VM displayed Ca 2+ alternans, and triggered activity as well as increased Ca 2+ transient decay time (Tau), Ca 2+ sparks, and cytosolic Ca 2+ and decreased SR Ca 2+ stores all of which were in part reversed in cells from statin treated mice. Homogenates of Akita ventricles demonstrated decreased SERCA2a/PLB ratio and increased ratio of protein phosphatase (PP-1) to the PP-1 inhibitor PPI-1 which were reversed in homogenates of pravastatin-treated Akita mice. Conclusion Pravastatin decreased the incidence of post-MI VT and Ca 2+ alternans in Akita mouse hearts in part by revering abnormalities of Ca 2+ handling via the PP-1/PPI-1 pathway.
تدمد: 1547-5271
DOI: 10.1016/j.hrthm.2017.05.026
URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0ed330e7ebcc74b9b4feb8581533f4cb
https://doi.org/10.1016/j.hrthm.2017.05.026
Rights: OPEN
رقم الانضمام: edsair.doi.dedup.....0ed330e7ebcc74b9b4feb8581533f4cb
قاعدة البيانات: OpenAIRE
الوصف
تدمد:15475271
DOI:10.1016/j.hrthm.2017.05.026