The dengue virus NS1 protein conveys pro-inflammatory signals by docking onto human high-density lipoproteins

التفاصيل البيبلوغرافية
العنوان: The dengue virus NS1 protein conveys pro-inflammatory signals by docking onto human high-density lipoproteins
المؤلفون: X Zhang, Tamietti C, Stéphane Petres, Anastassia Mikhailova, Fasséli Coulibaly, Benfrid S, Park K, Eva Harris, Duong, Mariano Dellarole, James E. Voss, Marie Flamand, Philippe Buchy, Sakunthabaï A, Gérard Pehau-Arnaudet, Patrick England, Philippe Dussart, Félix A. Rey, Scott B. Biering, Sébastien Brûlé, Marie-Noëlle Ungeheuer, François Bontems, Bertrand Raynal
بيانات النشر: Cold Spring Harbor Laboratory, 2021.
سنة النشر: 2021
مصطلحات موضوعية: biology, Chemistry, viruses, virus diseases, biochemical phenomena, metabolism, and nutrition, Dengue virus, medicine.disease_cause, medicine.disease, Virulence factor, Complement system, Dengue fever, Cell biology, Pathogenesis, Immune system, medicine, biology.protein, lipids (amino acids, peptides, and proteins), Antibody, Lipoprotein
الوصف: The nonstructural NS1 protein is a virulence factor secreted by dengue virus (DENV)-infected cells. NS1 is known to alter the complement system, activate immune cells and perturb endothelial barriers. Here we show that pro-inflammatory signals are triggered by a high affinity complex formed between NS1 and human high-density lipoproteins (HDL). Electron microscopy images of the NS1-HDL complexes show spherical HDL particles with rod-shaped NS1 protrusions on their surface. These complexes are readily detectable in the plasma of hospitalized dengue patients using anti-apolipoprotein A-I (ApoA-I) antibodies specific of the HDL moiety. The functional reprogramming of HDL particles by the NS1 protein as a means to exacerbate systemic inflammation during DENV infection provides a new paradigm linking the human lipoprotein network to dengue pathogenesis.
DOI: 10.1101/2021.05.05.442454
URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_________::d7b4531e61af29360ccd12ea32278c4b
https://doi.org/10.1101/2021.05.05.442454
Rights: OPEN
رقم الانضمام: edsair.doi...........d7b4531e61af29360ccd12ea32278c4b
قاعدة البيانات: OpenAIRE
الوصف
DOI:10.1101/2021.05.05.442454