يعرض 1 - 20 نتائج من 30 نتيجة بحث عن '"rdxA"', وقت الاستعلام: 0.53s تنقيح النتائج
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    Academic Journal
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    Academic Journal

    المصدر: van der Wouden , EJ , Thijs , JC , Kusters , JG , van Zwet , AA & Kleibeuker , JH 2001 , ' Mechanism and clinical significance of metronidazole resistance in Helicobacter pylori ' , SCANDINAVIAN JOURNAL OF GASTROENTEROLOGY , vol. 36 , pp. 10-14 .

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    Academic Journal

    المساهمون: 330006,南昌大学一附院消化研究所, 330006,北京大学第三医院消化内科, 330006,南昌大学一附院检验科

    مصطلحات موضوعية: 幽门螺杆菌, 耐药基因, rdxA

    Relation: 江西医学检验.2007,25,(3),197-200.; 958292; http://hdl.handle.net/20.500.11897/248454

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    Academic Journal

    المؤلفون: Han, F., Yan, Z., Yan, X., Liu, S., Ho, B.

    المساهمون: MICROBIOLOGY

    المصدر: Scopus

    Relation: Han, F., Yan, Z., Yan, X., Liu, S., Ho, B. (2007). Alterations in rdxA and frxA genes and their upstream regions in metronidazole-resistant Helicobacter pylori isolates. Research in Microbiology 158 (1) : 38-44. ScholarBank@NUS Repository. https://doi.org/10.1016/j.resmic.2006.10.001; http://scholarbank.nus.edu.sg/handle/10635/24718; 000244828200005

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    Academic Journal

    المؤلفون: Lui, S.Y., Ling, K.L., Ho, B.

    المساهمون: BIOLOGICAL SCIENCES

    المصدر: Scopus

    مصطلحات موضوعية: Helicobacter pylori, Metronidazole, RAPD, rdxA gene

    Relation: Lui, S.Y.,Ling, K.L.,Ho, B. (2003-10). rdxA gene is an unlikely marker for metronidazole resistance in the Asian Helicobacter pylori isolates. Journal of Microbiology and Biotechnology 13 (5) : 751-758. ScholarBank@NUS Repository.; http://scholarbank.nus.edu.sg/handle/10635/101529; NOT_IN_WOS

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    المؤلفون: 陳蓓諭, Pei Yu Chen

    المساهمون: 王雯靜, Wen Ching Wang

    Time: 38

    وصف الملف: 155 bytes; text/html

    Relation: 伍、參考資料 1. Marshall, B.J. and J.R. Warren, Unidentified curved bacilli in the stomach of patients with gastritis and peptic ulceration. Lancet, 1984. 1(8390): p. 1311-5. 2. Goodwin, C.S., et al., Enzyme-linked immunosorbent assay for Campylobacter pyloridis: correlation with presence of C. pyloridis in the gastric mucosa. J Infect Dis, 1987. 155(3): p. 488-94. 3. Romaniuk, P.J., et al., Campylobacter pylori, the spiral bacterium associated with human gastritis, is not a true Campylobacter sp. J Bacteriol, 1987. 169(5): p. 2137-41. 4. Tomb, J.F., et al., The complete genome sequence of the gastric pathogen Helicobacter pylori. Nature, 1997. 388(6642): p. 539-47. 5. Alm, R.A., et al., Genomic-sequence comparison of two unrelated isolates of the human gastric pathogen Helicobacter pylori. Nature, 1999. 397(6715): p. 176-80. 6. Eaton, K.A., et al., Colonization of gnotobiotic piglets by Helicobacter pylori deficient in two flagellin genes. Infect Immun, 1996. 64(7): p. 2445-8. 7. Eaton, K.A. and S. Krakowka, Effect of gastric pH on urease-dependent colonization of gnotobiotic piglets by Helicobacter pylori. Infect Immun, 1994. 62(9): p. 3604-7. 8. Ilver, D., et al., Helicobacter pylori adhesin binding fucosylated histo-blood group antigens revealed by retagging. Science, 1998. 279(5349): p. 373-7. 9. Rad, R., et al., The Helicobacter pylori blood group antigen-binding adhesin facilitates bacterial colonization and augments a nonspecific immune response. J Immunol, 2002. 168(6): p. 3033-41. 10. Gerhard, M., et al., Clinical relevance of the Helicobacter pylori gene for blood-group antigen-binding adhesin. Proc Natl Acad Sci U S A, 1999. 96(22): p. 12778-83. 11. Mizushima, T., et al., Clinical relevance of the babA2 genotype of Helicobacter pylori in Japanese clinical isolates. J Clin Microbiol, 2001. 39(7): p. 2463-5. 12. Lai, C.H., et al., High prevalence of cagA- and babA2-positive Helicobacter pylori clinical isolates in Taiwan. J Clin Microbiol, 2002. 40(10): p. 3860-2. 13. Cover, T.L., S.A. Halter, and M.J. Blaser, Characterization of HeLa cell vacuoles induced by Helicobacter pylori broth culture supernatant. Hum Pathol, 1992. 23(9): p. 1004-10. 14. Atherton, J.C., et al., Mosaicism in vacuolating cytotoxin alleles of Helicobacter pylori. Association of specific vacA types with cytotoxin production and peptic ulceration. J Biol Chem, 1995. 270(30): p. 17771-7. 15. Wang, H.J., et al., Vacuolating toxin production in clinical isolates of Helicobacter pylori with different vacA genotypes. J Infect Dis, 1998. 178(1): p. 207-12. 16. Yang, J.C., et al., Genetic analysis of the cytotoxin-associated gene and the vacuolating toxin gene in Helicobacter pylori strains isolated from Taiwanese patients. Am J Gastroenterol, 1997. 92(8): p. 1316-21. 17. Montecucco, C., et al., Helicobacter pylori vacuolating cytotoxin: cell intoxication and anion-specific channel activity. Curr Top Microbiol Immunol, 2001. 257: p. 113-29. 18. Yamaoka, Y., et al., Variants of the 3' region of the cagA gene in Helicobacter pylori isolates from patients with different H. pylori-associated diseases. J Clin Microbiol, 1998. 36(8): p. 2258-63. 19. Blaser, M.J., et al., Infection with Helicobacter pylori strains possessing cagA is associated with an increased risk of developing adenocarcinoma of the stomach. Cancer Res, 1995. 55(10): p. 2111-5. 20. Prinz, C., et al., Key importance of the Helicobacter pylori adherence factor blood group antigen binding adhesin during chronic gastric inflammation. Cancer Res, 2001. 61(5): p. 1903-9. 21. Wong, B.C., et al., Distribution of distinct vacA, cagA and iceA alleles in Helicobacter pylori in Hong Kong. Helicobacter, 2001. 6(4): p. 317-24. 22. Sheu, S.M., et al., Presence of iceA1 but not cagA, cagC, cagE, cagF, cagN, cagT, or orf13 genes of Helicobacter pylori is associated with more severe gastric inflammation in Taiwanese. J Formos Med Assoc, 2002. 101(1): p. 18-23. 23. Goodwin, C.S., M.M. Mendall, and T.C. Northfield, Helicobacter pylori infection. Lancet, 1997. 349(9047): p. 265-9. 24. Frenck, R.W., Jr. and J. Clemens, Helicobacter in the developing world. Microbes Infect, 2003. 5(8): p. 705-13. 25. Rothenbacher, D. and H. Brenner, Burden of Helicobacter pylori and H. pylori-related diseases in developed countries: recent developments and future implications. Microbes Infect, 2003. 5(8): p. 693-703. 26. Lin, J.T., et al., Ecological study of association between Helicobacter pylori infection and gastric cancer in Taiwan. Dig Dis Sci, 1995. 40(2): p. 385-8. 27. Lin, C.W., et al., Helicobacter pylori in gastric biopsies of Taiwanese patients with gastroduodenal diseases. Jpn J Med Sci Biol, 1998. 51(1): p. 13-23. 28. Blaser, M.J., Helicobacter pylori: its role in disease. Clin Infect Dis, 1992. 15(3): p. 386-91. 29. Roggero, E., et al., Eradication of Helicobacter pylori infection in primary low-grade gastric lymphoma of mucosa-associated lymphoid tissue. Ann Intern Med, 1995. 122(10): p. 767-9. 30. Wotherspoon, A.C., et al., Regression of primary low-grade B-cell gastric lymphoma of mucosa-associated lymphoid tissue type after eradication of Helicobacter pylori. Lancet, 1993. 342(8871): p. 575-7. 31. Forman, D., Review article: Is there significant variation in the risk of gastric cancer associated with Helicobacter pylori infection? Aliment Pharmacol Ther, 1998. 12 Suppl 1: p. 3-7. 32. Forman, D., et al., Association between infection with Helicobacter pylori and risk of gastric cancer: evidence from a prospective investigation. Bmj, 1991. 302(6788): p. 1302-5. 33. Voutilainen, M., et al., Impact of non-steroidal anti-inflammatory drug and aspirin use on the prevalence of dyspepsia and uncomplicated peptic ulcer disease. Scand J Gastroenterol, 2001. 36(8): p. 817-21. 34. Macarthur, C., N. Saunders, and W. Feldman, Helicobacter pylori, gastroduodenal disease, and recurrent abdominal pain in children. Jama, 1995. 273(9): p. 729-34. 35. Wyatt, J.I., et al., Helicobacter pylori, gastritis, and peptic ulceration in the elderly. J Clin Pathol, 1992. 45(12): p. 1070-4. 36. Graham, D.Y., et al., Effect of treatment of Helicobacter pylori infection on the long-term recurrence of gastric or duodenal ulcer. A randomized, controlled study. Ann Intern Med, 1992. 116(9): p. 705-8. 37. Queiroz, D.M., et al., Role of corpus gastritis and cagA-positive Helicobacter pylori infection in reflux esophagitis. J Clin Microbiol, 2002. 40(8): p. 2849-53. 38. Suerbaum, S. and P. Michetti, Helicobacter pylori infection. N Engl J Med, 2002. 347(15): p. 1175-86. 39. Malfertheiner, P., et al., Current European concepts in the management of Helicobacter pylori infection--the Maastricht Consensus Report. The European Helicobacter Pylori Study Group (EHPSG). Eur J Gastroenterol Hepatol, 1997. 9(1): p. 1-2. 40. Lam, S.K. and N.J. Talley, Report of the 1997 Asia Pacific Consensus Conference on the management of Helicobacter pylori infection. J Gastroenterol Hepatol, 1998. 13(1): p. 1-12. 41. de Boer, W.A., et al., Quadruple therapy compared with dual therapy for eradication of Helicobacter pylori in ulcer patients: results of a randomized prospective single-centre study. Eur J Gastroenterol Hepatol, 1995. 7(12): p. 1189-94. 42. McLoughlin, R., et al., Therapy of Helicobacter pylori. Helicobacter, 2004. 9 Suppl 1: p. 42-8. 43. Debets-Ossenkopp, Y.J., et al., Mechanism of clarithromycin resistance in clinical isolates of Helicobacter pylori. FEMS Microbiol Lett, 1996. 142(1): p. 37-42. 44. Lopez-Brea, M., et al., Evolution of resistance to metronidazole and clarithromycin in Helicobacter pylori clinical isolates from Spain. J Antimicrob Chemother, 1997. 40(2): p. 279-81. 45. Cameron, E.A., et al., Helicobacter pylori: antibiotic resistance and eradication rates in Suffolk, UK, 1991-2001. J Med Microbiol, 2004. 53(Pt 6): p. 535-8. 46. Ling, T.K., et al., An increase in Helicobacter pylori strains resistant to metronidazole: a five-year study. Helicobacter, 1996. 1(1): p. 57-61. 47. Poon, S.K., et al., Primary resistance to antibiotics and its clinical impact on the efficacy of Helicobacter pylori lansoprazole-based triple therapies. Aliment Pharmacol Ther, 2002. 16(2): p. 291-6. 48. Megraud, F., Epidemiology and mechanism of antibiotic resistance in Helicobacter pylori. Gastroenterology, 1998. 115(5): p. 1278-82. 49. Ching, C.K., et al., Prevalence of metronidazole resistant Helicobacter pylori strains among Chinese peptic ulcer disease patients and normal controls in Hong Kong. Gut, 1996. 38(5): p. 675-8. 50. Rautelin, H., et al., Role of metronidazole resistance in therapy of Helicobacter pylori infections. Antimicrob Agents Chemother, 1992. 36(1): p. 163-6. 51. Van Der Wouden, E.J., et al., Review article: nitroimidazole resistance in Helicobacter pylori. Aliment Pharmacol Ther, 2000. 14(1): p. 7-14. 52. Cederbrant, G., G. Kahlmeter, and A. Ljungh, Proposed mechanism for metronidazole resistance in Helicobacter pylori. J Antimicrob Chemother, 1992. 29(2): p. 115-20. 53. Smith, M.A. and D.I. Edwards, The influence of microaerophilia and anaerobiosis on metronidazole uptake in Helicobacter pylori. J Antimicrob Chemother, 1995. 36(3): p. 453-61. 54. Walsh, C., Molecular mechanisms that confer antibacterial drug resistance. Nature, 2000. 406(6797): p. 775-81. 55. Goodwin, A., et al., Metronidazole resistance in Helicobacter pylori is due to null mutations in a gene (rdxA) that encodes an oxygen-insensitive NADPH nitroreductase. Mol Microbiol, 1998. 28(2): p. 383-93. 56. Gerrits, M.M., et al., Role of the rdxA and frxA genes in oxygen-dependent metronidazole resistance of Helicobacter pylori. J Med Microbiol, 2004. 53(Pt 11): p. 1123-8. 57. Marais, A., et al., Characterization of the genes rdxA and frxA involved in metronidazole resistance in Helicobacter pylori. Res Microbiol, 2003. 154(2): p. 137-44. 58. Chisholm, S.A. and R.J. Owen, Mutations in Helicobacter pylori rdxA gene sequences may not contribute to metronidazole resistance. J Antimicrob Chemother, 2003. 51(4): p. 995-9. 59. Goddard, A.F. and R.P. Logan, Antimicrobial resistance and Helicobacter pylori. J Antimicrob Chemother, 1996. 37(4): p. 639-43. 60. Logan, R.P., et al., Eradication of Helicobacter pylori with clarithromycin and omeprazole. Gut, 1994. 35(3): p. 323-6. 61. Occhialini, A., et al., Macrolide resistance in Helicobacter pylori: rapid detection of point mutations and assays of macrolide binding to ribosomes. Antimicrob Agents Chemother, 1997. 41(12): p. 2724-8. 62. Stone, G.G., et al., Identification of a 23S rRNA gene mutation in clarithromycin-resistant Helicobacter pylori. Helicobacter, 1996. 1(4): p. 227-8. 63. Versalovic, J., et al., Point mutations in the 23S rRNA gene of Helicobacter pylori associated with different levels of clarithromycin resistance. J Antimicrob Chemother, 1997. 40(2): p. 283-6. 64. Mendonca, S., et al., Prevalence of Helicobacter pylori resistance to metronidazole, clarithromycin, amoxicillin, tetracycline, and furazolidone in Brazil. Helicobacter, 2000. 5(2): p. 79-83. 65. Midolo, P.D. and W. Sievert, Amoxycillin resistance in an isolate of Helicobacter pylori from Melbourne. Med J Aust, 2000. 173(10): p. 555. 66. Smith, S.I., et al., High amoxycillin resistance in Helicobacter pylori isolated from gastritis and peptic ulcer patients in western Nigeria. J Gastroenterol, 2001. 36(1): p. 67-8. 67. Watanabe, K., et al., Amoxicillin resistance in Helicobacter pylori: studies from Tokyo, Japan from 1985 to 2003. Helicobacter, 2005. 10(1): p. 4-11. 68. Dore, M.P., et al., Amoxycillin tolerance in Helicobacter pylori. J Antimicrob Chemother, 1999. 43(1): p. 47-54. 69. Dore, M.P., D.Y. Graham, and A.R. Sepulveda, Different penicillin-binding protein profiles in amoxicillin-resistant Helicobacter pylori. Helicobacter, 1999. 4(3): p. 154-61. 70. Akopyanz, N., et al., DNA diversity among clinical isolates of Helicobacter pylori detected by PCR-based RAPD fingerprinting. Nucleic Acids Res, 1992. 20(19): p. 5137-42. 71. Wong, W.M., et al., Standard treatment for Helicobacter pylori infection is suboptimal in non-ulcer dyspepsia compared with duodenal ulcer in Chinese. Aliment Pharmacol Ther, 2005. 21(1): p. 73-81. 72. Vakil, N., et al., Seven-day therapy for Helicobacter pylori in the United States. Aliment Pharmacol Ther, 2004. 20(1): p. 99-107. 73. Marko, D., et al., Comparison of two management strategies for Helicobacter pylori treatment: clinical study and cost-effectiveness analysis. Helicobacter, 2005. 10(1): p. 22-32. 74. Veldhuyzen van Zanten, S., et al., A randomized trial comparing seven-day ranitidine bismuth citrate and clarithromycin dual therapy to seven-day omeprazole, clarithromycin and amoxicillin triple therapy for the eradication of Helicobacter pylori. Can J Gastroenterol, 2003. 17(9): p. 533-8. 75. Coelho, L.G., et al., [Efficacy of the dosing regimen of pantoprazole 40 mg, amoxicillin 1000 mg and clarithromycin 500 mg, twice daily for 7 days, in the eradication of Helicobacter pylori in patients with peptic ulcer]. Arq Gastroenterol, 2004. 41(1): p. 71-6. 76. De Francesco, V., et al., Sequential treatment for Helicobacter pylori does not share the risk factors of triple therapy failure. Aliment Pharmacol Ther, 2004. 19(4): p. 407-14. 77. Koivisto, T.T., et al., Primary Helicobacter pylori resistance to metronidazole and clarithromycin in the Finnish population. Aliment Pharmacol Ther, 2004. 19(9): p. 1009-17. 78. Nahar, S., et al., Antimicrobial susceptibility of Helicobacter pylori strains isolated in Bangladesh. J Clin Microbiol, 2004. 42(10): p. 4856-8. 79. Lee, M.G., et al., Antibiotic sensitivity of Helicobacter pylori in Jamaica. West Indian Med J, 2004. 53(6): p. 374-7. 80. Ahmed, D., et al., Antibiotic resistance in Helicobacter pylori: is it a problem in New Zealand? N Z Med J, 2004. 117(1200): p. U1022. 81. Tangmankongworakoon, N., et al., Pattern of drug resistant Helicobacter pylori in dyspeptic patients in Thailand. J Med Assoc Thai, 2003. 86 Suppl 2: p. S439-44. 82. Eun, C.S., et al., Changing pattern of antimicrobial resistance of Helicobacter pylori in Korean patients with peptic ulcer diseases. J Gastroenterol, 2003. 38(5): p. 436-41. 83. Kuo, C.H., et al., Heterogeneous Helicobacter pylori isolates from H. pylori-infected couples in Taiwan. J Infect Dis, 1999. 180(6): p. 2064-8. 84. Yang, J.C., et al., Vacuolating toxin gene polymorphism among Helicobacter pylori clinical isolates and its association with m1, m2, or chimeric vacA middle types. Scand J Gastroenterol, 1998. 33(11): p. 1152-7. 85. Godoy, A.P., et al., Analysis of antimicrobial susceptibility and virulence factors in Helicobacter pylori clinical isolates. BMC Gastroenterol, 2003. 3(1): p. 20. 86. Megraud, F., Basis for the management of drug-resistant Helicobacter pylori infection. Drugs, 2004. 64(17): p. 1893-904. 87. Debets-Ossenkopp, Y.J., et al., Insertion of mini-IS605 and deletion of adjacent sequences in the nitroreductase (rdxA) gene cause metronidazole resistance in Helicobacter pylori NCTC11637. Antimicrob Agents Chemother, 1999. 43(11): p. 2657-62. 88. Kwon, D.H., et al., Analysis of rdxA and involvement of additional genes encoding NAD(P)H flavin oxidoreductase (FrxA) and ferredoxin-like protein (FdxB) in metronidazole resistance of Helicobacter pylori. Antimicrob Agents Chemother, 2000. 44(8): p. 2133-42. 89. Versalovic, J., et al., Mutations in 23S rRNA are associated with clarithromycin resistance in Helicobacter pylori. Antimicrob Agents Chemother, 1996. 40(2): p. 477-80. 90. Hsieh, P.F., et al., Molecular mechanisms of clarithromycin resistance in Helicobacter pylori. J Formos Med Assoc, 1998. 97(7): p. 445-52. 91. Arents, N.L., et al., The importance of vacA, cagA, and iceA genotypes of Helicobacter pylori infection in peptic ulcer disease and gastroesophageal reflux disease. Am J Gastroenterol, 2001. 96(9): p. 2603-8. 92. Toracchio, S., et al., Identification of a novel mutation affecting domain V of the 23S rRNA gene in Helicobacter pylori. Helicobacter, 2004. 9(5): p. 396-9. 93. de Boer, W.A., et al., Randomized study comparing 1 with 2 weeks of quadruple therapy for eradicating Helicobacter pylori. Am J Gastroenterol, 1994. 89(11): p. 1993-7. 94. Salcedo, J.A. and F. Al-Kawas, Treatment of Helicobacter pylori infection. Arch Intern Med, 1998. 158(8): p. 842-51. 95. Walsh, J.H. and W.L. Peterson, The treatment of Helicobacter pylori infection in the management of peptic ulcer disease. N Engl J Med, 1995. 333(15): p. 984-91. 96. Jeong, J.Y., et al., Sequential inactivation of rdxA (HP0954) and frxA (HP0642) nitroreductase genes causes moderate and high-level metronidazole resistance in Helicobacter pylori. J Bacteriol, 2000. 182(18): p. 5082-90.; http://nthur.lib.nthu.edu.tw/dspace/handle/987654321/30525

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    Dissertation/ Thesis

    المؤلفون: LUI SOOK YIN

    المساهمون: MICROBIOLOGY, HO BOW

    Relation: LUI SOOK YIN (2004-03-27). Characterization of metronidazole resistant Helicobacter pylori. ScholarBank@NUS Repository.; http://scholarbank.nus.edu.sg/handle/10635/13802; NOT_IN_WOS