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1Academic Journal
المؤلفون: Matthews, L., Berry, A., Tersigni, M., D'Acquisto, F., Ianaro, A., Ray, D.
المصدر: Matthews , L , Berry , A , Tersigni , M , D'Acquisto , F , Ianaro , A & Ray , D 2009 , ' Thiazolidinediones are partial agonists for the glucocorticoid receptor ' , Endocrinology , vol. 150 , no. 1 , pp. 75-86 . https://doi.org/10.1210/en.2008-0196
مصطلحات موضوعية: drug effects: Cell Division, Cell Line, Tumor, antagonists & inhibitors: Dexamethasone, Enzyme Activation, Genes, Reporter, Hela Cells, pharmacology: Hormone Antagonists, Humans, genetics: Luciferases, Lung Neoplasms, pharmacology: Mifepristone, physiology: PPAR gamma, agonists: Receptors, Glucocorticoid, pharmacology: Thiazolidinediones, Transfection
وصف الملف: application/octet-stream
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2
المؤلفون: Angelika Griep, Mitsugu Yamanaka, Daisy Axt, Markus P. Kummer, Michael T. Heneka, Taizo Ishikawa
المصدر: The journal of neuroscience 32(48), 17321-17331 (2012). doi:10.1523/JNEUROSCI.1569-12.2012
مصطلحات موضوعية: psychology [Alzheimer Disease], Journal Club, CD36, metabolism [Microglia], drug effects [Microglia], Amyloid beta-Protein Precursor, Mice, Cognition, metabolism [Amyloid beta-Protein Precursor], drug effects [Behavior, Animal], Amyloid precursor protein, drug therapy [Alzheimer Disease], Retinoid, Receptor, Behavior, Animal, biology, metabolism [Presenilin-1], General Neuroscience, physiology [Cognition], Brain, genetics [Presenilin-1], Cell biology, physiology [Behavior, Animal], Biochemistry, genetics [Amyloid beta-Protein Precursor], drug effects [Cognition], drug effects [Brain], physiology [Phagocytosis], Microglia, metabolism [Alzheimer Disease], medicine.drug_class, Phagocytosis, pharmacology [Hypoglycemic Agents], pharmacology [Thiazolidinediones], drug effects [Phagocytosis], drug effects [Maze Learning], Presenilin, physiology [Maze Learning], Downregulation and upregulation, Alzheimer Disease, Presenilin-1, medicine, Animals, Hypoglycemic Agents, ddc:610, Scavenger receptor, Maze Learning, therapeutic use [Thiazolidinediones], Pioglitazone, PPAR gamma, Disease Models, Animal, metabolism [Brain], biology.protein, therapeutic use [Hypoglycemic Agents], Thiazolidinediones, agonists [PPAR gamma]
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3
المؤلفون: Vercruysse, Pauline, Sinniger, Jérôme, Dreyhaupt, Jens, Grehl, Torsten, Hermann, Andreas, Grosskreutz, Julian, Witting, Anke, Van Den Bosch, Ludo, Spreux-Varoquaux, Odile, Group, GERP ALS Study, Ludolph, Albert C, Dupuis, Luc, El Oussini, Hajer, Borisow, Nadja, Holm, Theresa, Maier, Andre, Meyer, Thomas, Budde, Paula, Gruhn, Kai, Bewersdorff, Malte, Heneka, Michael, Storch, Alexander, Scekic-Zahirovic, Jelena, Frank, Tobias, Göricke, Bettina, Weishaupt, Jochen, Eger, Katharina, Hanisch, Frank, Zierz, Stephan, Boeck, Anna-Lena, Dengler, Reinhard, Koerner, Sonja, Kollewe, Katja, Dieterlé, Stéphane, Petri, Susanne, Prell, Tino, Ringer, Thomas, Zinke, Jan, Anneser, Johanna, Borasio, Gian Domenico, Chahli, Christine, Winkler, Andrea S, Boentert, Matthias, Stubbe-Draeger, Bianca, Young, Peter, Bogdahn, Ulrich, Franz, Steffen, Haringer, Verena, Weidner, Norbert, Benecke, Reiner, Meister, Stefanie, Prudlo, Johannes, Wittstock, Matthias, Dorst, Johannes, Hendrich, Corinna, Sperfeld, Anne-Dorte, Weiland, Ulrike, Neidhardt, Sabine, Schrank, Berthold, Beck, Marcus, Kraft, Peter, Toyka, Klaus, Ulzheimer, Jochen, Wessig, Carsten, Kassubek, Jan, Fischer, Wilhelm
المساهمون: Mécanismes Centraux et Périphériques de la Neurodégénérescence, Université de Strasbourg (UNISTRA)-Institut National de la Santé et de la Recherche Médicale (INSERM), Hannover Medical School [Hannover] (MHH), Charité - UniversitätsMedizin = Charité - University Hospital [Berlin], Martin-Luther-University Halle-Wittenberg, University of Ulm (UUlm), Ruhr-Universität Bochum [Bochum], Technische Universität Dresden = Dresden University of Technology (TU Dresden), German Research Center for Neurodegenerative Diseases - Deutsches Zentrum für Neurodegenerative Erkrankungen (DZNE), Jena University Hospital [Jena], Catholic University of Leuven - Katholieke Universiteit Leuven (KU Leuven), Université de Versailles Saint-Quentin-en-Yvelines (UVSQ), GERP ALS Study Group: Nadja Borisow, Theresa Holm, Andre Maier, Thomas Meyer, Paula Budde, Torsten Grehl, Kai Gruhn, Malte Bewersdorff, Michael Heneka, Andreas Hermann, Alexander Storch, Tobias Frank, Bettina Göricke, Jochen Weishaupt, Katharina Eger, Frank Hanisch, Stephan Zierz, Anna-Lena Boeck, Reinhard Dengler, Sonja Koerner, Katja Kollewe, Susanne Petri, Julian Grosskreutz, Tino Prell, Thomas Ringer, Jan Zinke, Johanna Anneser, Gian Domenico Borasio, Christine Chahli, Andrea S Winkler, Matthias Boentert, Bianca Stubbe-Draeger, Peter Young, Ulrich Bogdahn, Steffen Franz, Verena Haringer, Norbert Weidner, Reiner Benecke, Stefanie Meister, Johannes Prudlo, Matthias Wittstock, Johannes Dorst, Corinna Hendrich, Albert C Ludolph, Anne-Dorte Sperfeld, Ulrike Weiland, Sabine Neidhardt, Berthold Schrank, Marcus Beck, Peter Kraft, Klaus Toyka, Jochen Ulzheimer, Carsten Wessig., Dieterle, Stéphane
المصدر: Brain 139(4), 1106-1122 (2016). doi:10.1093/brain/aww004
Brain-A Journal of Neurology
Brain-A Journal of Neurology, Oxford University Press (OUP), 2016, 139 (4), pp.1106-1122. ⟨10.1093/brain/aww004⟩
Brain-A Journal of Neurology, 2016, 139 (4), pp.1106-1122. ⟨10.1093/brain/aww004⟩مصطلحات موضوعية: MESH: Signal Transduction, 0301 basic medicine, Male, Pro-Opiomelanocortin, calorie intake, [SDV]Life Sciences [q-bio], MESH: Thiazolidinediones, MESH: Synapses, MESH: Spatial Memory, [SCCO]Cognitive science, Mice, MESH: Riluzole, 0302 clinical medicine, Superoxide Dismutase-1, MESH: Transcription Factor RelA, Sod1 protein, mouse, thiazolinediones, genetics [Superoxide Dismutase], MESH: Pro-Opiomelanocortin, MESH: Animals, Amyotrophic lateral sclerosis, MESH: Superoxide Dismutase, therapeutic use [Riluzole], MESH: Superoxide Dismutase-1, 2. Zero hunger, Riluzole, Leptin, digestive, oral, and skin physiology, SOD1 protein, human, metabolism [Hypothalamus], 3. Good health, [SDV] Life Sciences [q-bio], genetics [Amyotrophic Lateral Sclerosis], Female, Melanocortin, hormones, hormone substitutes, and hormone antagonists, medicine.drug, Signal Transduction, medicine.medical_specialty, drug effects [Signal Transduction], MESH: Mice, Transgenic, MESH: Pioglitazone, metabolism [Superoxide Dismutase], SOD1, Hypothalamus, Mice, Transgenic, drug effects [Hypothalamus], pharmacology [Thiazolidinediones], Biology, TARDBP, pathology [Hypothalamus], 03 medical and health sciences, Proopiomelanocortin, MESH: Mice, Inbred C57BL, MESH: Rats, Long-Evans, Internal medicine, physiology [Signal Transduction], medicine, metabolism [Pro-Opiomelanocortin], Animals, Humans, ddc:610, weight loss, MESH: Mice, MESH: Humans, therapeutic use [Thiazolidinediones], drug therapy [Amyotrophic Lateral Sclerosis], Pioglitazone, genetics [Pro-Opiomelanocortin], Superoxide Dismutase, metabolism [Amyotrophic Lateral Sclerosis], Amyotrophic Lateral Sclerosis, pharmacology [Riluzole], [SCCO] Cognitive science, medicine.disease, MESH: Hypothalamus, MESH: Male, Mice, Inbred C57BL, 030104 developmental biology, Endocrinology, biology.protein, Thiazolidinediones, Neurology (clinical), MESH: Female, 030217 neurology & neurosurgery
وصف الملف: application/pdf
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4Pan-PPAR modulation effectively protects APP/PS1 mice from amyloid deposition and cognitive deficits
المؤلفون: Stephanie Schwartz, Markus P. Kummer, Dean W. Hum, Rafael Schwarzenberger, Robert Walczak, Mathieu Dubernet, Sakina Sayah-Jeanne, Michael T. Heneka, Daisy Axt
المصدر: Molecular neurobiology 51(2), 661-671 (2014). doi:10.1007/s12035-014-8743-4
Molecular Neurobiology
Mol Neurobiol. 51(2) 661-71 (2014)
ZENODO
Europe PubMed Centralمصطلحات موضوعية: Apolipoprotein E, Male, genetics [Plaque, Amyloid], Morris water navigation task, Plaque, Amyloid, genetics [Cognition Disorders], PPAR, PPAR agonist, Mice, Amyloid beta-Protein Precursor, 0302 clinical medicine, Chlorocebus aethiops, 0303 health sciences, Glial fibrillary acidic protein, biology, Chemistry, genetics [Presenilin-1], Treatment Outcome, Neurology, genetics [Amyloid beta-Protein Precursor], COS Cells, Female, medicine.drug, Agonist, medicine.medical_specialty, Amyloid, medicine.drug_class, Neuroscience (miscellaneous), prevention & control [Cognition Disorders], Mice, Transgenic, pharmacology [Thiazolidinediones], Article, physiology [PPAR gamma], 03 medical and health sciences, Cellular and Molecular Neuroscience, Internal medicine, ddc:570, medicine, Presenilin-1, Animals, Neuroinflammation, 030304 developmental biology, Inflammation, Behavior, therapeutic use [Thiazolidinediones], Pioglitazone, Mice, Inbred C57BL, prevention & control [Plaque, Amyloid], PPAR gamma, Alzheimer . . ., Endocrinology, biology.protein, Alzheimer, Thiazolidinediones, Cognition Disorders, agonists [PPAR gamma], 030217 neurology & neurosurgery
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5Academic Journal
المؤلفون: Heppner, T, Bonev, A, Eckman, D, Gomez, M, Petkov, G, Nelson, MT.
المصدر: Heppner , T , Bonev , A , Eckman , D , Gomez , M , Petkov , G & Nelson , MT 2005 , ' Novel PPARgamma agonists GI 262570, GW 7845, GW 1929, and pioglitazone decrease calcium channel function and myogenic tone in rat mesenteric arteries. ' , Pharmacology , vol. 73( 1) .
مصطلحات موضوعية: Algorithms, Animals, antagonists & inhibitors: Barium, pharmacology: Benzophenones, drug effects: Blood Pressure, drug effects: Calcium Channels, Electrophysiology, Female, pharmacology: Hypoglycemic Agents, drug effects: Membrane Potentials, cytology: Mesenteric Arteries, drug effects: Muscle Cells, drug effects: Muscle Tonus, cytology: Muscle, Smooth, Vascular, pharmacology: Oxazoles, agonists: PPAR gamma, Patch-Clamp Techniques, Rats, Sprague-Dawley, pharmacology: Thiazolidinediones, analogs & derivatives: Tyrosine, drug effects: Vasodilation
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6
المؤلفون: Mario M. Dorostkar, Jasmin Ohli, Yuan Shi, Jochen Herms, Chengyu Zou, Ulrich Schüller
المصدر: Acta neuropathologica 131(2), 235-246 (2016). doi:10.1007/s00401-015-1527-8
Acta Neuropathologicaمصطلحات موضوعية: 0301 basic medicine, Pathology, Dendritic spine, drug effects [Pyramidal Cells], Anti-Inflammatory Agents, genetics [Amyloid Precursor Protein Secretases], immunology [Dendritic Spines], drug effects [Neuroimmunomodulation], pathology [Alzheimer Disease], 0302 clinical medicine, drug effects [Somatosensory Cortex], Neuroinflammation, drug effects [Neuronal Plasticity], drug therapy [Alzheimer Disease], Aspartic Acid Endopeptidases, immunology [Pyramidal Cells], Neuronal Plasticity, Pyramidal Cells, metabolism [Aspartic Acid Endopeptidases], Pathophysiology, APPswe/PS1deltaE9 mice, pathology [Pyramidal Cells], Female, Genetically modified mouse, Preclinical AD, medicine.medical_specialty, Neuroimmunomodulation, Dendritic Spines, Bace1 protein, mouse, Clinical Neurology, Mice, Transgenic, pharmacology [Thiazolidinediones], immunology [Neuroimmunomodulation], Pathology and Forensic Medicine, 03 medical and health sciences, Cellular and Molecular Neuroscience, In vivo, Alzheimer Disease, medicine, Animals, ddc:610, drug effects [Dendritic Spines], Structural plasticity, Receptors, Interleukin-1 Type I, Environmental enrichment, Original Paper, Pioglitazone, business.industry, Somatosensory Cortex, pathology [Somatosensory Cortex], pharmacology [Anti-Inflammatory Agents], metabolism [Amyloid Precursor Protein Secretases], immunology [Neuronal Plasticity], immunology [Somatosensory Cortex], Spine (zoology), immunology [Alzheimer Disease], Mice, Inbred C57BL, Disease Models, Animal, 030104 developmental biology, Interleukin 1 receptor antagonist, genetics [Aspartic Acid Endopeptidases], metabolism [Receptors, Interleukin-1 Type I], antagonists & inhibitors [Receptors, Interleukin-1 Type I], Thiazolidinediones, Neurology (clinical), Amyloid Precursor Protein Secretases, business, pathology [Dendritic Spines], Neuroscience, 030217 neurology & neurosurgery