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1Electronic ResourceHuman Plasminogen Exacerbates Clostridioides difficile Enteric Disease and Alters the Spore Surface.
المؤلفون: Whisstock J.C., Medcalf R.L., Sanderson-Smith M., Cordwell S.J., Law R.H.P., Lyras D., Awad M.M., Hutton M.L., Quek A.J., Klare W.P., Mileto S.J., Mackin K., Ly D., Oorschot V., Bosnjak M., Jenkin G., Conroy P.J., West N., Fulcher A., Costin A., Day C.J., Jennings M.P.
مصطلحات الفهرس: animal tissue, article, bacterial spore, Clostridium difficile infection, controlled study, cytokine production, disease exacerbation, disease severity, human, immune dysregulation, intestine infection, intestine injury, intestine tissue, mouse, nonhuman, plasminogen activation, priority journal, proteomics, spore germination, survival, tissue injury, cytokine/ec [Endogenous Compound], granulocyte colony stimulating factor/ec [Endogenous Compound], granulocyte macrophage colony stimulating factor/ec [Endogenous Compound], interleukin 10/ec [Endogenous Compound], interleukin 12p40/ec [Endogenous Compound], interleukin 1alpha/ec [Endogenous Compound], interleukin 1beta/ec [Endogenous Compound], interleukin 3/ec [Endogenous Compound], keratinocyte derived chemokine/ec [Endogenous Compound], monocyte chemotactic protein 1/ec [Endogenous Compound], plasmin/ec [Endogenous Compound], plasminogen, tissue inhibitor of metalloproteinase/ec [Endogenous Compound], unclassified drug, male, animal cell, animal experiment, animal model, mp1a protein/ec [Endogenous Compound], mp1b protein/ec [Endogenous Compound], Article
URL:
https://repository.monashhealth.org/monashhealthjspui/handle/1/28950
Gastroenterology
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2Electronic ResourceHuman Plasminogen Exacerbates Clostridioides difficile Enteric Disease and Alters the Spore Surface.
المؤلفون: Whisstock J.C., Medcalf R.L., Sanderson-Smith M., Cordwell S.J., Law R.H.P., Lyras D., Awad M.M., Hutton M.L., Quek A.J., Klare W.P., Mileto S.J., Mackin K., Ly D., Oorschot V., Bosnjak M., Jenkin G., Conroy P.J., West N., Fulcher A., Costin A., Day C.J., Jennings M.P.
مصطلحات الفهرس: animal tissue, article, bacterial spore, Clostridium difficile infection, controlled study, cytokine production, disease exacerbation, disease severity, human, immune dysregulation, intestine infection, intestine injury, intestine tissue, mouse, nonhuman, plasminogen activation, priority journal, proteomics, spore germination, survival, tissue injury, cytokine/ec [Endogenous Compound], granulocyte colony stimulating factor/ec [Endogenous Compound], granulocyte macrophage colony stimulating factor/ec [Endogenous Compound], interleukin 10/ec [Endogenous Compound], interleukin 12p40/ec [Endogenous Compound], interleukin 1alpha/ec [Endogenous Compound], interleukin 1beta/ec [Endogenous Compound], interleukin 3/ec [Endogenous Compound], keratinocyte derived chemokine/ec [Endogenous Compound], monocyte chemotactic protein 1/ec [Endogenous Compound], plasmin/ec [Endogenous Compound], plasminogen, tissue inhibitor of metalloproteinase/ec [Endogenous Compound], unclassified drug, male, animal cell, animal experiment, animal model, mp1a protein/ec [Endogenous Compound], mp1b protein/ec [Endogenous Compound], Article
URL:
https://repository.monashhealth.org/monashhealthjspui/handle/1/28950
Gastroenterology
LibKey Link