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المؤلفون: Felix F. Brockschmidt, Rachid Tazi-Ahnini, Markus M. Nöthen, Hans Wolff, Kathrin A. Giehl, Nadine Kluck, Mary P. Birch, Silke Redler, Melanie Refke, K. Dobson, Regina C. Betz, Andrew G. Messenger, Tim Becker, Markus Böhm, Dmitriy Drichel, Roland Kruse, Gerhard Lutz
المصدر: Experimental dermatology 21(5), 390-393 (2012). doi:10.1111/j.1600-0625.2012.01469.x
مصطلحات موضوعية: medicine.medical_specialty, SRD5A2 protein, human, genetics [3-Oxo-5-alpha-Steroid 4-Dehydrogenase], Dermatology, Biology, Biochemistry, genetics [Estrogen Receptor beta], 3-Oxo-5-alpha-Steroid 4-Dehydrogenase, Germany, Internal medicine, Progesterone receptor, medicine, Humans, Estrogen Receptor beta, ddc:610, SRD5A1 protein, human, Receptor, estrogen receptor alpha, human, Molecular Biology, Alleles, genetics [Alopecia], Estrogen Receptor alpha, Genetic Variation, Membrane Proteins, Alopecia, medicine.disease, genetics [Genetic Variation], United Kingdom, ethnology [Alopecia], genetics [Membrane Proteins], Endocrinology, Hair loss, Receptors, Estrogen, Hormone receptor, Sex steroid, Case-Control Studies, SRD5A2, genetics [Receptors, Progesterone], Female, genetics [Estrogen Receptor alpha], genetics [Receptors, Estrogen], Receptors, Progesterone, Estrogen receptor alpha, Hormone
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المؤلفون: Eva Benito, Sankari Nagarajan, Andre Fischer, Steven A. Johnsen
المصدر: Nagarajan, S, Benito, E, Fischer, A & Johnsen, S A 2015, ' H4K12ac is regulated by estrogen receptor-alpha and is associated with BRD4 function and inducible transcription ', Oncotarget, vol. 6, no. 9, pp. 7305-7317 . https://doi.org/10.18632/oncotarget.v6i9
Oncotarget
OncoTarget 6(9), 7305-7317 (2015). doi:10.18632/oncotarget.3439مصطلحات موضوعية: metabolism [Histones], Transcription, Genetic, Enhancer RNAs, Cell Cycle Proteins, metabolism [Estrogen Receptor alpha], Epigenesis, Genetic, Histones, 0302 clinical medicine, bromodomain, metabolism [Estrogens], estrogen, metabolism [Transcription Factors], 0303 health sciences, Nuclear Proteins, Acetylation, Genomics, Histone acetylation, bromodomain, estrogen, epigenetics, chromatin, 3. Good health, Gene Expression Regulation, Neoplastic, Oncology, Histone acetylation, 030220 oncology & carcinogenesis, MCF-7 Cells, Female, metabolism [Nuclear Proteins], Research Paper, Breast Neoplasms, Biology, Chromatin remodeling, Histone H4, 03 medical and health sciences, Sp3 transcription factor, Humans, ddc:610, metabolism [Breast Neoplasms], Transcription factor, estrogen receptor alpha, human, 030304 developmental biology, Cell Proliferation, Binding Sites, epigenetics, Gene Expression Profiling, Pioneer factor, Estrogen Receptor alpha, Computational Biology, Promoter, Estrogens, BRD4 protein, human, Cancer research, chromatin, Estrogen receptor alpha, Transcription Factors
URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::80aff575c69baa93ea0324db546b657f
https://www.research.manchester.ac.uk/portal/en/publications/h4k12ac-is-regulated-by-estrogen -receptoralpha-and-is-associated-with-brd4-function-and-inducible-transcription(dcd37402-d5e6-4a7a-a29c-08c4661e81a9).html -
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المؤلفون: Gambino, Y.P., Maymó, J.L., Pérez-Pérez, A., Dueñas, J.L., Sánchez-Margalet, V., Calvo, J.C., Varone, C.L.
المصدر: Biol. Reprod. 2010;83(1):42-51
Biblioteca Digital (UBA-FCEN)
Universidad Nacional de Buenos Aires. Facultad de Ciencias Exactas y Naturales
instacron:UBA-FCENمصطلحات موضوعية: Leptin, Placenta, cancer cell culture, Western blotting, Phosphatidylinositol 3-Kinases, analogs and derivatives, Pregnancy, phosphatidylinositol 3 kinase, enzyme phosphorylation, Extracellular Signal-Regulated MAP Kinases, Promoter Regions, Genetic, enzyme inhibition, fulvestrant, Estradiol, mitogen activated protein kinase, article, 2 (2 amino 3 methoxyphenyl)chromone, luciferase, reporter gene, wortmannin, female, priority journal, transient transfection, 17beta-estradiol, signal transduction, estrogen receptor, in vitro study, MAP Kinase Signaling System, nidation, embryo, In Vitro Techniques, adipocyte, Bovinae, MAPK signal transduction pathway, promoter region, estrogen receptor alpha, plasmid, Cell Line, Tumor, choriocarcinoma, Humans, controlled study, human, estrogen receptor alpha, human, antagonists and inhibitors, gene deletion, human cell, explant, tumor cell line, Receptor Cross-Talk, trophoblast, human tissue, concentration response, gene expression, protein kinase B, metabolism, Proto-Oncogene Proteins c-akt
وصف الملف: application/pdf