المؤلفون: Michael Berlin, Jennifer Cantley, Mark Bookbinder, Elizabeth Bortolon, Fabio Broccatelli, Greg Cadelina, Emily W. Chan, Huifen Chen, Xin Chen, Yunxing Cheng, Tommy K. Cheung, Kim Davenport, Dean DiNicola, Debbie Gordon, Brian D. Hamman, Alicia Harbin, Roy Haskell, Mingtao He, Alison J. Hole, Thomas Januario, Philip S. Kerry, Stefan G. Koenig, Limei Li, Mark Merchant, Inmaculada Pérez-Dorado, Jennifer Pizzano, Connor Quinn, Christopher M. Rose, Emma Rousseau, Leofal Soto, Leanna R. Staben, Hongming Sun, Qingping Tian, Jing Wang, Weifeng Wang, Crystal S. Ye, Xiaofen Ye, Penghong Zhang, Yuhui Zhou, Robert Yauch, Peter S. Dragovich

مصطلحات موضوعية: Biophysics, Biochemistry, Cell Biology, Genetics, Molecular Biology, Immunology, Cancer, Computational Biology, Chemical Sciences not elsewhere classified, Physical Sciences not elsewhere classified, Information Systems not elsewhere classified, thus highly dependent, pd analysis established, also selectively impair, >) also supported, compounds identified protacs, selectively degraded brm, type calu6 line, h1944 “ brg1, fold degradation selectivity, protacs targeting brm, also known, brm protein, brm (<, vivo <, sw1573 cell, subsequent pk, plau <, mrna associated, molecules exhibit

Relation: https://figshare.com/articles/journal_contribution/PROTACs_Targeting_BRM_SMARCA2_Afford_Selective_i_In_Vivo_i_Degradation_over_BRG1_SMARCA4_and_Are_Active_in_BRG1_Mutant_Xenograft_Tumor_Models/24949319

الاتاحة: https://doi.org/10.1021/acs.jmedchem.3c01781.s001
https://figshare.com/articles/journal_contribution/PROTACs_Targeting_BRM_SMARCA2_Afford_Selective_i_In_Vivo_i_Degradation_over_BRG1_SMARCA4_and_Are_Active_in_BRG1_Mutant_Xenograft_Tumor_Models/24949319

المؤلفون: Michael Berlin, Jennifer Cantley, Mark Bookbinder, Elizabeth Bortolon, Fabio Broccatelli, Greg Cadelina, Emily W. Chan, Huifen Chen, Xin Chen, Yunxing Cheng, Tommy K. Cheung, Kim Davenport, Dean DiNicola, Debbie Gordon, Brian D. Hamman, Alicia Harbin, Roy Haskell, Mingtao He, Alison J. Hole, Thomas Januario, Philip S. Kerry, Stefan G. Koenig, Limei Li, Mark Merchant, Inmaculada Pérez-Dorado, Jennifer Pizzano, Connor Quinn, Christopher M. Rose, Emma Rousseau, Leofal Soto, Leanna R. Staben, Hongming Sun, Qingping Tian, Jing Wang, Weifeng Wang, Crystal S. Ye, Xiaofen Ye, Penghong Zhang, Yuhui Zhou, Robert Yauch, Peter S. Dragovich

مصطلحات موضوعية: Biophysics, Biochemistry, Cell Biology, Genetics, Molecular Biology, Immunology, Cancer, Computational Biology, Chemical Sciences not elsewhere classified, Physical Sciences not elsewhere classified, Information Systems not elsewhere classified, thus highly dependent, pd analysis established, also selectively impair, >) also supported, compounds identified protacs, selectively degraded brm, type calu6 line, h1944 “ brg1, fold degradation selectivity, protacs targeting brm, also known, brm protein, brm (<, vivo <, sw1573 cell, subsequent pk, plau <, mrna associated, molecules exhibit

Relation: https://figshare.com/articles/dataset/PROTACs_Targeting_BRM_SMARCA2_Afford_Selective_i_In_Vivo_i_Degradation_over_BRG1_SMARCA4_and_Are_Active_in_BRG1_Mutant_Xenograft_Tumor_Models/24949322

الاتاحة: https://doi.org/10.1021/acs.jmedchem.3c01781.s002
https://figshare.com/articles/dataset/PROTACs_Targeting_BRM_SMARCA2_Afford_Selective_i_In_Vivo_i_Degradation_over_BRG1_SMARCA4_and_Are_Active_in_BRG1_Mutant_Xenograft_Tumor_Models/24949322