المؤلفون: Annie J. Lee, Neha S. Raghavan, Prabesh Bhattarai, Tohid Siddiqui, Sanjeev Sariya, Dolly Reyes-Dumeyer, Xena E. Flowers, Sarah A. L. Cardoso, Philip L. De Jager, David A. Bennett, Julie A. Schneider, Vilas Menon, Yanling Wang, Rafael A. Lantigua, Martin Medrano, Diones Rivera, Ivonne Z. Jiménez-Velázquez, Walter A. Kukull, Adam M. Brickman, Jennifer J. Manly, Giuseppe Tosto, Caghan Kizil, Badri N. Vardarajan, Richard Mayeux

المصدر: Acta neuropathologica 144, 59-79 (2022). doi:10.1007/s00401-022-02431-6

مصطلحات موضوعية: Gliovascular interaction, Cerebrovascular risk factors, Mouse, metabolism [Amyloid beta-Peptides], Formins, FMNL2, Mice, Transgenic, Pathology and Forensic Medicine, Amyloid beta-Protein Precursor, pathology [Alzheimer Disease], Mice, Cellular and Molecular Neuroscience, Alzheimer Disease, pathology [Brain], Risk Factors, Blood–brain-barrier, metabolism [Amyloid beta-Protein Precursor], Animals, Humans, GWAS, ddc:610, metabolism [Zebrafish], Zebrafish, Amyloid beta-Peptides, Brain, Amyloidosis, complications [Amyloidosis], Disease Models, Animal, genetics [Amyloid beta-Protein Precursor], Neurovascular unit, Neurology (clinical), Alzheimer’s disease, Human

URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::6ffdebddbbbc62cfa5b9b39c56e04f88
https://doi.org/10.1007/s00401-022-02431-6