المؤلفون: Elisabetta Bonfanti, Tiziana Bonifacino, Stefano Raffaele, Marco Milanese, Erica Morgante, Giambattista Bonanno, Maria P. Abbracchio, Marta Fumagalli

المصدر: International Journal of Molecular Sciences; Volume 21; Issue 7; Pages: 2395

مصطلحات موضوعية: G protein-coupled receptor 17 (GPR17), amyotrophic lateral sclerosis (ALS), SOD1 G93A ALS mouse model, oligodendrocytes, montelukast

جغرافية الموضوع: agris

وصف الملف: application/pdf

Relation: Biochemistry; https://dx.doi.org/10.3390/ijms21072395

الاتاحة: https://doi.org/10.3390/ijms21072395

المؤلفون: Valbuena, G, Keun, H, Bendotti, C, Cantoni, L, Tortarolo, M

المساهمون: Commission of the European Communities

المصدر: 16 ; 1

مصطلحات موضوعية: Science & Technology, Life Sciences & Biomedicine, Neurosciences, Neurosciences & Neurology, amyotrophic lateral sclerosis (ALS), SOD1(G93A) ALS mouse model, metabolism, metabolomics, spinal cord, oxidative stress, TCA cycle, energy metabolism, TRANSGENIC MOUSE MODEL, MOTOR-NEURONS, SOD1 MOUSE, PHENOTYPIC HETEROGENEITY, DISEASE PROGRESSION, ALS, DYSFUNCTION, TOXICITY, INOSITOL, GENES, SOD1G93A ALS mouse model, 1109 Neurosciences, 1701 Psychology, 1702 Cognitive Sciences

Relation: Frontiers in Neuroscience; http://hdl.handle.net/10044/1/74914; 259867

الاتاحة: http://hdl.handle.net/10044/1/74914
https://doi.org/10.3389/fnins.2019.01276

المؤلفون: BA Mir, SA Mason, AK May, Aaron Russell, VC Foletta

مصطلحات موضوعية: Animal physiology - biophysics, Animal physiology - cell, Animal physiology - systems, Medical physiology not elsewhere classified, Sports science and exercise not elsewhere classified, Science & Technology, Life Sciences & Biomedicine, Physiology, fasting, N-myc downstream-regulated gene 2, NDRG2, skeletal muscle, SOD1(G93A)mice, stress response, AMYOTROPHIC-LATERAL-SCLEROSIS, UBIQUITIN-PROTEASOME PATHWAY, MOUSE MODEL, MYOBLAST PROLIFERATION, ALZHEIMERS-DISEASE, SOD1(G93A) ALS, P38 MAPK, AUTOPHAGY, PROTEIN, GENE, 3208 Medical physiology

Relation: http://hdl.handle.net/10536/DRO/DU:30138862; https://figshare.com/articles/journal_contribution/Overexpression_of_NDRG2_in_skeletal_muscle_does_not_ameliorate_the_effects_of_stress_in_vivo/20702527

الاتاحة: http://hdl.handle.net/10536/DRO/DU:30138862
https://figshare.com/articles/journal_contribution/Overexpression_of_NDRG2_in_skeletal_muscle_does_not_ameliorate_the_effects_of_stress_in_vivo/20702527

المؤلفون: Lavinia Cantoni, Gabriel N. Valbuena, Caterina Bendotti, Hector C. Keun, Massimo Tortarolo

المساهمون: Commission of the European Communities

المصدر: Frontiers in Neuroscience, Vol 13 (2019)
Frontiers in Neuroscience

مصطلحات موضوعية: 0301 basic medicine, amyotrophic lateral sclerosis (ALS), 1702 Cognitive Sciences, Disease, Bioinformatics, medicine.disease_cause, TOXICITY, Energy homeostasis, 0302 clinical medicine, energy metabolism, oxidative stress, Amyotrophic lateral sclerosis, Original Research, General Neuroscience, metabolomics, SOD1G93A ALS mouse model, medicine.anatomical_structure, Life Sciences & Biomedicine, GENES, lcsh:RC321-571, 03 medical and health sciences, PHENOTYPIC HETEROGENEITY, Metabolomics, INOSITOL, Metabolome, medicine, TRANSGENIC MOUSE MODEL, TCA cycle, lcsh:Neurosciences. Biological psychiatry. Neuropsychiatry, SOD1(G93A) ALS mouse model, Science & Technology, business.industry, DISEASE PROGRESSION, Neurosciences, spinal cord, Spinal cord, medicine.disease, DYSFUNCTION, Lumbar Spinal Cord, 030104 developmental biology, 1701 Psychology, MOTOR-NEURONS, Neurosciences & Neurology, ALS, 1109 Neurosciences, business, metabolism, 030217 neurology & neurosurgery, Oxidative stress, SOD1 MOUSE, Neuroscience

URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f333f14b1278fc9ba1cbaf85009eb97a
https://www.frontiersin.org/article/10.3389/fnins.2019.01276/full

المؤلفون: Mir, Bilal A., Mason, Shaun A., May, Anthony K., Russell, Aaron P., Foletta, Victoria C.

مصطلحات الفهرس: Science & Technology, Life Sciences & Biomedicine, Physiology, fasting, N-myc downstream-regulated gene 2, NDRG2, skeletal muscle, SOD1(G93A)mice, stress response, AMYOTROPHIC-LATERAL-SCLEROSIS, UBIQUITIN-PROTEASOME PATHWAY, MOUSE MODEL, MYOBLAST PROLIFERATION, ALZHEIMERS-DISEASE, SOD1(G93A) ALS, P38 MAPK, AUTOPHAGY, PROTEIN, GENE, SOD1G93A mice, Journal Article

URL: http://hdl.handle.net/10536/DRO/DU:30138862
https://dro.deakin.edu.au/eserv/DU:30138862/may-overexpressionofndrg-post-2020.pdf
http://doi.org/10.1113/EP088620
issn: 0958-0670
issn: 1469-445X
https://dro.deakin.edu.au/eserv/DU:30138862/may-overexpressionofndrg-post-2020.pdf
http://doi.org/10.1113/EP088620
Experimental Physiology