يعرض 1 - 20 نتائج من 20 نتيجة بحث عن '"Ramanand, Susmita G."', وقت الاستعلام: 0.50s تنقيح النتائج
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    المصدر: Nature Medicine. 25(10)

    وصف الملف: application/pdf

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    المؤلفون: Ramanand, Susmita G., Mani, Ram S.

    المساهمون: National Cancer Institute, Cancer Prevention and Research Institute of Texas, National Institutes of Health, U.S. Department of Defense

    المصدر: Molecular Cell ; volume 82, issue 15, page 2730-2731 ; ISSN 1097-2765

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    المساهمون: UT Southwestern Live Cell Imaging Facility, Shared Resource of the Harold C. Simmons Cancer Center, NCI Cancer Center, NIH Pathway to Independence (PI) Award, UT Southwestern, ACS-IRG New Investigator Award in Cancer Research, Prostate Cancer Research Program (PCRP) – Impact Award – US Department of Defense, Prostate Cancer UK, Prostate Cancer Foundation, Movember, Stand Up To Cancer, US Department of Defense, Cancer Research UK, UK Department of Health, Academy of Medical Sciences, NHS, NIHR Biomedical Research Centre at the Royal Marsden, The Institute of Cancer Research, Medical Research Council fellowship, NIH, National Aeronautics and Space Administration, CPRIT, Welch Foundation

    المصدر: Cell Reports ; volume 22, issue 3, page 796-808 ; ISSN 2211-1247

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    المساهمون: NIH Pathway to Independence (PI) Award, UT Southwestern, ACS-IRG New Investigator Award in Cancer Research, Stewart Rahr-Prostate Cancer Foundation Young Investigator Award, NIH Prostate SPORE, Early Detection Research Network, Prostate Cancer Foundation

    المصدر: Cell Reports ; volume 17, issue 10, page 2620-2631 ; ISSN 2211-1247

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    Book

    المؤلفون: Ramanand, Susmita G., Mani, Ram S.

    المصدر: Advances in Experimental Medicine and Biology ; Prostate Cancer ; page 57-66 ; ISSN 0065-2598 2214-8019 ; ISBN 9783030326555 9783030326562

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    المصدر: Nature medicine, vol 25, iss 10

    وصف الملف: application/pdf

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    المساهمون: Internal Medicine, Division of Hematology–Oncology, University of Michigan Medical Center, Ann Arbor, Michigan, Department of Oral Medicine/Pathology and Oncology, School of Dentistry, University of Michigan, Ann Arbor, Michigan, University of Michigan Medical Center, Department of Radiation Oncology, 1500 E. Medical Center Drive, Ann Arbor, MI 48109, Department of Radiation Oncology, University of Michigan Medical Center, Ann Arbor, Michigan, Department of Otolaryngology, University of Michigan Medical Center, Ann Arbor, Michigan, Oral and Maxillofacial Surgery, University of Michigan Medical Center, Ann Arbor, Michigan

    وصف الملف: application/pdf

    Relation: Tsien, Christina I.; Nyati, Mukesh K.; Ahsan, Aarif; Ramanand, Susmita G.; Chepeha, Douglas B.; Worden, Francis P.; Helman, Joseph I.; D'Silva, Nisha; Bradford, Carol R.; Wolf, Gregory T.; Lawrence, Theodore S.; Eisbruch, Avraham (2013). "Effect of erlotinib on epidermal growth factor receptor and downstream signaling in oral cavity squamous cell carcinoma ." Head & Neck 35(9): 1323-1330.; https://hdl.handle.net/2027.42/99603; Head & Neck; Ahsan A, Hiniker SM, Davis MA, Lawrence TS, Nyati MK. Role of cell cycle in epidermal growth factor receptor inhibitor‐mediated radiosensitization. Cancer Res 2009; 69: 5108 – 5114.; Ciardiello F, Tortora G. Epidermal growth factor receptor (EGFR) as a target in cancer therapy: understanding the role of receptor expression and other molecular determinants that could influence the response to anti‐EGFR drugs. Eur J Cancer 2003; 39: 1348 – 1354.; Scartozzi M, Bearzi I, Berardi R, Mandolesi A, Fabris G, Cascinu S. Epidermal growth factor receptor (EGFR) status in primary colorectal tumors does not correlate with EGFR expression in related metastatic sites: implications for treatment with EGFR‐targeted monoclonal antibodies. J Clin Oncol 2004; 22: 4772 – 4778.; Parra HS, Cavina R, Latteri F, et al. Analysis of epidermal growth factor receptor expression as a predictive factor for response to gefitinib (‘Iressa’, ZD1839) in non‐small‐cell lung cancer. Br J Cancer 2004; 91: 208 – 212.; Brugger W, Triller N, Blasinska–Morawiec M, et al. Prospective molecular marker analyses of EGFR and KRAS from a randomized, placebo‐controlled study of erlotinib maintenance therapy in advanced non‐small‐cell lung cancer. J Clin Oncol 2011; 29: 4113 – 4120.; Shigematsu H, Lin L, Takahashi T, et al. Clinical and biological features associated with epidermal growth factor receptor gene mutations in lung cancers. J Natl Cancer Inst 2005; 97: 339 – 346.; Cappuzzo F, Hirsch FR, Rossi E, et al. Epidermal growth factor receptor gene and protein and gefitinib sensitivity in non‐small‐cell lung cancer. J Natl Cancer Inst 2005; 97: 643 – 655.; Paez JG, Jänne PA, Lee JC, et al. EGFR mutations in lung cancer: correlation with clinical response to gefitinib therapy. Science 2004; 304: 1497 – 1500.; Szabó B, Nelhubel GA, Kárpáti A, et al. Clinical significance of genetic alterations and expression of epidermal growth factor receptor (EGFR) in head and neck squamous cell carcinomas. Oral Oncol 2011; 47: 487 – 496.; Sheikh Ali MA, Gunduz M, Nagatsuka H, et al. Expression and mutation analysis of epidermal growth factor receptor in head and neck squamous cell carcinoma. Cancer Sci 2008; 99: 1589 – 1594.; Lee JW, Soung YH, Kim SY, et al. Somatic mutations of EGFR gene in squamous cell carcinoma of the head and neck. Clin Cancer Res 2005; 11: 2879 – 2882.; Lemos–González Y, Páez de la Cadena MP, Rodríguez–Berrocal FJ, Rodríguez–Piñeiro AM, Pallas E, Valverde D. Absence of activating mutations in the EGFR kinase domain in Spanish head and neck cancer patients. Tumour Biol 2007; 28: 273 – 279.; Chung CH, Ely K, McGavran L, et al. Increased epidermal growth factor receptor gene copy number is associated with poor prognosis in head and neck squamous cell carcinomas. J Clin Oncol 2006; 24: 4170 – 4176.; Sok JC, Coppelli FM, Thomas SM, et al. Mutant epidermal growth factor receptor (EGFRvIII) contributes to head and neck cancer growth and resistance to EGFR targeting. Clin Cancer Res 2006; 12: 5064 – 5073.; Fukuoka M, Wu YL, Thongprasert S, et al. Biomarker analyses and final overall survival results from a phase III, randomized, open‐label, first‐line study of gefitinib versus carboplatin/paclitaxel in clinically selected patients with advanced non‐small‐cell lung cancer in Asia (IPASS). J Clin Oncol 2011; 29: 2866 – 2874.; Feng FY, Lopez CA, Normolle DP, et al. Effect of epidermal growth factor receptor inhibitor class in the treatment of head and neck cancer with concurrent radiochemotherapy in vivo. Clin Cancer Res 2007; 13: 2512 – 2518.; Nyati MK, Lawrence TS. Hitting the target: measuring EGFR response to tyrosine kinase inhibitors. Cancer Biol Ther 2005; 4: 1387 – 1388.; Nyati MK, Maheshwari D, Hanasoge S, et al. Radiosensitization by pan ErbB inhibitor CI‐1033 in vitro and in vivo. Clin Cancer Res 2004; 10: 691 – 700.; Ray D, Ahsan A, Helman A, et al. Regulation of EGFR protein stability by the HECT‐type ubiquitin ligase SMURF2. Neoplasia 2011; 13: 570 – 578.; Argiris A, Duffy AG, Kummar S, et al. Early tumor progression associated with enhanced EGFR signaling with bortezomib, cetuximab, and radiotherapy for head and neck cancer. Clin Cancer Res 2011; 17: 5755 – 5764.; Ahsan A, Hiniker SM, Ramanand SG, et al. Role of epidermal growth factor receptor degradation in cisplatin‐induced cytotoxicity in head and neck cancer. Cancer Res 2010; 70: 2862 – 2869.; Feng FY, Varambally S, Tomlins SA, et al. Role of epidermal growth factor receptor degradation in gemcitabine‐mediated cytotoxicity. Oncogene 2007; 26: 3431 – 3439.; Weihua Z, Tsan R, Huang WC, et al. Survival of cancer cells is maintained by EGFR independent of its kinase activity. Cancer Cell 2008; 13: 385 – 393.; Rosenthal DI, Lewin JS, Eisbruch A. Prevention and treatment of dysphagia and aspiration after chemoradiation for head and neck cancer. J Clin Oncol 2006; 24: 2636 – 2643.; Eisbruch A, Lyden T, Bradford CR, et al. Objective assessment of swallowing dysfunction and aspiration after radiation concurrent with chemotherapy for head‐and‐neck cancer. Int J Radiat Oncol Biol Phys 2002; 53: 23 – 28.; Nyati MK, Feng FY, Maheshwari D, et al. Ataxia telangiectasia mutated down‐regulates phospho‐extracellular signal‐regulated kinase 1/2 via activation of MKP‐1 in response to radiation. Cancer Res 2006; 66: 11554 – 11559.; Toyoshima H, Hunter T. p27, a novel inhibitor of G1 cyclin‐Cdk protein‐kinase activity, is related to p21. Cell 1994; 78: 67 – 74.; Bonner JA, Harari PM, Giralt J, et al. Radiotherapy plus cetuximab for locoregionally advanced head and neck cancer: 5‐year survival data from a phase 3 randomised trial, and relation between cetuximab‐induced rash and survival. Lancet Oncol 2010; 11: 21 – 28.; Cooper JS, Pajak TF, Forastiere AA, et al. Postoperative concurrent radiotherapy and chemotherapy for high‐risk squamous‐cell carcinoma of the head and neck. N Engl J Med 2004; 350: 1937 – 1944.; Dittmann K, Mayer C, Rodemann HP. Inhibition of radiation‐induced EGFR nuclear import by C225 (cetuximab) suppresses DNA‐PK activity. Radiother Oncol 2005; 76: 157 – 161.; Ang KK, Zhang QE, Rosenthal DI. A randomized phase III trial (RTOG 0522) of concurrent accelerated radiation plus cisplatin with or without cetuximab for stage III–IV head and neck squamous cell carcinomas (HNC). J Clin Oncol 2011; 29 ( Suppl ):Abstract 5500.; Wheeler DL, Huang S, Kruser TJ, et al. Mechanisms of acquired resistance to cetuximab: role of HER (ErbB) family members. Oncogene 2008; 27: 3944 – 3956.; Thomas F, Rochaix P, Benlyazid A, et al. Pilot study of neoadjuvant treatment with erlotinib in nonmetastatic head and neck squamous cell carcinoma. Clin Cancer Res 2007; 13: 7086 – 7092.; Agulnik M, da Cunha Santos G, Hedley D, et al. Predictive and pharmacodynamic biomarker studies in tumor and skin tissue samples of patients with recurrent or metastatic squamous cell carcinoma of the head and neck treated with erlotinib. J Clin Oncol 2007; 25: 2184 – 2190.; Bentzen SM, Atasoy BM, Daley FM, et al. Epidermal growth factor receptor expression in pretreatment biopsies from head and neck squamous cell carcinoma as a predictive factor for a benefit from accelerated radiation therapy in a randomized controlled trial. J Clin Oncol 2005; 23: 5560 – 5567.; Van Waes C, Allen CT, Citrin D, et al. Molecular and clinical responses in a pilot study of gefitinib with paclitaxel and radiation in locally advanced head‐and‐neck cancer. Int J Radiat Oncol Biol Phys 2010; 77: 447 – 454.; Leemans CR, Braakhuis BJ, Brakenhoff RH. The molecular biology of head and neck cancer. Nat Rev Cancer 2011; 11: 9 – 22.; Perez–Ordoñez B, Beauchemin M, Jordan RC. Molecular biology of squamous cell carcinoma of the head and neck. J Clin Pathol 2006; 59: 445 – 453.; O‐charoenrat P, Rhys–Evans PH, Modjtahedi H, Eccles SA. The role of c‐erbB receptors and ligands in head and neck squamous cell carcinoma. Oral Oncol 2002; 38: 627 – 640.; Khademi B, Shirazi FM, Vasei M, et al. The expression of p53, c‐erbB‐1 and c‐erbB‐2 molecules and their correlation with prognostic markers in patients with head and neck tumors. Cancer Lett 2002; 184: 223 – 230.

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