المؤلفون: Damo Xu, José C. Alves-Filho, Foo Y. Liew, Fabiola Reis Oliveira, Ana Tereza Gomes Guerrero, Thiago M. Cunha, Fabricio O. Souto, Sandra Y. Fukada, Silvio M. Vieira, Paulo Louzada-Junior, Sérgio H. Ferreira, Rafaela B Mattos-Guimaraes, Mauro M. Teixeira, João Santana da Silva, Fernando Q. Cunha, Waldiceu A. Verri, Iain B. McInnes, Sérgio C. L. Almeida

المصدر: Annals of the Rheumatic Diseases. 69:1697-1703

مصطلحات موضوعية: Chemokine, Immunology, Arthritis, Inflammation, General Biochemistry, Genetics and Molecular Biology, Arthritis, Rheumatoid, Mice, Rheumatology, medicine, Animals, Humans, Immunology and Allergy, RNA, Messenger, Mice, Inbred BALB C, Chemotactic Factors, biology, Tumor Necrosis Factor-alpha, business.industry, Interleukins, Synovial Membrane, Cell migration, Receptors, Interleukin, Neutrophil extracellular traps, Macrophage Activation, Interleukin-33, medicine.disease, Arthritis, Experimental, Interleukin-1 Receptor-Like 1 Protein, Mice, Inbred C57BL, CXCL1, Interleukin 33, Chemotaxis, Leukocyte, Gene Expression Regulation, Neutrophil Infiltration, Antirheumatic Agents, biology.protein, Cytokines, Tumor necrosis factor alpha, medicine.symptom, business

URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e2bf3bbcad7f0c1b4d90b02197381f98
https://doi.org/10.1136/ard.2009.122655