المؤلفون: Nathan A. Truchan (10980474), Rachel J. Fenske (10980477), Harpreet K. Sandhu (10980480), Alicia M. Weeks (10980483), Chinmai Patibandla (10980486), Benjamin Wancewicz (3815842), Samantha Pabich (10980489), Austin Reuter (10980492), Jeffrey M. Harrington (10980495), Allison L. Brill (10980498), Darby C. Peter (10980501), Randall Nall (10980504), Michael Daniels (10980507), Margaret Punt (10980510), Cecilia E. Kaiser (10980513), Elizabeth D. Cox (9525800), Ying Ge (175278), Dawn B. Davis (9525803), Michelle E. Kimple (9592664)

مصطلحات موضوعية: Biochemistry, Genetics, Molecular Biology, Neuroscience, Physiology, Immunology, Developmental Biology, Chemical Sciences not elsewhere classified, upregulated islet PGE 2 production, plasma arachidonic acid, Prostaglandin E 2, islet COX -2 expression, obesity, Islet insulin content, arachidonic acid metabolite, BMI, PGE 2 metabolite levels, Insulin secretion assays, PGE 2 production, type 2 diabetes, prostaglandin E 2 receptor subtype EP 3, EP 3 expression, β- cell dysfunction, T 2D, IL -6 expression, EP 3-specific antagonist, donor body mass index, β- cell adaption response, Human Islet Expression Levels, pathway influences nondiabetic β- c.

Relation: https://figshare.com/articles/journal_contribution/Human_Islet_Expression_Levels_of_Prostaglandin_E_sub_2_sub_Synthetic_Enzymes_But_Not_Prostaglandin_EP3_Receptor_Are_Positively_Correlated_with_Markers_of_Cell_Function_and_Mass_in_Nondiabetic_Obesity/14791794

الاتاحة: https://doi.org/10.1021/acsptsci.1c00045.s001