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المؤلفون: Rannikko, Emmy H., Weber, Stephanie S., Kahle, Philipp J.
المصدر: BMC neuroscience 16(1), 57 (2015). doi:10.1186/s12868-015-0192-0
BMC Neuroscienceمصطلحات موضوعية: metabolism [Recombinant Proteins], metabolism [Toll-Like Receptor 4], Nitric Oxide Synthase Type II, physiology [Active Transport, Cell Nucleus], p38 Mitogen-Activated Protein Kinases, NF-κB, Ptgs2 protein, mouse, Neuroinflammation, Phosphorylation, Cells, Cultured, Mice, Knockout, Brain, Endocytosis, Recombinant Proteins, Cyclooxygenase, genetics [Recombinant Proteins], MAP kinases, genetics [alpha-Synuclein], alpha-Synuclein, Cytokines, immunology [Brain], Research Article, Nos2 protein, mouse, metabolism [p38 Mitogen-Activated Protein Kinases], Toll-like receptor TLR4, Active Transport, Cell Nucleus, Mice, Transgenic, immunology [Astrocytes], toxicity [alpha-Synuclein], metabolism [RNA, Messenger], metabolism [Cyclooxygenase 2], Cellular and Molecular Neuroscience, Tlr4 protein, mouse, Escherichia coli, metabolism [Extracellular Space], Animals, Humans, ddc:610, RNA, Messenger, SNCA protein, human, Synuclein, Nitric oxide synthase, Transcription Factor RelA, JNK Mitogen-Activated Protein Kinases, Rela protein, mouse, genetics [Toll-Like Receptor 4], metabolism [Nitric Oxide Synthase Type II], Toll-Like Receptor 4, Cyclooxygenase 2, Astrocytes, metabolism [JNK Mitogen-Activated Protein Kinases], Extracellular Space, metabolism [Transcription Factor RelA]