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1Academic Journal
المؤلفون: Tan, Qiumin, Brunetti, Lorenzo, Rousseaux, Maxime W. C., Lu, Hsiang-Chih, Wan, Ying-Wooi, Revelli, Jean-Pierre, Liu, Zhandong, Goodell, Margaret A., Zoghbi, Huda Y.
المصدر: Proceedings of the National Academy of Sciences of the United States of America, 2018 Feb . 115(7), E1511-E1519.
URL الوصول: https://www.jstor.org/stable/26507387
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2Academic Journal
المؤلفون: Tan Q., Brunetti L., Rousseaux M. W. C., Lu H. -C., Wan Y. -W., Revelli J. -P., Liu Z., Goodell M. A., Zoghbi H. Y.
المساهمون: Tan, Q., Brunetti, L., Rousseaux, M. W. C., Lu, H. -C., Wan, Y. -W., Revelli, J. -P., Liu, Z., Goodell, M. A., Zoghbi, H. Y.
مصطلحات موضوعية: Capicua, MYC transcriptional program, NOTCH activation, T cell development, T-ALL, Animal, Cells, Cultured, Mice, Knockout, Mutation, Precursor T-Cell Lymphoblastic Leukemia-Lymphoma, Proto-Oncogene Proteins c-myc, Receptor, Notch1, Repressor Protein, Signal Transduction, T-Lymphocyte, ras Protein, Cell Differentiation, Disease Susceptibility
وصف الملف: STAMPA
Relation: info:eu-repo/semantics/altIdentifier/pmid/29382756; info:eu-repo/semantics/altIdentifier/wos/WOS:000424876000024; volume:115; issue:7; firstpage:E1511; lastpage:E1519; journal:PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA; http://hdl.handle.net/11391/1463178; info:eu-repo/semantics/altIdentifier/scopus/2-s2.0-85041998348; https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5816173/
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المؤلفون: Maxime W.C. Rousseaux, Qiumin Tan, Jean-Pierre Revelli, Zhandong Liu, Margaret A. Goodell, Lorenzo Brunetti, Huda Y. Zoghbi, Ying-Wooi Wan, Hsiang Chih Lu
مصطلحات موضوعية: 0301 basic medicine, Spinocerebellar Ataxia Type 1, Lymphoblastic Leukemia, T cell, Cells, Knockout, T-Lymphocytes, Biology, medicine.disease_cause, Precursor T-Cell Lymphoblastic Leukemia-Lymphoma, law.invention, Proto-Oncogene Proteins c-myc, 03 medical and health sciences, Mice, law, medicine, Animals, Capicua, Receptor, Notch1, Cells, Cultured, Mice, Knockout, Notch1, Multidisciplinary, NOTCH activation, Cultured, T cell development, MYC transcriptional program, Cell Differentiation, medicine.disease, Embryonic stem cell, T-ALL, Mutation, Repressor Proteins, Signal Transduction, ras Proteins, Disease Susceptibility, Lymphoma, Haematopoiesis, 030104 developmental biology, medicine.anatomical_structure, PNAS Plus, Cancer research, Suppressor, KRAS, Receptor