نتائج البحث - "Koh, Andrew S."

1

Academic Journal

PU.1 and BCL11B sequentially cooperate with RUNX1 to anchor mSWI/SNF to poise the T cell effector landscape.

المؤلفون: Gamble, Noah, Bradu, Alexandra, Caldwell, Jason A, McKeever, Joshua, Bolonduro, Olubusayo, Ermis, Ebru, Kaiser, Caroline, Kim, YeEun, Parks, Benjamin, Klemm, Sandy, Greenleaf, William J, Crabtree, Gerald R, Koh, Andrew S

المصدر: Nat Immunol ; ISSN:1529-2916 ; Volume:25 ; Issue:5

Relation: https://doi.org/10.1038/s41590-024-01807-y; https://pubmed.ncbi.nlm.nih.gov/38632339; https://www.ncbi.nlm.nih.gov/pmc/articles/PMC11089574/

الاتاحة: https://doi.org/10.1038/s41590-024-01807-y
https://pubmed.ncbi.nlm.nih.gov/38632339
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC11089574/

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2

Academic Journal

Publisher Correction: PU.1 and BCL11B sequentially cooperate with RUNX1 to anchor mSWI/SNF to poise the T cell effector landscape

المؤلفون: Gamble, Noah, Bradu, Alexandra, Caldwell, Jason A., McKeever, Joshua, Bolonduro, Olubusayo, Ermis, Ebru, Kaiser, Caroline, Kim, YeEun, Parks, Benjamin, Klemm, Sandy, Greenleaf, William J., Crabtree, Gerald R., Koh, Andrew S.

المصدر: Nature Immunology ; volume 25, issue 6, page 1123-1123 ; ISSN 1529-2908 1529-2916

الاتاحة: https://doi.org/10.1038/s41590-024-01864-3
https://www.nature.com/articles/s41590-024-01864-3.pdf
https://www.nature.com/articles/s41590-024-01864-3

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3

Academic Journal

Venetoclax Induces BCL-2-Dependent Treg to TH17 Plasticity to Enhance the Antitumor Efficacy of Anti-PD-1 Checkpoint Blockade.

المؤلفون: Liao, Rosy, Hsu, Jocelyn Y, Aboelella, Nada S, McKeever, Joshua A, Thomas-Toth, Anika T, Koh, Andrew S, LaBelle, James L

المصدر: Cancer Immunol Res ; ISSN:2326-6074 ; Volume:12 ; Issue:8

Relation: https://doi.org/10.1158/2326-6066.CIR-23-0344; https://pubmed.ncbi.nlm.nih.gov/38810242; https://www.ncbi.nlm.nih.gov/pmc/articles/PMC11293981/

الاتاحة: https://doi.org/10.1158/2326-6066.CIR-23-0344
https://pubmed.ncbi.nlm.nih.gov/38810242
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC11293981/

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4

Academic Journal

Global relevance of Aire binding to hypomethylated lysine-4 of histone-3

المؤلفون: Koh, Andrew S., Kingston, Robert E., Benoist, Christophe, Mathis, Diane

المصدر: Proceedings of the National Academy of Sciences of the United States of America, 2010 Jul . 107(29), 13016-13021.

URL الوصول: https://www.jstor.org/stable/25708656

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5

Academic Journal

Aire Employs a Histone-Binding Module to Mediate Immunological Tolerance, Linking Chromatin Regulation with Organ-Specific Autoimmunity

المؤلفون: Koh, Andrew S., Kuo, Alex J., Park, Sang Youn, Cheung, Peggie, Abramson, Jakub, Bua, Dennis, Carney, Dylan, Shoelson, Steven E., Gozani, Or, Kingston, Robert E., Benoist, Christophe, Mathis, Diane

المصدر: Proceedings of the National Academy of Sciences of the United States of America, 2008 Oct . 105(41), 15878-15883.

URL الوصول: https://www.jstor.org/stable/25464509

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6

Academic Journal

AIRE in context: Leveraging chromatin plasticity to trigger ectopic gene expression*

المؤلفون: Kaiser, Caroline, Bradu, Alexandra, Gamble, Noah, Caldwell, Jason A., Koh, Andrew S.

المساهمون: National Institute of General Medical Sciences

المصدر: Immunological Reviews ; volume 305, issue 1, page 59-76 ; ISSN 0105-2896 1600-065X

الاتاحة: http://dx.doi.org/10.1111/imr.13026
https://onlinelibrary.wiley.com/doi/pdf/10.1111/imr.13026
https://onlinelibrary.wiley.com/doi/full-xml/10.1111/imr.13026

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7

Academic Journal

AIRE in context: Leveraging chromatin plasticity to trigger ectopic gene expression*.

المؤلفون: Kaiser, Caroline, Bradu, Alexandra, Gamble, Noah, Caldwell, Jason A., Koh, Andrew S.

المصدر: Immunological Reviews; Jan2022, Vol. 305 Issue 1, p59-76, 18p

مصطلحات موضوعية: CHROMATIN-remodeling complexes, CHROMATIN, ANTIGEN receptors, T cells, EPITHELIAL cells, GENES

8

Academic Journal

Rapid chromatin repression by Aire provides precise control of immune tolerance

المؤلفون: Koh, Andrew S., Miller, Erik L., Buenrostro, Jason D., Moskowitz, David M., Wang, Jing, Greenleaf, William J., Chang, Howard Y., Crabtree, Gerald R.

المصدر: Nature Immunology ; volume 19, issue 2, page 162-172 ; ISSN 1529-2908 1529-2916

الاتاحة: http://dx.doi.org/10.1038/s41590-017-0032-8
http://www.nature.com/articles/s41590-017-0032-8
http://www.nature.com/articles/s41590-017-0032-8.pdf

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9

Academic Journal

The CD8+ T cell tolerance checkpoint triggers a distinct differentiation state defined by protein translation defects.

المؤلفون: Van Der Byl, Willem^1,2 (AUTHOR), Nüssing, Simone^1,3,4 (AUTHOR), Peters, Timothy J.^1,5,6 (AUTHOR), Ahn, Antonio^3,4 (AUTHOR), Li, Hanjie⁷ (AUTHOR), Ledergor, Guy⁷ (AUTHOR), David, Eyal⁷ (AUTHOR), Koh, Andrew S.⁸ (AUTHOR), Wagle, Mayura V.^5,9 (AUTHOR), Deguit, Christian Deo T.³ (AUTHOR), de Menezes, Maria N.^3,4 (AUTHOR), Travers, Avraham³ (AUTHOR), Sampurno, Shienny³ (AUTHOR), Ramsbottom, Kelly M.³ (AUTHOR), Li, Rui¹⁰ (AUTHOR), Kallies, Axel^11,12 (AUTHOR), Beavis, Paul A.^3,4 (AUTHOR), Jungmann, Ralf^13,14 (AUTHOR), Bastings, Maartje M.C.^15,16 (AUTHOR), Belz, Gabrielle T.^17,18 (AUTHOR)

المصدر: Immunity (10747613). Jun2024, Vol. 57 Issue 6, p1324-1324. 1p.

مصطلحات موضوعية: *T cells, *GENETIC translation, *T cell receptors, *T cell differentiation, *GENE regulatory networks, *CD8 antigen, *RECOGNITION (Psychology)

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