المؤلفون: H-G. Knaus, Hans Zischka, Nikolaus Plesnila, Matthias Höllerhage, Carsten Culmsee, Günter U. Höglinger, A de Andrade, Lilja Meissner, Amalia M. Dolga, P Christophersen

المصدر: Cell Death & Disease
Cell death & disease 5(1), e999-e999 (2014). doi:10.1038/cddis.2013.530

مصطلحات موضوعية: Cancer Research, Small-Conductance Calcium-Activated Potassium Channels, KCNN3 protein, human, Parkinson's disease, Immunology, Excitotoxicity, metabolism [Parkinson Disease], KCNN2 protein, human, Pharmacology, Biology, medicine.disease_cause, Neuroprotection, SK channel, Cellular and Molecular Neuroscience, SK3, genetics [Parkinson Disease], Dopaminergic Cell, ddc:570, metabolism [Mitochondrial Membranes], medicine, Humans, metabolism [Calcium], metabolism [Small-Conductance Calcium-Activated Potassium Channels], KCNN1 protein, human, Membrane potential, cytology [Dopaminergic Neurons], Membrane Potential, Mitochondrial, Dopaminergic Neurons, Dopaminergic, metabolism [Dopaminergic Neurons], Cell Differentiation, Parkinson Disease, Cell Biology, pre-conditioning, Potassium channel, mitochondria, Protein Transport, NS309, Neuroprotective Agents, genetics [Small-Conductance Calcium-Activated Potassium Channels], Mitochondrial Membranes, Calcium, Original Article, physiopathology [Parkinson Disease], SK channels, metabolism [Neuroprotective Agents]

URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::acf9e3f82cac3b3215981b433de92087
https://pubmed.ncbi.nlm.nih.gov/24434522