نتائج البحث - "K. Mezghenna"

1

Differential sensitivity of human islets from obese versus lean donors to chronic high glucose or palmitate

المؤلفون: K. Mezghenna, Anne Wojtusciszyn, René Gross, Vanessa Lavallard, Fanny Lebreton, Flore Duranton, F. Castex, Christophe Broca, Jérémy Leroy, Anne-Dominique Lajoix

المساهمون: Université de Montpellier (UM), Biocommunication en Cardio-Métabolique (BC2M), Cellules Souches, Plasticité Cellulaire, Médecine Régénératrice et Immunothérapies (IRMB), Université de Montpellier (UM)-Institut National de la Santé et de la Recherche Médicale (INSERM)-Centre Hospitalier Régional Universitaire [Montpellier] (CHRU Montpellier), RD-Néphrologie (R&D), Université de Genève (UNIGE), Hôpital Universitaire de Genève, Centre Hospitalier Régional Universitaire [Montpellier] (CHRU Montpellier)-Institut National de la Santé et de la Recherche Médicale (INSERM)-Université de Montpellier (UM), Université de Genève = University of Geneva (UNIGE), Hôpital Universitaire de Genève = University Hospitals of Geneva (HUG), Milhau, Jacqueline

المصدر: Journal of Diabetes
Journal of Diabetes, John Wiley & Sons Australia, Ltd ; Ruijin Hospital, Shanghai Jiaotong University School of Medicine, 2020, 12 (7), pp.532-541. ⟨10.1111/1753-0407.13026⟩
Journal of Diabetes, Vol. 12, No 7 (2020) pp. 532-541

مصطلحات موضوعية: Male, endocrine system diseases, [SDV]Life Sciences [q-bio], Endocrinology, Diabetes and Metabolism, Palmitates, FOXO1, 030204 cardiovascular system & hematology, 0302 clinical medicine, MESH: Islets of Langerhans / metabolism, Middle Aged, Obesity / metabolism, Palmitates / pharmacology, Islet transplantation, ComputingMilieux_MISCELLANEOUS, [SDV.MHEP.EM] Life Sciences [q-bio]/Human health and pathology/Endocrinology and metabolism, chemistry.chemical_classification, geography.geographical_feature_category, ddc:617, biology, Insulin secretion, [SDV.MHEP.EM]Life Sciences [q-bio]/Human health and pathology/Endocrinology and metabolism, [SDV.SP]Life Sciences [q-bio]/Pharmaceutical sciences, Islet, [SDV.SP] Life Sciences [q-bio]/Pharmaceutical sciences, Nitric oxide synthase, MESH: Aged Glucose / pharmacology, Humans, Insulin Secretion / drug effects, Islets of Langerhans / drug effect, endocrine system, medicine.medical_specialty, 030209 endocrinology & metabolism, Islets of Langerhans, 03 medical and health sciences, Internal medicine, Diabetes mellitus, medicine, Obesity, Aged, Human islets, geography, business.industry, Fatty acid, medicine.disease, Transplantation, Glucose, Endocrinology, Basal (medicine), chemistry, biology.protein, Pancreatic islet transplantation, business

وصف الملف: application/pdf

URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::8d175ff6aff688281ddbe03cfe754af1
https://doi.org/10.1111/1753-0407.13026

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2

P166 La protéine PIN module la migration des vésicules sécrétoires d’insuline dans la lignée INS-1

المؤلفون: K. Mezghenna, Jérémy Leroy, Sylvie Peraldi-Roux, Sebastian Barg, René Gross, Jacqueline Azay-Milhau, Anne-Dominique Lajoix

المصدر: Diabetes & Metabolism. 41:A74-A75

مصطلحات موضوعية: Endocrinology, Endocrinology, Diabetes and Metabolism, Internal Medicine, General Medicine

URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_________::6697c4f4203488a258b0f32b0c3e1cd7
https://doi.org/10.1016/s1262-3636(15)30279-2

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3

Counteracting neuronal nitric oxide synthase proteasomal degradation improves glucose transport in insulin-resistant skeletal muscle from Zucker fa/fa rats

المؤلفون: René Gross, Viviana Delgado-Betancourt, K. Mezghenna, F. Castex, Jérémy Leroy, Sylvain Gervais, Jacqueline Azay-Milhau, Didier Tousch, Anne-Dominique Lajoix

المساهمون: Biocommunication en Cardio-Métabolique (BC2M), Université de Montpellier (UM), Laboratoire de Mathématiques Jean Leray (LMJL), Centre National de la Recherche Scientifique (CNRS)-Université de Nantes - UFR des Sciences et des Techniques (UN UFR ST), Université de Nantes (UN)-Université de Nantes (UN), Institut de Génomique Fonctionnelle - Montpellier GenomiX (IGF MGX), Institut de Génomique Fonctionnelle (IGF), Université de Montpellier (UM)-Université Montpellier 1 (UM1)-Institut National de la Santé et de la Recherche Médicale (INSERM)-Université Montpellier 2 - Sciences et Techniques (UM2)-Centre National de la Recherche Scientifique (CNRS)-Université de Montpellier (UM)-Université Montpellier 1 (UM1)-Institut National de la Santé et de la Recherche Médicale (INSERM)-Université Montpellier 2 - Sciences et Techniques (UM2)-Centre National de la Recherche Scientifique (CNRS)

المصدر: Diabetologia
Diabetologia, Springer Verlag, 2014, 57 (1), pp.177-186. ⟨10.1007/s00125-013-3084-9⟩

URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::668790e8079b08622ac6fce6204f05ba
https://pubmed.ncbi.nlm.nih.gov/24186360

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4

M19 modulates skeletal muscle differentiation and insulin secretion in pancreatic β-cells through modulation of respiratory chain activity

المؤلفون: K. Mezghenna, Linda Cambier, Béatrice Chabi, René Gross, Patrice Rassam, Charles Auffray, Eric Eveno, Anne-Dominique Lajoix, Chantal Wrutniak-Cabello, Pascal Pomiès

المساهمون: Centre de pharmacologie et innovation dans le diabète (CPID), Université Montpellier 1 (UM1)-Centre Hospitalier Régional Universitaire [Montpellier] (CHRU Montpellier)-Centre National de la Recherche Scientifique (CNRS)

المصدر: PLoS ONE
Clinical Neurology and Neurosurgery
Clinical Neurology and Neurosurgery, Elsevier, 2012, 7 (2), pp.e31815. ⟨10.1371/journal.pone.0031815⟩
PLoS ONE, Vol 7, Iss 2, p e31815 (2012)
Plos One 2 (7), e31815. (2012)

مصطلحات موضوعية: Anatomy and Physiology, muscle, Organogenesis, Respiratory chain, lcsh:Medicine, MESH: Monitoring, Physiologic, Mitochondrion, Biochemistry, Mice, Adenosine Triphosphate, Insulin-Secreting Cells, maladie génétique, Insulin Secretion, Molecular Cell Biology, Myocyte, Insulin, lcsh:Science, Musculoskeletal System, Mitochondrial nucleoid, 0303 health sciences, Multidisciplinary, 030302 biochemistry & molecular biology, adn, Cell Differentiation, [SDV.MHEP.EM]Life Sciences [q-bio]/Human health and pathology/Endocrinology and metabolism, myogénèse, Cell biology, Mitochondria, acide aminé, MESH: Evoked Potentials, Protein Transport, medicine.anatomical_structure, mitochondrie, protéine, myopathies métaboliques, Medicine, diabète, Research Article, Mitochondrial DNA, Molecular Sequence Data, Endocrine System, Biology, Protein Sorting Signals, MESH: Nervous System Diseases, Electron Transport, Mitochondrial Proteins, 03 medical and health sciences, Oxygen Consumption, cellules bêta pancréatiques, medicine, cancer, Nucleoid, Animals, Humans, Amino Acid Sequence, Muscle, Skeletal, insuline, MESH: Coma, 030304 developmental biology, Muscle Cells, MESH: Humans, lcsh:R, Skeletal muscle, MESH: Multiple Sclerosis, maladie respiratoire, Metabolic Disorders, DNAJA3, lcsh:Q, MESH: Alzheimer Disease, [SDV.MHEP]Life Sciences [q-bio]/Human health and pathology, Biomarkers, HeLa Cells

وصف الملف: application/pdf

URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::75c51ac3a9048540e251f26bc1222f20
https://pubmed.ncbi.nlm.nih.gov/22363741

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5

Increased neuronal nitric oxide synthase dimerisation is involved in rat and human pancreatic beta cell hyperactivity in obesity

المؤلفون: B Nadal, F. Castex, Pascal Pomiès, K. Mezghenna, Pierre Petit, René Gross, J. Leroy, Linda Cambier, Nadja Niclauss, Anne-Dominique Lajoix, Ramon Gomis, C Cazevieille, Thierry Berney, A Chalançon

المصدر: Diabetologia, Vol. 54, No 11 (2011) pp. 2856-66

مصطلحات موضوعية: Male, medicine.medical_specialty, Insulin-Secreting Cells/drug effects/metabolism/secretion/ultrastructure, Endocrinology, Diabetes and Metabolism, RNA, Messenger/metabolism, Type 2 diabetes, Nitric Oxide Synthase Type I, Gene Expression Regulation, Enzymologic, Rats, Mutant Strains, Tissue Culture Techniques, Islets of Langerhans, Insulin resistance, Internal medicine, Insulin-Secreting Cells, Obesity/metabolism, Insulin Secretion, Internal Medicine, medicine, Animals, Humans, Insulin, Obesity, RNA, Messenger, Cells, Cultured, Regulation of gene expression, ddc:617, business.industry, Pancreatic islets, Nitric Oxide Synthase Type I/antagonists & inhibitors/genetics/metabolism, Middle Aged, medicine.disease, Transport protein, Mitochondria, Rats, Rats, Zucker, Protein Transport, Endocrinology, medicine.anatomical_structure, Insulin/metabolism/secretion, Islets of Langerhans/drug effects/metabolism/secretion/ultrastructure, Female, Beta cell, Insulin Resistance, Mitochondria/drug effects/metabolism/ultrastructure, business, Dimerization, Function (biology)

URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::8f575cc0dfefb7329d77efecea4a33f7
https://pubmed.ncbi.nlm.nih.gov/21847584

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6

Deregulation of hepatic insulin sensitivity induced by central lipid infusion in rats is mediated by nitric oxide

المؤلفون: Céline Cruciani-Guglielmacci, K. Mezghenna, Maud Soty, René Gross, Carine Zitoun, José Vilar, Nadim Kassis, Christophe Magnan, Bruno Pillot, Nicolas Marsollier, Xavier Fioramonti, Aurélie Joly, Vanessa H. Routh, Gilles Mithieux, Anne-Dominique Lajoix, Amélie Lacombe

المساهمون: Centre de pharmacologie et innovation dans le diabète (CPID), Centre National de la Recherche Scientifique (CNRS)-Centre Hospitalier Régional Universitaire [Montpellier] (CHRU Montpellier)-Université Montpellier 1 (UM1)

المصدر: PLoS ONE
PLoS ONE, Public Library of Science, 2009, 4 (8), pp.e6649. ⟨10.1371/journal.pone.0006649⟩
PLoS ONE, Vol 4, Iss 8, p e6649 (2009)

مصطلحات موضوعية: medicine.medical_specialty, medicine.medical_treatment, Hypothalamus, lcsh:Medicine, Nitric Oxide Synthase Type I, Carbohydrate metabolism, Nitric Oxide, Nitric oxide, 03 medical and health sciences, chemistry.chemical_compound, 0302 clinical medicine, Insulin resistance, Internal medicine, Nutrition/Obesity, medicine, Glucose homeostasis, Animals, Nitric Oxide Donors, Enzyme Inhibitors, lcsh:Science, 030304 developmental biology, chemistry.chemical_classification, 0303 health sciences, Multidisciplinary, Chemistry, Physiology/Endocrinology, Insulin, lcsh:R, Fatty acid, Feeding Behavior, [SDV.MHEP.EM]Life Sciences [q-bio]/Human health and pathology/Endocrinology and metabolism, medicine.disease, Rats, Oleic acid, Endocrinology, Glucose, Liver, Physiology/Integrative Physiology, lcsh:Q, Insulin Resistance, 030217 neurology & neurosurgery, Oleic Acid, Research Article