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1Academic Journal
المؤلفون: Razan Orfali, Ali AlFaiz, Mohammad Asikur Rahman, Liz Lau, Young-Woo Nam, Miao Zhang
المصدر: Biomedicines, Vol 11, Iss 7, p 1780 (2023)
مصطلحات موضوعية: K Ca 2 channels, lungs, motile cilia, cystic fibrosis, anosmia, chronic obstructive pulmonary diseases, Biology (General), QH301-705.5
Relation: https://www.mdpi.com/2227-9059/11/7/1780; https://doaj.org/toc/2227-9059; https://doaj.org/article/3c020c35823c4a62aa96542fbdbe1ffb
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2Academic Journal
المؤلفون: Fenner, M.F., Gatta, G., Sattler, S., Kuiper, M., Hesselkilde, E.M., Adler, D.M.T., Smerup, M., Schotten, U., Sorensen, U., Diness, J.G., Jespersen, T., Verheule, S., Van Hunnik, A., Buhl, R.
المصدر: Fenner , M F , Gatta , G , Sattler , S , Kuiper , M , Hesselkilde , E M , Adler , D M T , Smerup , M , Schotten , U , Sorensen , U , Diness , J G , Jespersen , T , Verheule , S , Van Hunnik , A & Buhl , R 2021 , ' Inhibition of Small-Conductance Calcium-Activated Potassium Current (IK,Ca) Leads to Differential Atrial Electrophysiological Effects in a Horse Model of Persistent Atrial Fibrillation ' , Frontiers in physiology , vol. 12 , 614483 . https://doi.org/10.3389/fphys.2021.614483
مصطلحات موضوعية: af conduction, atrial selectivity, epicardial contact mapping, equine, horse model, inter-atrial heterogeneity, k(ca)2 channels, ns8593, persistent atrial fibrillation, sk
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3Academic Journal
المؤلفون: Dolga, A. M., Terpolilli, N., Kepura, F., Nijholt, I. M., Knaus, H-G, D'Orsi, B., Prehn, J. H. M., Eisel, U. L. M., Plant, T., Plesnila, N., Culmsee, C.
المصدر: Dolga , A M , Terpolilli , N , Kepura , F , Nijholt , I M , Knaus , H-G , D'Orsi , B , Prehn , J H M , Eisel , U L M , Plant , T , Plesnila , N & Culmsee , C 2011 , ' K Ca 2 channels activation prevents [Ca 2+ ] i deregulation and reduces neuronal death following glutamate toxicity and cerebral ischemia ' , Cell Death and Disease , vol. 2 , no. 4 , 147 . https://doi.org/10.1038/cddis.2011.30
مصطلحات موضوعية: calcium homeostasis, cerebral ischemia, K(Ca)2 channels, glutamate excitotoxicity, neuroprotection, CA2+-ACTIVATED K+ CHANNELS, FACTOR-KAPPA-B, SMALL-CONDUCTANCE, HIPPOCAMPAL-NEURONS, SK CHANNELS, CELL-DEATH, CALCIUM, BRAIN, INHIBITION, EXCITOTOXICITY
وصف الملف: application/pdf
الاتاحة: https://hdl.handle.net/11370/c907339e-c539-4c7d-9a2f-72dceb5f43a6
https://research.rug.nl/en/publications/c907339e-c539-4c7d-9a2f-72dceb5f43a6
https://doi.org/10.1038/cddis.2011.30
https://pure.rug.nl/ws/files/6761880/2011CellDeathDisDolga.pdf
https://pure.rug.nl/ws/files/6761879/2011CellDeathDisDolgaSupp.pdf -
4Electronic Resource
المؤلفون: Verkhrasky, A., Dolga, Amalia, Terpolilli, N., Kepura, F., Nijholt, I. M., Knaus, H-G, D'Orsi, B., Prehn, J. H. M., Eisel, U. L. M., Plant, T., Plesnila, N., Culmsee, C., Orsi, B. D’
المصدر: Dolga , A , Terpolilli , N , Kepura , F , Nijholt , I M , Knaus , H-G , D'Orsi , B , Prehn , J H M , Eisel , U L M , Plant , T , Plesnila , N , Culmsee , C , Orsi , B D & Verkhrasky , A (ed.) 2011 , ' K(Ca)2 channels activation prevents [Ca2+](i) deregulation and reduces neuronal death following glutamate toxicity and cerebral ischemia ' Cell death & disease , vol 2 , no. 4 , 147 . DOI: 10.1038/cddis.2011.30