-
1Academic Journal
المؤلفون: Balcázar, Norman, Sánchez, Gloria I, Garcia-Vallejo, Felipe
المصدر: Memórias do Instituto Oswaldo Cruz. July 2003 98(5)
مصطلحات موضوعية: human T cell lymphotropic virus type 1, HTLV-1 associated myelopathy/tropical spastic paraparesis, HTLV-1 phylogeny, Colombia
وصف الملف: text/html
-
2Academic Journal
المؤلفون: Mina-Paz, Yecid, Tafur Tascón, Luis Javier, Cabrera Hernández, Moisés Arturo, Povea Combariza, Camilo, Tejada Rojas, Carmen Ximena, Hurtado Gutiérrez, Hugo, García-Corzo, Sergio Andrés, Garcia-Vallejo, Felipe
مصطلحات موضوعية: Ventilatory threshold, Ergospirometry, Heart rate variability, Cyclists, Women, Educación Física y Deportiva
Relation: https://doi.org/10.14198/jhse.2023.181.01; Journal of Human Sport and Exercise. 2023, 18(1): 1-10. https://doi.org/10.14198/jhse.2023.181.01; http://hdl.handle.net/10045/114884
-
3Academic Journal
المؤلفون: Mina-Paz, Yecid, Zambrano, Diana Carolina, Matta, Andrés Jenuer, Rodríguez, Alejandra, Garcia-Vallejo, Felipe
مصطلحات موضوعية: Aerobic training, Muscle, DNA microarray, Differential expression, Network, Educación Física y Deportiva
Relation: https://doi.org/10.14198/jhse.2022.173.11; Journal of Human Sport and Exercise. 2022, 17(3): 598-608. https://doi.org/10.14198/jhse.2022.173.11; http://hdl.handle.net/10045/111900
-
4Book
مصطلحات موضوعية: Genoma humano, Biología molecular, Genética humana
وصف الملف: 1 recurso en línea (184 páginas); application/pdf
Relation: Colección Salud; https://hdl.handle.net/10893/20229
الاتاحة: https://hdl.handle.net/10893/20229
-
5Book
المؤلفون: Rodríguez Ortiz, Alejandra Rocio, Saldarriaga, Mailyn Alejandra Bedoya, Villegas, Julio César Montoya, García-Vallejo, Felipe
مصطلحات موضوعية: Gene Regulation
-
6Academic Journal
المؤلفون: Garcia-Vallejo, Felipe
المساهمون: Rodriguez, Alejandra, Bedoya, Mailyn, Montoya, Julio Cesar
المصدر: MODID-6d55e02e354:IntechOpen
مصطلحات موضوعية: Science / Life Sciences / Biochemistry, bisacsh:SCI007000
وصف الملف: application/pdf
Relation: https://openresearchlibrary.org/viewer/d4cc9870-e468-4c84-9126-1a3f241063b1; https://openresearchlibrary.org/ext/api/media/d4cc9870-e468-4c84-9126-1a3f241063b1/assets/external_content.pdf
-
7Book
المؤلفون: García-Vallejo, Felipe, Rodríguez Ortiz, Alejandra Rocío, Azcárate Gómez, Camila, Santiago Ospina, Meliza, Montoya Villegas, Julio César, Sánchez Gómez, Adalberto, Satizábal Soto, José María
مصطلحات موضوعية: Advances in Research on Down Syndrome
-
8Academic Journal
المؤلفون: Rodríguez, Alejandra, Echandía, Carlos, Sánchez, Adalberto, Satizábal, José María, Montoya, Julio César, García Vallejo, Felipe
المصدر: Revista Med; Vol. 26 No. 1 (2018): january - june; 14-25 ; Revista Med; Vol. 26 Núm. 1 (2018): enero - junio; 14-25 ; 1909-7700 ; 0121-5256
وصف الملف: application/xml; application/pdf
Relation: http://revistas.unimilitar.edu.co/index.php/rmed/article/view/3978/3296; http://revistas.unimilitar.edu.co/index.php/rmed/article/view/3978/3306; http://revistas.unimilitar.edu.co/index.php/rmed/article/view/3978
-
9Academic Journal
المؤلفون: Azcarate Gomez, Camila, Rodríguez, Alejandra, Sánchez, Adalberto, Montoya, Julio Cesar, Satizabal, José María, García Vallejo, Felipe
المصدر: REVISTA DE LA ASOCIACION COLOMBIANA DE CIENCIAS BIOLOGICAS; Vol. 1 Núm. 29 (2017): REVISTA DE LA ASOCIACION COLOMBIANA DE CIENCIAS BIOLOGICAS; Artículo Páginas 98-102 ; 2500-7459 ; 0120-4173
وصف الملف: application/pdf
-
10Academic Journal
المؤلفون: Ceballos Núñez, Verónika, Rodríguez, Alejandra, Satizábal, José María, Montoya, Julio César, Sánchez, Adalberto, García Vallejo, Felipe
المصدر: Revista Med; Vol. 24 No. 1 (2016): january - june; 21-32 ; Revista Med; Vol. 24 Núm. 1 (2016): enero - junio; 21-32 ; 1909-7700 ; 0121-5256
مصطلحات موضوعية: Preeclampsia, Gene expression, Expression profiles, Meta-analysis, Data mining, Bioinformatics, Chromatin, expresión génica, perfiles de expresión, meta-análisis, minería de datos, bioinformática, cromatina, Pré-eclampsia, expressão do gene, perfis de expressão, meta-análise, mineração de dados, bioin¬formática
وصف الملف: application/pdf
Relation: http://revistas.unimilitar.edu.co/index.php/rmed/article/view/2329/2416; Herrera JA, Herrera-Miranda R, Herrera-Escobar JP, Nieto-Díaz A. Reducción de la mortalidad materna por preeclampsia en Colombia - un análisis de series de tiempo interrumpido. Colombia Médica. 2014; 45:25-31.; Sircar M, Thadhani R, Karumanchi SA. Pathogenesis of preeclampsia. Curr Opin Nephrol Hypertens. 2015; 24:131-138.http://dx.doi.org/10.1097/MNH.0000000000000105; Mol BW, Roberts CT, Thangaratinam S, Magee LA, de Groot CJ, Hofmeyr GJ. Pre-eclampsia. Lancet. 2015; pii: S0140-6736.; Cohen JM, Beddaoui M, Kramer MS, Platt RW, Basso O, Kahn SR. Maternal Antioxidant Levels in Pregnancy and Risk of Preeclampsia and Small for Gestational Age Birth: A Systematic Review and Meta-Analysis. PLoS One. 2015; 10(8):e0135192.http://dx.doi.org/10.1371/journal.pone.0135192; Dekker GA. Management of preeclampsia. Pregnancy Hypertens. 2014;4(3):246-247.http://dx.doi.org/10.1016/j.preghy.2014.04.021; Said J, G. Dekker G. Pre-eclamsia and trombophilia. Best Practice & Research Clinical Obstetrics & Gynaecology 2003;17(3): 441-458.http://dx.doi.org/10.1016/S1521-6934(03)00008-7; Gillon TE, Pels A, von Dadelszen P, MacDonell K, Magee LA. Hypertensive disorders of pregnancy: a systematic review of international clinical practice guidelines. PLoS One. 2014;9(12):e113715.http://dx.doi.org/10.1371/journal.pone.0113715; Kajii T, Ohama K. Androgenetic origin of hydatidiform mole. Nature 1977; 268(5621): 633-634.http://dx.doi.org/10.1038/268633a0; Hansson SR, Chen Y, Brodszki J, Chen M, Hernandez-Andrade E, Inman JM, et. al. Gene expression profiling of human placentas from preeclamptic and normotensive pregnancies. Mol Hum Reproduction. 2006; 12: 169-179.http://dx.doi.org/10.1093/molehr/gal011; Lyall F. The human placental bed revisited. Placenta 2002; 23: 555-562.http://dx.doi.org/10.1053/plac.2002.0850; Lyall F. Development of the utero-placental circulation: the role of carbon monoxide and nitric oxide in trophoblast invasion and spiral artery transformation. Microscopy Res Technique. 2003; 60(4): 402-411.http://dx.doi.org/10.1002/jemt.10278; Garoby-Salom S, Vayssiere C, Salvayre R, Parant O, Negre-Salvayre A. Oxidative stress and preeclampsia: A review. Gynecol Obstet Fertility. 2015; pii: S1297-9589.; Gram M, Dolberg Anderson U, Johansson ME, Edström-Hägerwall A, Larsson I, Jälmby M, Hansson SR, Åkerström B. The Human Endogenous Protection System against Cell-Free Hemoglobin and Heme Is Overwhelmed in Preeclampsia and Provides Potential Biomarkers and Clinical Indicators. PLoS One. 2015; http://dx.doi.org/10.1371/journal.pone.0138111; Vanwijk MJ, Kublickiene K, Boer K, Vanbavel E. Vascular function in preeclampsia. Cardiovascular Research. 2000; 47(1): 38-48.http://dx.doi.org/10.1016/S0008-6363(00)00087-0; Nishizawa H, Pryor-Koishi K, Kato T, Kowa H, Kurahashi H, Udagawa Y. Microarray Analysis of Differentially Expressed Fetal Genes in Placental Tissue Derived from Early and Late Onset Severe Pre-eclampsia. Placenta. 2007; 28: 487-497.http://dx.doi.org/10.1016/j.placenta.2006.05.010; Cox B, Leavey K, Nosi U, Wong F, Kingdom J. Placental transcriptome in development and pathology: expression, function, and methods of analysis. Am J Obstet Gynecol. 2015; 213 (Suppl):S138-151.http://dx.doi.org/10.1016/j.ajog.2015.07.046; Sõber S, Reiman M, Kikas T, Rull K, Inno R, Vaas P, Teesalu P, Marti JM, Mattila P, Laan M. Extensive shift in placental transcriptome profile in preeclampsia and placental origin of adverse pregnancy outcomes. Sci Rep. 2015; 5: 13336.ttp://dx.doi.org/10.1038/srep13336; Winn VD, Gormley M, Paquet AC, Kjaer-Sorensen K, Kramer A, Rumer KK, et al. Severe Preeclampsia-Related Changes in Gene Expression at the Maternal-Fetal Interface Include Sialic Acid-Binding Immunoglobulin-Like Lectin-6 and Pappalysin-2. Endocrinology. 2009; 150(1):452-462.http://dx.doi.org/10.1210/en.2008-0990; Enquobahrie DA, Meller M, Rice K, Psaty BM, Siscovick DS, Williams MA. Differential placental gene expression in preeclampsia. Am J Obst Gynecol. 2008; 199:566.e1-566.e11.http://dx.doi.org/10.1016/j.ajog.2008.04.020; Rajakumar A, Chua T, Handley DE, Bunce KD, Burke B, Hubel CA, et al. Maternal gene expression profiling during pregnancy and preeclampsia in human peripheral blood mononuclear cells. Placenta 2011; 32: 70-78.http://dx.doi.org/10.1016/j.placenta.2010.10.004; Su AI, Wiltshire T, Batalov S, Lapp H, Ching KA, Block D, et. al. A gene atlas of the mouse and human protein-encoding transcriptomes. Proc Natl Acad Sci USA. 2004; 101(16):6062-6067.http://dx.doi.org/10.1073/pnas.0400782101; Wu C, Orozco C, Boyer J, Leglise M, Goodale J, Batalov S, et al. BioGPS: an extensible and customizable portal for querying ang organizing gene annotation resources. Genome Biology 2009; 10(11): R130.http://dx.doi.org/10.1186/gb-2009-10-11-r130; Winn VD, Haimov-Kochman R, Paquet AC, Yang YJ, Madhusudhan MS, Gormley M, et. al. Gene expression profiling of the human maternal-fetal interface reveals dramatic changes between midgestation and term. Endocrinology 2007; 148:1059-1079.http://dx.doi.org/10.1210/en.2006-0683; Laresgoiti-Servitje E, Gomez-Lopez N. The pathophysiology of preeclampsia involves altered levels of angiogenic factors promoted by hypoxia and autoantibody-mediated mechanisms. Biol Reprod. 2012; 87(2):36.http://dx.doi.org/10.1095/biolreprod.112.099861; Jeha D, Usta I, Ghulmiyyah L, Nassar A. A review of the risks and consequences of adolescent pregnancy. J Neonatal Perinatal Med. 2015 Mar 12. [Epub ahead of print].http://dx.doi.org/10.3233/NPM-15814038; Martinussen MP, Bracken MB, Triche EW, Jacobsen GW, Risnes KR. Folic acid supplementation in early pregnancy and the risk of preeclampsia, small for gestational age offspring and preterm delivery. Eur J Obstet Gynecol Reprod Biol. 2015;195:94-99. http://dx.doi.org/10.1016/j.ejogrb.2015.09.022; Boeldt DS, Hankes AC, Alvarez RE, Khurshid N, Balistreri M, Grummer MA, Yi F, Bird IM. Pregnancy programming and preeclampsia: identifying a human endothelial model to study pregnancy-adapted endothelial function and endothelial adaptive failure in preeclamptic subjects. Adv Exp Med Biol. 2014; 814:27-47.http://dx.doi.org/10.1007/978-1-4939-1031-1_4; Murphy MS, Bytautiene E, Saade G, Smith GN. Alterations to the maternal circulating proteome after pre-eclampsia. Am J Obstet Gynecol. 2015 Oct 14. pii: S0002-9378(15)01271-5.; Monk D. Genomic imprinting in the human placenta. Am J Obstet Gynecol. 2015; 213(4 Suppl):S152-S162.http://dx.doi.org/10.1016/j.ajog.2015.06.032; Calicchio R, Doridot L, Miralles F, Méhats C, Vaiman D1. DNA methylation, an epigenetic mode of gene expression regulation in reproductive science. Curr Pharm Des. 2014; 20(11):1726-1750.http://dx.doi.org/10.2174/13816128113199990517; Ichiyanagi K, Okada N. Mobility Pathways for Vertebrate L1, L2, CR1, and RTE Clade Retrotransposons. Mol Biol Evol 2001; 25(6):1148-1157.http://dx.doi.org/10.1093/molbev/msn061; Khazina E, Weichenrieder O. Non-LTR retrotransposons encode noncanonical RRM domains in their first open reading frame. Proc Natl Acad Sci USA. 2009; 106:731-736.http://dx.doi.org/10.1073/pnas.0809964106; Schulz LC, Widmaier EP, Qiu J, Roberts RM. Effect of leptin on mouse trophoblast giant cells. Biol Reprod 2009; 80: 415-424.http://dx.doi.org/10.1095/biolreprod.108.073130; Cameo P, Bischof P, Calvo JC. Effect of leptin on progesterone, human chorionic gonadotropin, and interleukin-6 secretion by human term trophoblast cells in culture. Biol Reprod 2003; 68: 472-477.http://dx.doi.org/10.1095/biolreprod.102.006122; Schulz LC, Widmaier EP. The effect of leptin on mouse trophoblast cell invasion. Biol Reprod 2004; 71:1963-1967.http://dx.doi.org/10.1095/biolreprod.104.032722; Jansson N, Greenwood SL, Johansson BR, Powell TL, Jansson T. Leptin stimulates the activity of the system A amino acid transporter in human placental villous fragments. J Clin Endocrinol Metab 2003; 88: 1205-1211.http://dx.doi.org/10.1210/jc.2002-021332; Turgut A, Ozler A, Goruk NY, Tunç SY, Sak ME, Evsen MS, Evliyaoglu O, Gul T. Serum levels of the adipokines, free fatty acids, and oxidative stress markers in obese and non-obese preeclamptic patients. Clin Exp Obstet Gynecol. 2015; 42:473-479.; Henson MC, Castracane VD. Leptin in pregnancy: an update. Biology of Reproduction 2006; 74:218-229. http://dx.doi.org/10.1095/biolreprod.105.045120; Rahardjo B, Widjajanto E, Sujuti H, Keman K. Different levels of IL-1α, IL-6, TNF-α, NF-κB and PPAR-γ in monocyte cultures exposed by plasma preeclampsia and normotensive pregnancy. Pregnancy Hypertens. 2014; 4:187-193. http://dx.doi.org/10.1016/j.preghy.2014.03.001; Kaartokallio T, Cervera A, Kyllönen A, Laivuori K. Gene expression profiling of pre-eclamptic placentae by RNA sequencing.Adicionalmente. Sci Rep. 2015; 5:14107. http://dx.doi.org/10.1038/srep14107; Challier JC, Uzan S. The human placenta and its pathologies: focus on oxygen. Med Sci (Paris). 2003; 19(11):1111-1120. http://dx.doi.org/10.1051/medsci/200319111111; Louwen F, Muschol-Steinmetz C, Reinhard J, Reitter A, Yuan J. A lesson for cancer research: placental microarray gene analysis in Preeclampsia. Oncotarget. 2012; 3(8): 759-773. http://dx.doi.org/10.18632/oncotarget.595; Redman C. The six stages of pre-eclampsia. Pregnancy Hypertens. 2014; 4(3):246. http://dx.doi.org/10.1016/j.preghy.2014.04.020; Torrado J, Farro I, Zócalo Y, Farro F, Sosa C, Scasso S, Alonso J, Bia D. Preeclampsia Is Associated with Increased Central Aortic Pressure, Elastic Arteries Stiffness and Wave Reflections, and Resting and Recruitable Endothelial Dysfunction. Int J Hypertens. 2015; 2015:720683. http://dx.doi.org/10.1155/2015/720683; Reyna-Villasmil E, Brice-o-Pérez C, Torres-Cepeda D. Inmunología, inflamación y Preeclampsia. Revista de Obstetricia y Ginecología de Venezuela. 2009; 69(2): 97-110.; Devergne O, Coulomb-L'Herminé A, Capel F, Moussa M, Capron, F. Expression of the Epstein-Barr virus-induced gene 3, an interleukin-12 p40-related molecule, throughout human pregnancy: involvement of syncytiotrophoblasts and extravillous trophoblasts. The American Journal of Pathology. 2001; 159(5): 1763-1776. http://dx.doi.org/10.1016/S0002-9440(10)63023-4; Van Der Hoorn ML, Keijser R, Ris-Stalpers C, Afink G, Claas FH, Van Der Post JA et al. Increased EBI3 expression in placentas of preeclamptic patients. Journal of Reproductive Immunology. 2010; 81: 1-61. http://dx.doi.org/10.1016/j.jri.2010.06.129; Guo J, Tian T, Lu D, Xia G, Wang H, Dong M. Alterations of maternal serum and placental follistatin-like 3 and myostatin in pre-eclampsia. Journal of Obstetrics and Gynecology Research. 2012; 38(7): 988-996. http://dx.doi.org/10.1111/j.1447-0756.2011.01823.x; Kleinrouweler CE, Van Uitert M, Moerland PD, Ris-Stalpers C, Van der Post JAM, Afink GB. Differentially expressed genes in the Pre-eclamptic placenta: A systematic review and meta-analysis. PLoS ONE. 2013; 8(7): e68991. http://dx.doi.org/10.1371/journal.pone.0068991; Dijke PT, Arthur HM. Extracellular control of TGF_ signaling in vascular development and disease. Nature Reviews. 2007; 8: 857-869. http://dx.doi.org/10.1038/nrm2262; Lowe SA, Bowyer L, Lust K, McMahon LP, Morton M, North RA, Paech M, Said JM. SOMANZ guidelines for the management of hypertensive disorders of pregnancy 2014. Aust N Z J Obstet Gynaecol. 2015; 55(5):e1-e29. http://dx.doi.org/10.1111/ajo.12399; Enquobahrie DA, Meller M, Rice K, Psaty BM, Siscovick DS, Williams MA. Differential placental gene expression in preeclampsia. American J Obstetrics & Gynecology 2008; 199:566.e1-566.e11. http://dx.doi.org/10.1016/j.ajog.2008.04.020; http://revistas.unimilitar.edu.co/index.php/rmed/article/view/2329
-
11Academic Journal
المؤلفون: Ospina, Meliza Santiago, Rodríguez, Alejandra, Montoya, Julio Cesar, Sánchez, Adalberto, Satizábal, José María, García Vallejo, Felipe
المصدر: REVISTA DE LA ASOCIACION COLOMBIANA DE CIENCIAS BIOLOGICAS; Vol. 1 Núm. 28 (2016): REVISTA DE LA ASOCIACION COLOMBIANA DE CIENCIAS BIOLOGICAS; Páginas 117-124 ; 2500-7459 ; 0120-4173
وصف الملف: application/pdf
-
12Academic Journal
المؤلفون: Garcia Vallejo, Felipe
المساهمون: Academia Colombiana de Ciencias Exactas, Físicas y Naturales
المصدر: Revista de la Academia Colombiana de Ciencias Exactas, Físicas y Naturales
مصطلحات موضوعية: Retrovirus, Cointegración retroviral, Linfocitos, Isla CpG, Genes clase II, Simulación computacional, Retroviral co-integration, Lymphocytes, CpG Island, Class II Genes, Computational simulation
وصف الملف: application/pdf
Relation: Revista de la Academia Colombiana de Ciencias Exactas, Físicas y Naturales; 40; 382; 394; 156; https://repositorio.accefyn.org.co/handle/001/934; https://doi.org/10.18257/raccefyn.364
-
13
-
14Academic Journal
المساهمون: Academia Colombiana de Ciencias Exactas, Físicas y Naturales
مصطلحات موضوعية: Retrovirus, Integración viral, Linfocitos, Islas de CpG, Genes clase II, Simulación por computador, Provirus integration, Lymphocytes, CpG island, Class II genes, Computer simulation
وصف الملف: 11 páginas; application/pdf
Relation: Revista de la Academia Colombiana de Ciencias Exactas, Físicas y Naturales; 39; 239; 249; 151; https://repositorio.accefyn.org.co/handle/001/857; https://doi.org/10.18257/raccefyn.183
-
15Academic Journal
المصدر: Revista Médicas UIS, ISSN 0121-0319, Vol. 34, Nº. 3, 2021, pags. 9-18
مصطلحات موضوعية: Fútbol, Lesiones, Polimorfismo Genético, ACTN3, MMP, Inhibidores Tisulares de Metaloproteinasas, VEFGA, Soccer, Injuries, Genetic Polymorphism, TIMP
وصف الملف: application/pdf
Relation: https://dialnet.unirioja.es/servlet/oaiart?codigo=8222964; (Revista) ISSN 1794-5240; (Revista) ISSN 0121-0319
-
16Academic Journal
المؤلفون: Montoya Villegas, Julio César, Fajardo Colorado, Dianora, Peña-Gonzalez, Angela, Sanchez, Adalberto, Domínguez Narváez, Martha C, Satizábal Soto, José María, García Vallejo, Felipe
مصطلحات موضوعية: Down Syndrome, Nervioso Nervous System Diseases, Cerebro, Corteza cerebral, Homeostasis, Región crítica del Síndrome de Down, Análisis secuencial en arreglo de oligonucleótidos, Transcriptoma, Perfil de expresión génica, Brain, Cerebral cortex, Down Syndrome critical region, Oligonucleotide array sequence analysis, Transcriptome, Gene expression profiling
Time: Universidad Autónoma de Occidente. Calle 25 115-85. Km 2 vía Cali-Jamundí
وصف الملف: application/pdf; 8 páginas
Relation: Colombia Médica. Volumen 45, número 4 (2014); pages 154-161; 161; 154; 45; Montoya, J., Fajardo, D., Peña, Ángela, Sánchez, A., Domínguez, M., Satizábal, J., & García Vallejo, F. (2014). Expresión diferencial global de genes ubicados en la Región Crítica del Síndrome de Down en el cerebro humano normal. Colombia Médica , 45 (4), 154-161. http://red.uao.edu.co//handle/10614/11812; Colombia Médica; Critchley HD, Harrison NA. Visceral influences on brain and behavior. Neuron. 2013; 77: 624–638.; Johnson M, Kawasawa M, Mason C, Krsnik Z, Coppola G, et al. Bogdanovic´ D Functional and evolutionary insights into human brain development through global transcriptome analysis. Neuron. 2009; 62: 494–509.; Oldham M, Konopka G, Iwamoto K, Langfelder P, Kato T, Horvath S, et al. Functional organization of the transcriptome in human brain. Nat Neurosci. 2008; 11: 1271–1282.; Nieuwenhuis-Mark R Diagnosing Alzheimer's dementia in Down syndrome: Problems and possible solutions. Res Dev Disabil. 2009; 30: 827–838.; Abbeduto L, McDuffie A. Genetic Syndromes Associated with Intellectual Disabilities. In: Handbook of Medical Neuropsychology: Applications of Cognitive Neuroscience. Armstrong, CL, Morrow L (Eds) New York: Springer; 2010. pp. 193–221.; Korenberg Julie R, Kawashima Hiroko, Pulst Stefan-M, Ikeuchi T, Ogasawara N, Yamamoto K, et al. Molecular definition of a region of chromosome 21 that causes features of the Down syndrome phenotype. Am J Human Genet. 1990; 47: 236–46.; Montoya J, Soto J, Satizábal J, Sánchez A, García Vallejo F. Genomic study of the critical region of chromosome 21 associated to Down syndrome. Colomb Med (Cali). 2011; 42: 26–38.; Weitzdoerfer R, Dierssen M, Fountoulakis M, Lubec G. Fetal life in Down syndrome starts with normal neuronal density but impaired dendritic spines and synaptosomal structure. J Neural Transm Suppl. 2001; 61: 59–70.; Ferrando-Miguel R, Cheon M, Lubec G. Protein levels of genes encoded on chromosome 21 in fetal Down syndrome brain (Part V): Overexpression of phosphatidyl-inositol-glycan class P protein (DSCR5) Amino Acids. 2004; 26: 255–61.; VanGilder R, Huber J, Rosen C, Barr T. The transcriptome of cerebral ischemia. Brain Res Bull. 2012; 88: 313–9.; Zeng H, Shen E, Hohmann J, Oh S, Bernard A, Royall J, et al. Large-scale cellular-resolution gene profiling in human neocortex reveals species-specific molecular signatures. Cell. 2012; 149: 483– 96.; Cheadle C, Cho-Chung YS, Becker KG, Vawter MP. Application of z-score transformation to Affymetrix data. Appl Bioinformatics. 2003; 2: 209–17.; Montoya J, Peña A, Satizábal J, García-Vallejo F. In silico systemic analysis of the differential expression of genes located in críticalregion of Down syndrome in the human brain. Rev Med. 2012; 20: 15–26.; Amano K, Sago H, Uchikawa C, Suzuki T, Kotliarova SE, Nukina N, Epstein CJ, Yamakawa K. Dosage-dependent over-expression of genes in the trisomic region of Ts1Cje mouse model for Down syndrome. Hum Mol Genet. 2004; 13: 1333–40.; Shao M, Liu ZZ, Wang CD, Li HY, Carron C, Zhang HW, Shi DL. Down syndrome critical region protein 5 regulates membrane localization of Wnt receptors, Dishevelled stability and convergent extension in vertebrate embryos. Development. 2009; 136: 2121-1.; Ferrando-Miguel R, Cheon MS, Yang JW, Lubec G. Overexpression of transcription factor BACH1 in fetal Down syndrome brain. J Neural Transm Suppl. 2003; 67: 193–205.; Richard C, Drevon C, Canto PY, Villain G, Bollérot K, Lempereur A, et al. Endothelio-Mesenchymal Interaction Controls runx1 Expression and Modulates the notch Pathway to Initiate Aortic Hematopoiesis. Dev Cell. 2013; 24: 600–11.; Giambra V, Jenkins CR, Wang H, Lam SH, Shevchuk OO, Nemirovsky O, et al. NOTCH1 promotes T cell leukemia-initiating activity by RUNX-mediated regulation of PKC-? and reactive oxygen species. Nat Med. 2012; 18(11): 1693–8.; Wang W, Zhu JZ, Chang KT, Min KT. DSCR1 interacts with FMRP and is required for spine morphogenesis and local protein synthesis. EMBO J. 2012; 31(18): 3655–66.; Ermak G, Pritchard MA, Dronjak S, Niu B, Davies KJ. Do RCAN1 proteins link chronic stress with neurodegeneration. FASEB J. 2011; 25(10): 3306–11.; Guedj F, Pereira PL, Najas S, Barallobre MJ, Chabert C, Souchet B, et al. DYRK1A: a master regulatory protein controlling brain growth. Neurobiol Dis. 2012; 46(1): 190–200.; Park J, Oh Y, Chung KC. Two key genes closely implicated with the neuropathological characteristics in Down syndrome: DYRK1A and RCAN1. BMB Rep. 2009; 42(1): 6–15.; Packard MG, Goodman J. Emotional arousal and multiple memory systems in the mammalian brain. Front Behav Neurosci. 2012; 6: 14.; Ishizu T, Zeki S. Toward a brain-based theory of beauty. PLoS ONE. 2011; 6: 21852.; Wegiel J, Gong CX, Hwang YW. The role of DYRK1A in neurodegenerative diseases. FEBS J. 2011; 278: 236–45.; Grahn JA, Parkinson JA, Owen AM. The cognitive functions of the caudate nucleus. Prog Neurobiol. 2008; 86(3): 141–55.; Kistler P, Ropper A, Martin J. Enfermedades cerebrovasculares. En: Fauci A, Braunwald E, Kasper D, Hauser S, Longo D, Jameson L, Loscalzo J (eds.). Harrison Principios de Medicina Interna. 17a edición. Barcelona: McGraw-Hill; 2010. pp. 2570–96.; Dauphinot L, Lyle R, Rivals I, Dang MT, Moldrich RX, Golfier G, et al. The cerebellar transcriptome during postnatal development of the Ts1Cje mouse, a segmental trisomy model for Down syndrome. Hum Mol Genet. 2005; 14: 373–84.; Potier MC, Rivals I, Mercier G, Ettwiller L, Moldrich RX, Laffaire J, et al. Transcriptional disruptions in Down syndrome: a case study in the Ts1Cje mouse cerebellum during post-natal development. J Neurochem. 2006; 97(1): 104–9.; Minami T. Calcineurin-NFAT activation and DSCR-1 auto-inhibitory loop: how is homoeostasis regulated? J Biochem. 2014; 155: 217–26.; Arron JR, et al. NFAT dysregulation by increased dosage of DSCR1 and DYRK1A on chromosome 21. Nature. 2006; 441: 595–600; 1657-9534 (Online ); http://red.uao.edu.co//handle/10614/11812
-
17Academic Journal
المؤلفون: Montoya Villegas, Julio César, Sánchez Gómez, Adalberto, Satizábal Soto, José María, García Vallejo, Felipe
مصطلحات موضوعية: Preeclampsia, Bioinformática, Micromatrices de ADN, Red de Expresión Génica, Secuencias repetidas, Bioinformatics, Microarrays of ADN, Gene Expression Network, Repeated sequences
Time: Universidad Autónoma de Occidente. Calle 25 115-85. Km 2 vía Cali-Jamundí
وصف الملف: application/pdf; 11 páginas
Relation: Revista de la Asociación Colombiana de Ciencias Biológicas. 1(26) (2014); p.p. 35-45; 45; 26; 35; Montoya Villegas, J. C., Sánchez Gómez, A., Satizabal Soto, J. M., García Vallejo, F. (2014). Complejidad y construcción de una red de genes asociados a preeclampsia. Revista de la Asociación Colombiana de Ciencias Biológicas. 1(26), 35-45. http://red.uao.edu.co//handle/10614/12070; Revista de la Asociación Colombiana de Ciencias Biológicas; Ajayi, F., Kongoasa, N., Gaffey, T., Asmann, Y.W., Watson, W.J., Baldi, A., Lala, P., Shrindhar, V., Brost, B. & Chien, J. (2008), “Elevated expression of serine protease Htra1 in Preeclampsia and its role in trophoblast cell migration and invasion”, American Journal of Obstetrics & Gynecology, Vol.199 No. 5, pp. 557e1-557e10; Bell, M.J., BSN, R.N. & Conley, Y.P. (2013), “A systematic review of endoglin gene expression in Preeclampsia”, Biological Research for Nursing, Vol. 15 No. 2, pp. 129-136; Ceballos, V. (2011), “Análisis y caracterización genómica y funcional de genes asociados a preeclampsia”, Universidad del Valle; Chelbi, S.T. & Vaiman, D. (2008), “Genetic and epigenetic factors contribute to the onset of preeclampsia”, Molecular and cellular endocrinology, Vol. 282, pp. 120-129; Cline, M.S., Smoot, M., Cerami, E., Kuchinsky, A., Landys N., Workman, C., Christmas, R., AvilaCampilo, I., Creech, M., Gross, B., Hanspers, K., Isserlin, R., Kelley, R., Killcoyne, S., Lotia, S., Maere, S., Morris J., Ono, K., Pavlovic, V., Pico, A.R., Vailaya, A., Wang, P., Adler, A., Conklin, B.,Hood, L., Kuiper, M., Sander, C., Schmulevich, I., Schwikowski, B., Warner, G.J., Ideker, T & Bader, G.D. (2007), “Integration of biological networks and gene expression data using Cytoscape”, Nature Protocols, Vol. 2 No.10, pp. 2366-2382; Enquobahrie, D.A., Meller, M., Rice, K., Psaty, B.M., Siscovick, D.S. & Williams A.M. (2008), “Differential placental gene expression in preeclampsia”, American journal of obstetrics and gynecology, Vol. 199 No. 5, pp. 566.e1–11; Guo, J., Tian, T., Lu, D., Xia, G., Wang, H. & Dong, M. (2012), “Alterations of maternal serum and placental follistatin-like 3 and myostatin in pre-eclampsia”, Journal of Obstetrics and Gynecology Research, Vol. 38 No. 7, pp. 988-996; Iciek, R., Wender-Ozegowska, E., Zawiejska, A., Mikolajczak, P., Mrozikiewicz, P.M., Pietryga, M. & Brazert, J. (2013), “Placental leptin and its receptor genes expression in pregnancies complicated by type 1 diabetes”, Journal of Physiology and Pharmacology, Vol. 64 No. 5, pp. 579-585; Kajii, T. & Ohama, K. (1977), “Androgenetic origin of hydatidiform mole”, Nature, Vol. 268 No. 5621, pp. 633-634; Kellis, M., Wold, B., Snyder, M.P., Bernstein, B.E., Kundaje, A., Marinov, G.K., Ward, L.D., Birney, E., Crawford, G.E., Dekker, J., Dunham, I., Elnitski, L.L., Farnham, P.J., Feingold, E.A., Gerstein, M.,Giddings, M.C., Gilbert, D.M., Gingeras, T.R., Green, E.D., Guigo, R., Hubbard, T., Kent, J., Lieb,J.D., Myers, R.M., Pazin, M.J., Ren, B., Stamatoyannopoulos, J.A., Weng, Z., White, K.P. & Hardison,R. (2014), “Defining functional DNA elements in the human genome”, Proceedings of the National Academy of Sciences, Vol. 111 No. 17, pp. 6131-6138.; Nishizawa, H., Ota, S., Suzuki, M., Kato, T., Sekiya, T., Kurahashi, H. & Udagawa Y. (2011), “Comparative gene expression profiling of placentas from patients with severe preeclampsia and unexplained fetal growth restriction”, Reproductive Biology and Endocrinology, Vol. 9 No. 107, pp. 1-12; Oshima, T., Yoshihara K., Aoyama, T., Hasegawa, S., Sato, T., Yamamoto, N., Akito, N., Shiozawa M., Yoshikawa, T., Numata, K., Rino, Y., Kunisaki, C., Tanaka, K., Akaide, M., Imada, T. & Masuda, M.(2014), “Relation of INHBA gene expression to outcomes in gastric cancer after surgery”, Anticancer Research, Vol. 34 No. 5, pp. 2303-2309.; Powe, C.E., Levine, R.J. & Karumanchi, S.A. (2011), “Preeclampsia, a disease of the maternal endothelium: The role of antiangiogenic factor and implications for later cardiovascular disease”, Circulation, Vol. 123, pp. 2856-2869; Reimer, T., Koczan, D., Gerber, B., Richter, D., Thiesen, H.J & Friese, K. (2002), “Microarray analysis of differentially expressed genes in placental tissue of pre-eclampsia: up-regulation of obesity-related genes”, Molecular Human Reproduction, Vol. 8 No. 7, pp. 674-680.; Reyna-Villasmil, E., Briceño-Pérez, C. & Torres-Cepeda, D. (2009), “Inmunología, inflamación y Preeclampsia”, Revista de Obstetricia y Ginecología de Venezuela, Vol. 69 No. 2, pp. 97-110; Shannon, P., Markiel, A., Ozier, O., Baliga, N.S., Wang, J.T., Ramage, D., Amin, N., Schwikowski, B. & Ideker, T. (2003), “Cytoscape: A software environment for integrated models of biomolecular interaction networks”, Genome Research, Vol. 13, pp. 2498-2504.; Sundrani, D. P., Reddy, U. S., Joshi, A.A., Mehendale, S.S., Chavan-Gautam, P.M., Hardikar, A.A., Chandak, G.R. & Joshi, S.R. (2013), “Differential placental methylation and expression of VEGF, FLT-1 and KDR genes in human term and preterm preeclampsia”, Clinical epigenetics journal, Vol. 5, pp. 1-11; Toft, J.H., Lian, I.A., Tarca, A.L., Erez, O., Espinoza, J., Eide, I.P., Bjorge, L., Sun, C., Draghici, S., Romero, R. & Austgulen, R. (2008), “Whole-genome microarray and targeted analysis of angiogenesis-regulating gene expression (ENG, FLT1, VEGF, PlGF) in placentas from pre-eclamptic and smallfor-gestational-age pregnancies”, The Journal of Maternal-Fetal and Neonatal Medicine, Vol. 21 No. 4, pp. 267-273; Trifonova, E.A., Gabidulina, T.V., Ershov, N.I., Serebrova, V.N., Vorozhishcheva, A.Y. & Stepanov, V.A. (2014), “Analysis of the placental tissue transcriptome of normal and preeclampsia complicated pregnancies”, Acta Naturae, Vol. 6 No. 2, pp. 71-83.; Van Der Hoorn, M.L., Keijser, R., Ris-Stalpers, C., Afink, G., Claas, F.H., Van Der Post, J.A & Scherjon, S.A. (2010), “Increased EBI3 expression in placentas of preeclamptic patients”, Journal of Reproductive Immunology, Vol. 81, pp. 1-61; Wang, L., Cheong, M., Lee, Y., Lee, M. & Chen, H. (2012), “High-Temperature Requirement Protein A4 (HtrA4) suppresses the fusogenic activity of Syncytin-1 and promotes trophoblast invasion”, Molecular and Cellular Biology, Vol. 32 No. 18, pp. 3707-3717; Xiang, Y., Cheng, Y., Li, Xiaotian, Li, Q., Xu, J., Zhang, J., Liu, Y., Xiang, Q., Wang, L., He, L. & Zhao, X. (2013), “Up-regulared expression and aberrant DNA methylation of LEP and SH3PXD2A in Preeclampsia”, PLoS ONE, Vol. 8(3), 1-9 pp.; http://red.uao.edu.co//handle/10614/12070
-
18Electronic Resource
المؤلفون: Mina-Paz, Yecid, Tafur Tascón, Luis Javier, Cabrera Hernández, Moisés Arturo, Povea Combariza, Camilo, Tejada Rojas, Carmen Ximena, Hurtado Gutiérrez, Hugo, García-Corzo, Sergio Andrés, Garcia-Vallejo, Felipe
مصطلحات الفهرس: Ventilatory threshold, Ergospirometry, Heart rate variability, Cyclists, Women, info:eu-repo/semantics/article
-
19Academic Journal
المؤلفون: Montoya Villegas, Julio César, Peña González, Ángela, Satizábal Soto, José María, García-Vallejo, Felipe
المصدر: Revista Med; Vol. 20 No. 1 (2012); 15-26 ; Revista Med; Vol. 20 Núm. 1 (2012); 15-26 ; 1909-7700 ; 0121-5256
مصطلحات موضوعية: análisis de micromatrices, perfilación de la expresión génica, biología computacional, cerebro, síndrome de Down
وصف الملف: application/pdf
Relation: https://revistas.unimilitar.edu.co/index.php/rmed/article/view/1197/917; Stevens CF. Neuronal diversity: too many cell types for comfort? Curr Biol. 1998; 8(20):R708-10.; Oldham MC, Konopka G, Iwamoto K, Langfelder P, Kato T, Horvath S, et al. Functional organization of the transcriptome in human brain. Nat Neurosci. 2008; 11(11):1271-82.; Loebrich S, Nedivi E. The function of activity-regulated genes in the nervous systen. Physiol Rev. 2009; 89:1079-103.; Sutcliffe JG. mRNA in the mammalian central nervous system. Annu Rev Neurosci. 1988; 11:157-98.; Sandberg R, Yasuda R, Pankratz DG, Carter TA, Del Rio JA, Wodicka L, et al. Regional and strain-specific gene expression mapping in the adult mouse brain. Proc Natl Acad Sci USA. 2000; 97:11038-43.; Geschwind DH. Mice, microarrays, and the genetic diversity of the brain. Proc Natl Acad Sci USA. 2000; 97:10676-78.; Zirlinger M, Kreiman G, Anderson DJ. Amygdala-enriched genes identified by microarray technology are restricted to specific amygdaloid subnuclei. Proc Natl Acad Sci USA. 2001; 98:5270-75.; Lein ES, Zhao X, Gage FH. Defining a molecular atlas of the hippocampus using ADN microarrays and high-throughput in situ hybridization. J Neurosci. 2004;24:3879-89.; McClung CA, Nestler E. Regulation of gene expression and cocaine reward by CREB and Delta Fos B. Nat Neurosci. 2003; 6:1208-15.; Lewis NE, Schramm G, Bordbar A, Schellenberger J, Andersen MP, Cheng JK, et al. Large-scale in silico modeling of metabolic interactions between cell types in the human brain. Nature Biotechnol. 2010; 28(12):1279-85.; Hattori M, Fujiyama A, Taylor TD, Watanabe H, Yada T, Park HS, et al. Chromosome 21 mapping and sequencing consortium. The ADN sequence of human chromosome 21. Nature. 2000; 405:311-9.; Gardiner K, Herault Y, Lott IT, Antonarakis SE, Reeves RH, Dierssen M. Down syndrome: from understanding the neurobiology to therapy. J Neurosci.2010; 30(45):14943-5.; Toyoda A, Noguchi H, Taylor TD, Ito T, Pletcher MT, Sakaki S, et al. Comparative genomic sequence analysis of the human chromosome 21 Down Syndrome Critical Region. Genome Res. 2002; 12:1323-32.; Eggermann T, Schönherr N, Spengler S, Jäger S, Denecke B, Binder G, et al. Identification of a 21q22 duplication in a Silver-Russell syndrome patient further narrows down the Down syndrome critical region.Am J Med Genet. 2010; 152A:356-59.; Montoya JC, Soto J, Satizábal JM, Sánchez A, García-Vallejo F. Genomic study of the critical region of chromosome 21 associated to Down syndrome. Colombia Médica. 2011; 42:26-38.; Cheadle C, Vawter MP, Freed WJ, Becker KG. Analysis of microarray data using Z score transformation. J Mol Diagn. 2003; 5:73-81.; Liang WS, Reiman EN, Valla J, Dunckley T, Beach TG, Grover A, et al. Alzheimer’s disease is associated with reduced expression of energy metabolism genes in posterior cingulate neurons. Proc Natl Acad Sci USA. 2008;105:4441-46.; Amano K, Sago H, Uchikawa C, Suzuki T, Kotliarova SE, Nukina N, et al. Dosage-dependent over-expression of genes in the trisomic region of Ts1Cje mouse model for Down syndrome. Hum Mol Genet. 2004; 13:1333-40.; Shao M, Liu ZZ, Wang CD, Li HY, Carron C, Zhang HW, et al. Down syndrome critical region protein 5 regulates membrane localization of Wnt receptors, Dishevelled stability and convergent extension in vertebrate enbryos. Development. 2009; 136:2121-31.; Head E, Lott IT, Patterson D, Doran E, Haier RJ. Possible compensatory events in adult Down syndrome brain prior to the development of Alzheimer disease neuropathology: targets for nonpharmacological intervention. J Alzheimers Dis.2007;11:61-76.; Ryu YS, Park SY, Jung MS, Yoon SH, Kwen MY, Lee SY, et al. Dyrk1A-mediated phosphorylation of Presenilin 1: a functional link between Down syndrome and Alzheimer’s disease. J Neurochem. 2010;115:574-84.; Sun X, Wu Y, Chen B, Zhang Z, Zhou W, Tong Y, et al. Regu la tor of calcineurin 1 (RCAN1) facilitates neuronal apoptosis through caspase-3 activation. J Biol Chem. 2011; 286:9049-62.; Ferrando-Miguel R, Cheon MS, Lubec G. Protein levels of genes encoded on chromosome 21 in fetal Down syndrome brain (Part V): overexpression of phosphatidyl-inositol-glycan class P protein (DSCR5). Amino Acids. 2004; 26:255-61.; Shibuya K, Kudoh J, Minoshima S, Kawasaki K, Asakawa S, Shimizu N. Isolation of two novel genes, DSCR5 and DSCR6, from Down syndrome critical region on human chromosome 21q22.2. Biochem Biophys Res Commun2000; 271:693-8.; Graybiel AM. The basal ganglia: learning new tricks and loving it. Curr Opin Neurobiol. 2005; 15:638-44.; Packard MG, Knowlton BJ. Learning and memory functions of the Basal Ganglia. Annu Rev Neurosci. 2002; 25:563-93.; Dierssen M, Herault Y, Estivill X. Aneuploidy: from a physiological mechanism of variance to Down syndrome. Physiol Rev. 2009;89(3):887-920.; Osada T, Adachi Y, Kimura HM, Miyashita Y. Towards understanding of the cortical network underlying associative memory. Philos Trans R Soc Lond B Biol Sci. 2008;363:2187-99.; Potier MC, Rivals I, Mercier G, Ettwiller L, Moldrich RX, Laffaire J, et al. Transcriptional disruptions in Down syndrome: a case study in the Ts1Cje mouse cerebellum during post-natal development. J Neurochen. 2006; 97 Suppl 1:104-9.; Lee S, Lee E, Lee KH, Lee D. Predicting disease phenotypes based on the molecular networks with condition-responsive correlation. Int J Data Min Bioinform.2011; 5:131-42.; Miller JA, Horvath S, Geschwind DH. Divergence of human and mouse brain transcriptome highlights Alzheimer disease pathways. Proc Natl Acad Sci USA.2010; 107:12698-703.; O’Doherty A, Ruf S, Mulligan C, Hildreth V, Errington ML, Cooke S, et al. An aneuploid mouse strain carrying human chromosome 21 with Down syndrome phenotypes. Science. 2005;309:2033-7.; Morice E, Andreae LC, Cooke SF, Vanes L, Fisher EM, Tybulewicz VL, et al. Preservation of long-term memory and synaptic plasticity despite short-term impairments in the Tc1 mouse model of Down syndrome. Learn Mem.2008; 15(7): 492-500.; Belichenko PV, Kleschevnikov AM, Masliah E, Wu C, Takimoto-Kimura R, Salehi A, et al. Excitatory-inhibitory relationship in the fascia dentata in the Ts65Dn mouse model of Down syndrome. J Comp Neurol.2009; 512(4): 453-66.; Korbel JO, Tirosh-Wagner T, Urban AE, Chen XN, Kasowski M, Dai L, et al. The genetic architecture of Down syndrome phenotypes revealed by high-resolution analysis of human segmental trisomies. Proc Natl Acad Sci USA.2009; 106: 12031-6.; Prandini P, Deutsch S, Lyle R, Gagnebin M, Delucinge Vivier C, Delorenzi M, Natural gene-expression variation in Down syndrome modulates the outcome of gene-dosage imbalance. Am J Hum Genet. 2007; 81:252-63.; Yahya-GraisonAït E, Aubert J, Dauphinot L, Rivals I, Prieur M, Golfier G, et al. Classification of human chromosome 21 gene-expression variations in Down syndrome: impact on disease phenotypes. Am J Hum Genet. 2007; 81(3):475-91.; Antonarakis SE, Lyle R, Dermitzakis ET, Reymond A, Deutsch S. Chromosome 21 and Down syndrome: from genomics to pathophysiology. Nat Rev Genet 2004; 5:725-38.; Wiseman FK, Alford KA, Tybulewicz VL, Fisher EM. Down syndrome--recent progress and future prospects. Hum Mol Genet. 2009;18(R1):R75-83.; Vilardell M, Rasche A, Thormann A, Maschke-Dutz E, Pérez-Jurado LA, Lehrach H, et al. Meta-analysis of heterogeneous Down Syndrome data reveals consistent genome-wide dosage effects related to neurological processes. BMC Genomics. 2011;12:229.; https://revistas.unimilitar.edu.co/index.php/rmed/article/view/1197
-
20Academic Journal
المؤلفون: Peña, Ángela, Yosa, Juvenal, Cuesta-Astroz, Yesid, Acevedo, Orlando, Lareo, Leonardo, García-Vallejo, Felipe
المصدر: Universitas Scientiarum; Vol. 17 No. 1 (2012); 5-15 ; Universitas Scientiarum; Vol. 17 Núm. 1 (2012); 5-15 ; Universitas Scientiarum; v. 17 n. 1 (2012); 5-15 ; 2027-1352 ; 0122-7483
مصطلحات موضوعية: 5 -Dicaffeoylquinic Acid, Human T-Lymphotropic Type I (HTLV-1), Integrase (IN), Homology Model, Molecular Docking, Binding Free Energy, Mg2 Ions
Time: 3
وصف الملف: application/pdf; text/html
Relation: https://revistas.javeriana.edu.co/index.php/scientarium/article/view/2477/1756; https://revistas.javeriana.edu.co/index.php/scientarium/article/view/2477/html; https://revistas.javeriana.edu.co/index.php/scientarium/article/view/2477