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1Academic Journal
المؤلفون: Zachar, Rikke, Al-Mashhadi, Ammar, Dimke, Henrik, Svenningsen, Per, Jensen, Boye L, Carlström, Mattias
المصدر: Zachar , R , Al-Mashhadi , A , Dimke , H , Svenningsen , P , Jensen , B L & Carlström , M 2018 , ' Hydronephrosis is associated with elevated plasmin in urine in pediatric patients and rats and changes in NCC and γ-ENaC abundance in rat kidney ' , American Journal of Physiology: Renal Physiology , vol. 315 , no. 3 , pp. F547-F557 . https://doi.org/10.1152/ajprenal.00635.2017
مصطلحات موضوعية: ENaC, Hydronephrosis, NCC, Plasmin, Up-Regulation, Humans, Male, Renin-Angiotensin System/drug effects, Case-Control Studies, Kidney/drug effects, Antihypertensive Agents/pharmacology, Hydronephrosis/etiology, Solute Carrier Family 12, Member 3/metabolism, Albuminuria/etiology, Captopril/pharmacology, Disease Models, Animal, Hypertension/drug therapy, Ureteral Obstruction/complications, Sodium/urine, Epithelial Sodium Channels/metabolism, Angiotensin-Converting Enzyme Inhibitors/pharmacology, Rats, Sprague-Dawley, Fibrinolysin/urine, Animals, Blood Pressure/drug effects
وصف الملف: application/pdf
الاتاحة: https://portal.findresearcher.sdu.dk/da/publications/b917d6de-2844-4478-8043-a97ac89a21c2
https://doi.org/10.1152/ajprenal.00635.2017
https://findresearcher.sdu.dk/ws/files/142969339/Hydronephrosis_is_associated_with_elevated_plasmin_in_urine_in_pediatric_patients_and_rats_and_changes_in_NCC_and_ENaC_abundance_in_rat_kidney.pdf -
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المؤلفون: René Frydensbjerg Andersen, Boye L. Jensen, Ulla G. Friis, Kristian B Buhl, Bente Jespersen, Per Svenningsen, Søren Rittig
المصدر: Andersen, R F, Buhl, K B, Jensen, B L, Svenningsen, P, Friis, U G, Jespersen, B & Rittig, S 2013, ' Remission of nephrotic syndrome diminishes urinary plasmin content and abolishes activation of ENaC ', Pediatric nephrology (Berlin, Germany), vol. 28, no. 8, pp. 1227-34 . https://doi.org/10.1007/s00467-013-2439-2
Andersen, M R, Buhl, K B, Jensen, B L, Svenningsen, P, Friis, U G, Jespersen, B & Rittig, S 2013, ' Remission of nephrotic syndrome diminishes urinary plasmin content and abolishes activation of ENaC ', Pediatric Nephrology, vol. 28, no. 8, pp. 1227-1234 . https://doi.org/10.1007/s00467-013-2439-2مصطلحات موضوعية: Male, Epithelial sodium channel, Nephrotic Syndrome, Patch-Clamp Techniques, Sodium retention, Plasmin, Denmark, Urine, Aldosterone/urine, Membrane Potentials, chemistry.chemical_compound, Creatinine/urine, Edema, Fibrinolysin, Lymphocytes, Longitudinal Studies, Child, Aldosterone, Fibrinolysin/urine, Remission Induction, Lymphocytes/metabolism, Amiloride, Nephrology, Creatinine, Child, Preschool, Female, Kidney Tubules, Collecting/metabolism, Immunosuppressive Agents, medicine.drug, medicine.medical_specialty, Adolescent, Urinary system, ENaC, Blotting, Western, Nephrotic Syndrome/blood, Down-Regulation, Enzyme-Linked Immunosorbent Assay, Plasminogen/urine, Cell Line, Internal medicine, Epithelial Sodium Channel Blockers, medicine, Humans, Kidney Tubules, Collecting, Epithelial Sodium Channels, business.industry, Plasminogen, medicine.disease, Endocrinology, chemistry, Pediatrics, Perinatology and Child Health, Immunosuppressive Agents/therapeutic use, Epithelial Sodium Channels/drug effects, business, Epithelial Sodium Channel Blockers/pharmacology, Nephrotic syndrome, Biomarkers, Biomarkers/blood