المؤلفون: Sven Unger, Markus Christmann, Alicia Poplawski, Christian Schwarzenbach, H. Irem Baymaz, Juliana Brandstetter Vilar, Petra Beli, Maja T. Tomicic, Thomas R. Reich, Teodora Nikolova, Fabian Mühlhäusler

المصدر: Cellular and Molecular Life Sciences

مصطلحات موضوعية: DNA Repair, Survivin, 610 Medizin, RAD51, Apoptosis, Mice, 0302 clinical medicine, 610 Medical sciences, Tumor Cells, Cultured, Homologous Recombination, Cellular Senescence, 0303 health sciences, Gene knockdown, biology, Brain Neoplasms, Chemistry, BIRC5, Gene Expression Regulation, Neoplastic, 030220 oncology & carcinogenesis, Molecular Medicine, Female, Original Article, Alkylation damage, Senescence, Programmed cell death, Mice, Nude, Clastogenic effects, 03 medical and health sciences, Cellular and Molecular Neuroscience, Glioma, Biomarkers, Tumor, Temozolomide, medicine, Animals, Humans, Nuclear export signal, Antineoplastic Agents, Alkylating, Molecular Biology, Cell Proliferation, 030304 developmental biology, Cell Nucleus, Pharmacology, Inhibitor of apoptosis (IAP), Cell Biology, medicine.disease, Xenograft Model Antitumor Assays, Proliferating cell nuclear antigen, Drug Resistance, Neoplasm, biology.protein, Cancer research, Glioblastoma, DNA Damage

URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::96464994e00e47ed62c4ec8e026514a8
https://doi.org/10.1007/s00018-021-03864-0