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1Academic Journal
المؤلفون: Yeola, Asmita Pradeep, Akbar, Irshad, Baillargeon, Joanie, Doss, Prenitha Mercy Ignatius Arokia, Paavilainen, Ville O., Rangachari, Manu
المساهمون: Institute of Biotechnology, University Management
مصطلحات موضوعية: Th1, Th17, Protein translocation, ERAD, Sec61, P97, Experimental autoimmune encephalomyelitis, Eeyarestatin-1 (ESI), Apratoxin A (ApraA), NMS-873, TH17 CELLS, DEGRADATION, APRATOXIN, PATHWAY, DIFFERENTIATION, INHIBITOR, RESPONSES, BLOCKADE, STRESS, T(H)17, Biochemistry, cell and molecular biology
وصف الملف: application/pdf
Relation: We acknowledge Alexandre Brunet and Stephanie Fiola for technical assistance, Aline Dumas for critical discussions, and Lisa Kate Radden for assistance with figure preparation. The work was supported by a Discovery Grant from the Natural Sciences Engineering Research Council of Canada (NSERC), to M.R. V.O.P is supported by the Academy of Finland (grants 289737 and 314672), US National Institutes of Health (Project 1R01GM132649-01) and the Sigrid Juselius Foundation. M.R. is a Junior-2 scholar of the Fonds de recherche de Quebec -Sante (FRQS).; Yeola , A P , Akbar , I , Baillargeon , J , Doss , P M I A , Paavilainen , V O & Rangachari , M 2020 , ' Protein translocation and retro-translocation across the endoplasmic reticulum are crucial to inflammatory effector CD4(+) T cell function ' , Cytokine , vol. 129 , 154944 . https://doi.org/10.1016/j.cyto.2019.154944; http://hdl.handle.net/10138/327538; 9ef440a5-375f-437b-ab1c-5d434447937b; 000527732100014
الاتاحة: http://hdl.handle.net/10138/327538
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المؤلفون: Joanie Baillargeon, Prenitha Mercy Ignatius Arokia Doss, Irshad Akbar, Asmita Pradeep Yeola, Manu Rangachari, Ville O. Paavilainen
المساهمون: Institute of Biotechnology, University Management
المصدر: Cytokine. 129:154944
مصطلحات موضوعية: Eeyarestatin-1 (ESI), 0301 basic medicine, Sec61, STRESS, BLOCKADE, T cell, Immunology, Biochemistry, Proinflammatory cytokine, Th1, PATHWAY, 03 medical and health sciences, 0302 clinical medicine, Interferon, NMS-873, medicine, Immunology and Allergy, Secretion, Molecular Biology, Protein translocation, P97, Experimental autoimmune encephalomyelitis, Effector, Chemistry, Endoplasmic reticulum, T(H)17, INHIBITOR, Hematology, ERAD, DEGRADATION, Cell biology, Apratoxin A (ApraA), DIFFERENTIATION, 030104 developmental biology, medicine.anatomical_structure, 030220 oncology & carcinogenesis, 1182 Biochemistry, cell and molecular biology, Tumor necrosis factor alpha, Th17, TH17 CELLS, APRATOXIN, RESPONSES, medicine.drug