يعرض 1 - 20 نتائج من 401 نتيجة بحث عن '"Buratowski, Stephen"', وقت الاستعلام: 0.58s تنقيح النتائج
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    Academic Journal

    المصدر: Pérez Ortín, José Enrique Rodríguez Navarro, Susana Nuño Cabanes, Carme Pérez Martínez, María E. Soares, Luis M. Oliete Calvo, Paula Serrano Quílez, Joan Buratowski, Stephen Dichtl, Bernhard 2018 A role for Mog1 in H2Bub1 and H3K4me3 regulation affecting RNAPII transcription and mRNA export Embo Reports 19 e 45992 1 17

    مصطلحات موضوعية: medicina, biotecnologia

    وصف الملف: application/pdf

    Relation: Embo Reports, 2018, vol. 19, num. e 45992, p. 1-17; Oliete‐Calvo, P., Serrano‐Quílez, J., Nuño‐Cabanes, C., Pérez‐Martínez, M. E., Soares, L. M., Dichtl, B., Buratowski, S., Pérez‐Ortín, J. E., & Rodríguez‐Navarro, S. (2018). A role for Mog1 in H2Bub1 and H3K4me3 regulation affecting RNAPII transcription and mRNA export. En EMBO reports (Vol. 19, Issue 11). Springer Science and Business Media LLC. https://doi.org/10.15252/embr.201845992; https://hdl.handle.net/10550/94630; 128107

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    Academic Journal

    المصدر: Proceedings of the National Academy of Sciences of the United States of America, 2020 Dec . 117(51), 32348-32357.

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    Periodical
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    News
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    Academic Journal

    المساهمون: Department of Biochemistry and Molecular Pharmacology

    المصدر: PLoS genetics ; 8 ; 7 ; e1002811

    Relation: Link to Article in PubMed; Radman-Livaja M, Quan TK, Valenzuela L, Armstrong JA, van Welsem T, et al. (2012) A Key Role for Chd1 in Histone H3 Dynamics at the 3′ Ends of Long Genes in Yeast. PLoS Genet 8(7): e1002811. doi:10.1371/journal.pgen.1002811. Link to article on publisher's site; 1553-7390 (Linking); http://hdl.handle.net/20.500.14038/39537; https://escholarship.umassmed.edu/cgi/viewcontent.cgi?article=3331&context=oapubs&unstamped=1; https://escholarship.umassmed.edu/oapubs/2331; 3299916; oapubs/2331

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    Academic Journal

    المساهمون: Department of Biochemistry and Molecular Pharmacology, Hsiuyi V. Chen

    المصدر: PLoS biology ; 10 ; 7 ; e1001369

    Relation: Link to article in PubMed; PLoS Biol. 2012;10(7):e1001369. doi:10.1371/journal.pbio.1001369. Epub 2012 Jul 31. Link to article on publisher's website; http://hdl.handle.net/20.500.14038/33458; https://escholarship.umassmed.edu/cgi/viewcontent.cgi?article=3009&context=gsbs_sp&unstamped=1; https://escholarship.umassmed.edu/gsbs_sp/1988; 8635816; gsbs_sp/1988

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    Academic Journal
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    Academic Journal
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    Academic Journal
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    Academic Journal
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  12. 12
    Academic Journal

    المصدر: Proceedings of the National Academy of Sciences of the United States of America, 1999 Oct . 96(22), 12542-12547.

  13. 13
    Academic Journal
  14. 14
    Academic Journal

    المساهمون: Harvard Medical School Boston (HMS), Centre de Recherche en Cancérologie de Marseille (CRCM), Aix Marseille Université (AMU)-Institut Paoli-Calmettes (IPC), Fédération nationale des Centres de lutte contre le Cancer (FNCLCC)-Fédération nationale des Centres de lutte contre le Cancer (FNCLCC)-Institut National de la Santé et de la Recherche Médicale (INSERM)-Centre National de la Recherche Scientifique (CNRS), Korea Advanced Institute of Science and Technology (KAIST), Foghorn Therapeutics Cambridge, MA, États-Unis, Institut de génétique humaine (IGH), Université de Montpellier (UM)-Centre National de la Recherche Scientifique (CNRS), H.B. was supported by a postdoctoral fellowship from the Basic Science Research Program of the National Research Foundation of Korea (NRF), funded by the Ministry of Education (2015R1A6A3A03017730). This work was supported by grants from the National Research Foundation of Korea (NRF-2019R1A2C2090830) to J.K., from “Ligue Nationale Contre le Cancer” (LNCC) (Equipe labellisée) to M.D. and V.G., and grants GM046498 and GM056663 from the U.S. National Institutes of Health to S.B.

    المصدر: ISSN: 2041-1723.

    Relation: info:eu-repo/semantics/altIdentifier/pmid/32358498; inserm-02617416; https://inserm.hal.science/inserm-02617416; https://inserm.hal.science/inserm-02617416/document; https://inserm.hal.science/inserm-02617416/file/s41467-020-16082-2.pdf; PUBMED: 32358498; PUBMEDCENTRAL: PMC7195483

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    Academic Journal

    المصدر: Proceedings of the National Academy of Sciences of the United States of America, 1996 Dec 01. 93(25), 14654-14658.

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    Academic Journal
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    Academic Journal

    المساهمون: Massachusetts Institute of Technology. Department of Biology, Massachusetts Institute of Technology. Department of Biological Engineering, Massachusetts Institute of Technology. Department of Mechanical Engineering, Koch Institute for Integrative Cancer Research at MIT

    المصدر: Elsevier

    وصف الملف: application/pdf

    Relation: http://dx.doi.org/10.1016/j.cell.2019.01.018; Cell; https://hdl.handle.net/1721.1/125970; Neurohr, Gabriel E. et al. "Excessive Cell Growth Causes Cytoplasm Dilution And Contributes to Senescence." Cell 175, 5 (February 2019): P1083-1097.e18 © 2019 The Author(s)

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    Academic Journal
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    Academic Journal
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    Academic Journal