المؤلفون: Foltman, Magdalena, Méndez Guzmán, Iván, Bech-Serra, Joan J., Torre, Carolina de la, Brace, Jennifer L., Weiss, Eric L., Lucas Gay, María, Queralt, Ethel, Sánchez Díaz, Alberto

المساهمون: Universidad de Cantabria

المصدر: PLoS biology, 2023, 21(8), e3002263

Relation: https://doi.org/10.1371/journal.pbio.3002263; https://hdl.handle.net/10902/29928

الاتاحة: https://hdl.handle.net/10902/29928
https://doi.org/10.1371/journal.pbio.3002263

المؤلفون: Foltman, Magdalena, Méndez, Iván, Bech-Serra, Joan J., de la Torre, Carolina, Brace, Jennifer L., Weiss, Eric L., Lucas, María, Queralt, Ethel, Sánchez-Díaz, Alberto

المساهمون: Queralt, Ethel, Consejo Superior de Investigaciones Científicas https://ror.org/02gfc7t72

مصطلحات موضوعية: Underlying molecular mechanisms, Signalling pathway plays, Cells following cytokinesis, Anaphase remain unknown, Cell cycle machinery, Since sec3 activates, Reproduces torc1 control, Promote membrane fusion, 2 tor complexes, Exocyst component sec3, Control cell separation, Cell separation, Snare component, %22">Xlink ">, Turn controls, Torc1 alters, Snare complex, Nuclear dbf2, Kinase family, Key substrate, Key role, First time, Data indicate, Cellular growth, Budding yeast

Relation: Publisher's version; Foltman M.; Mendez I.; Bech-Serra J.J.; de la Torre C.; Brace J.L.; Weiss E.L.; Lucas M.; Queralt E.; Sanchez-Diaz A. (2023). TOR complex 1 negatively regulates NDR kinase Cbk1 to control cell separation in budding yeast. PLoS Biol 21(8): e3002263. https//doi.org/10.1371/journal.pbio.3002263. http://hdl.handle.net/10261/334646; https://doi.org/10.1371/journal.pbio.3002263.s001; Sí; Foltman, Magdalena; Méndez, Iván; Bech-Serra, Joan J.; de la Torre, Carolina; Brace, Jennifer L.; Weiss, Eric L.; Lucas, María; Queralt, Ethel; Sánchez-Díaz, Alberto; 2023; cdc15-2 cells complete mitosis and cytokinesis after anaphase block and release [Dataset]; Figshare; https://doi.org/10.1371/journal.pbio.3002263.s001; http://hdl.handle.net/10261/360758

الاتاحة: http://hdl.handle.net/10261/360758
https://doi.org/10.1371/journal.pbio.3002263.s001

المؤلفون: Ortiz-Barahona, Vanessa, Soler, Marta, Davalos, Veronica, García-Prieto, Carlos A., Janin, Maxime, Setien, Fernando, Fernández-Rebollo, Irene, Bech-Serra, Joan J., De La Torre, Carolina, Guil, Sonia, Villanueva, Alberto, Zhang, Pei-Hong, Yang, Li, Guarnacci, Marco, Schumann, Ulrike, Preiss, Thomas, Balaseviciute, Ugne, Montal, Robert, Llovet, Josep M., Esteller, Manel

المساهمون: Generalitat de Catalunya, MCIN/AIE

المصدر: Molecular Cancer ; volume 22, issue 1 ; ISSN 1476-4598

الاتاحة: http://dx.doi.org/10.1186/s12943-023-01785-z
https://link.springer.com/content/pdf/10.1186/s12943-023-01785-z.pdf
https://link.springer.com/article/10.1186/s12943-023-01785-z/fulltext.html

المؤلفون: Matas-Nadal, Clara, Bech-Serra, Joan J., Gatius, Sònia, Gomez, Xavier, Ribes-Santolaria, Marina, Guasch-Vallés, Marta, Pedraza, Neus, Casanova, Josep M., de la Torre Gómez, Carolina, Garí, Eloi, Aguayo-Ortiz, Rafael S.

المصدر: Molecular & Cellular Proteomics ; volume 22, issue 6, page 100547 ; ISSN 1535-9476

الاتاحة: http://dx.doi.org/10.1016/j.mcpro.2023.100547
https://api.elsevier.com/content/article/PII:S1535947623000579?httpAccept=text/xml
https://api.elsevier.com/content/article/PII:S1535947623000579?httpAccept=text/plain

المؤلفون: Diesch, Jeannine, Pannérer, Marguerite Marie Le, Winkler, René, Casquero, Raquel, Muhar, Matthias, Van Der Garde, Mark, Maher, Michael, Martínez Herráez, Carolina, Bech Serra, Joan J., Fellner, Michaela, Rathert, Philipp, Brooks, Nigel, Zamora, Lurdes, Gentilella, Antonio, Torre, Carolina de la, Zuber, Johannes, Götze, Katharina S., Buschbeck, Marcus

المصدر: Articles publicats en revistes (Institut d'lnvestigació Biomèdica de Bellvitge (IDIBELL))

مصطلحات موضوعية: Hematopoesi, Terapèutica, Hematopoiesis, Therapeutics

وصف الملف: 13 p.; application/pdf

Relation: Reproducció del document publicat a: https://doi.org/10.1038/s41467-021-26258-z; Nature Communications, 2021, vol. 12, num. 1; https://doi.org/10.1038/s41467-021-26258-z; http://hdl.handle.net/2445/181016

الاتاحة: http://hdl.handle.net/2445/181016

المؤلفون: Ministerio de Ciencia e Innovación (España), Gobierno de Cantabria, Generalitat Valenciana, Queralt, Ethel [0000-0003-0045-0039], Sánchez-Díaz, Alberto [0000-0002-3308-4824], Foltman, Magdalena, Méndez Guzmán, Iván, Bech-Serra, Joan J., Torre, Carolina de la, Brace, Jennifer L., Weiss, Eric L., Lucas, María, Queralt, Ethel, Sánchez-Díaz, Alberto

مصطلحات الفهرس: artículo

URL: http://hdl.handle.net/10261/334646
Publisher's version
https://dx.doi.org/10.1371/journal.pbio.3002263
Sí
info:eu-repo/grantAgreement/AEI/Plan Estatal de Investigación Científica y Técnica y de Innovación 2017-2020/PID2019-106745GB-I00/ES/ESTUDIO DE LA FUNCION MOLECULAR DEL COMPLEJO TOR 1 EN LA DIVISION CELULAR EUCARIOTA
info:eu-repo/grantAgreement/AEI/Plan Estatal de Investigación Científica y Técnica y de Innovación 2017-2020/PID2019-109027GB-I00/ES/ESTUDIO DE LAS FUNCIONES CANONICAS Y NO CANONICAS DE PROTEINAS REGULADORAS DE LA MITOSIS
info:eu-repo/grantAgreement/AEI/Plan Estatal de Investigación Científica y Técnica y de Innovación 2017-2020/RTI2018-097801-B-I00/ES/UN NUEVO MECANISMO DE SELECCION Y RECICLADO DE PROTEINAS INTEGRALES DE MEMBRANA EN ENDOSOMAS

المؤلفون: Queralt, Ethel [0000-0003-0045-0039], Sánchez-Díaz, Alberto [0000-0002-3308-4824], Foltman, Magdalena [0000-0002-2827-2679], Lucas, María [0000-0002-7854-4249], Foltman, Magdalena, Sánchez-Díaz, Alberto, Méndez, Iván, Bech-Serra, Joan J., de la Torre, Carolina, Brace, Jennifer L., Weiss, Eric L., Lucas, María, Queralt, Ethel

مصطلحات الفهرس: Underlying molecular mechanisms, Signalling pathway plays, Cells following cytokinesis, Anaphase remain unknown, Cell cycle machinery, Since sec3 activates, Reproduces torc1 control, Promote membrane fusion, 2 tor complexes, Exocyst component sec3, Control cell separation, Cell separation, Snare component, Turn controls, Torc1 alters, Snare complex, Nuclear dbf2, Kinase family, Key substrate, Key role, First time, Data indicate, Cellular growth, Budding yeast, dataset

URL: http://hdl.handle.net/10261/361053
Publisher's version
Foltman M.; Mendez I.; Bech-Serra J.J.; de la Torre C.; Brace J.L.; Weiss E.L.; Lucas M.; Queralt E.; Sanchez-Diaz A. (2023). TOR complex 1 negatively regulates NDR kinase Cbk1 to control cell separation in budding yeast. PLoS Biol 21(8): e3002263. https//doi.org/10.1371/journal.pbio.3002263. http://hdl.handle.net/10261/334646
https://dx.doi.org/10.1371/journal.pbio.3002263.s005
Sí

المؤلفون: Queralt, Ethel [0000-0003-0045-0039], Sánchez-Díaz, Alberto [0000-0002-3308-4824], Foltman, Magdalena [0000-0002-2827-2679], Lucas, María [0000-0002-7854-4249], Foltman, Magdalena, Sánchez-Díaz, Alberto, Méndez, Iván, Bech-Serra, Joan J., de la Torre, Carolina, Brace, Jennifer L., Weiss, Eric L., Lucas, María, Queralt, Ethel

مصطلحات الفهرس: Underlying molecular mechanisms, Signalling pathway plays, Cells following cytokinesis, Anaphase remain unknown, Cell cycle machinery, Since sec3 activates, Reproduces torc1 control, Promote membrane fusion, 2 tor complexes, Exocyst component sec3, Control cell separation, Cell separation, Snare component, Turn controls, Torc1 alters, Snare complex, Nuclear dbf2, Kinase family, Key substrate, Key role, First time, Data indicate, Cellular growth, Budding yeast, dataset

URL: http://hdl.handle.net/10261/360960
Publisher's version
Foltman M.; Mendez I.; Bech-Serra J.J.; de la Torre C.; Brace J.L.; Weiss E.L.; Lucas M.; Queralt E.; Sanchez-Diaz A. (2023). TOR complex 1 negatively regulates NDR kinase Cbk1 to control cell separation in budding yeast. PLoS Biol 21(8): e3002263. https//doi.org/10.1371/journal.pbio.3002263. http://hdl.handle.net/10261/334646
https://dx.doi.org/10.1371/journal.pbio.3002263.s004
Sí

المؤلفون: Queralt, Ethel [0000-0003-0045-0039], Sánchez-Díaz, Alberto [0000-0002-3308-4824], Foltman, Magdalena [0000-0002-2827-2679], Lucas, María [0000-0002-7854-4249], Foltman, Magdalena, Sánchez-Díaz, Alberto, Consejo Superior de Investigaciones Científicas [https://ror.org/02gfc7t72], Méndez, Iván, Bech-Serra, Joan J., de la Torre, Carolina, Brace, Jennifer L., Weiss, Eric L., Lucas, María, Queralt, Ethel

مصطلحات الفهرس: Underlying molecular mechanisms, Signalling pathway plays, Cells following cytokinesis, Anaphase remain unknown, Cell cycle machinery, Since sec3 activates, Reproduces torc1 control, Promote membrane fusion, 2 tor complexes, Exocyst component sec3, Control cell separation, Cell separation, Snare component, Turn controls, Torc1 alters, Snare complex, Nuclear dbf2, Kinase family, Key substrate, Key role, First time, Data indicate, Cellular growth, Budding yeast, dataset

URL: http://hdl.handle.net/10261/360848
Publisher's version
Foltman M.; Mendez I.; Bech-Serra J.J.; de la Torre C.; Brace J.L.; Weiss E.L.; Lucas M.; Queralt E.; Sanchez-Diaz A. (2023). TOR complex 1 negatively regulates NDR kinase Cbk1 to control cell separation in budding yeast. PLoS Biol 21(8): e3002263. https//doi.org/10.1371/journal.pbio.3002263. http://hdl.handle.net/10261/334646
https://dx.doi.org/10.1371/journal.pbio.3002263.s002
Sí

المؤلفون: Janin, Maxime, Ortiz‑Barahona, Vanessa, Esteller, Manel, Castro de Moura, Manuel, Carrato, Cristina, Martínez Cardús, Anna, Llinàs-Arias, Pere, Soler, Marta, Nachmani, Daphna, Pelletier, Joffrey, Schumann, Ulrike, Calleja Cervantes, Maria E., Moran, Sebastian, Guil, Sonia, Bueno Costa, Alberto, Piñeyro, David, Pérez Salvia, Montserrat, Rosselló-Tortella, Margalida, Piqué, Laia, Bech-Serra, Joan J., Torre Gómez, Carolina de la, Vidal-Bel, August, Martínez Iniesta, María, Martín-Tejera, Juan F., Villanueva Garatachea, Alberto, Arias, Alexandra, Cuartas, Isabel, Aransay, Ana M., Morales La Madrid, Andrés, Carcaboso, Ángel M., Santa-Maria Lopez, Vicente, Mora Graupera, Jaume, Fernández, Agustín F., Fraga, Mario F., Aldecoa Ansórregui, Iban, Pedrosa, Leire, Graus Ribas, Francesc, Martínez Soler, Fina, Tortosa i Moreno, Avelina

المصدر: Articles publicats en revistes (Infermeria Fonamental i Clínica)

مصطلحات موضوعية: Avaluació de resultats (Assistència mèdica), Glioma, RNA, Metilació, Outcome assessment (Medical care), Gliomas, Methylation

وصف الملف: 22 p.; application/pdf

Relation: Reproducció del document publicat a: https://doi.org/10.1007/s00401-019-02062-4; Acta Neuropathologica, 2019, vol. 138, num. 6, p. 1053-1074; https://doi.org/10.1007/s00401-019-02062-4; info:eu-repo/grantAgreement/EC/FP7/268626/EU//EPINORC; http://hdl.handle.net/2445/148583; 691920

الاتاحة: http://hdl.handle.net/2445/148583

المؤلفون: Janin, Maxime, Ortiz-Barahona, Vanessa, de Moura, Manuel Castro, Martínez-Cardús, Anna, Llinàs-Arias, Pere, Soler, Marta, Nachmani, Daphna, Pelletier, Joffrey, Schumann, Ulrike, Calleja-Cervantes, Maria E., Moran, Sebastian, Guil, Sonia, Bueno-Costa, Alberto, Piñeyro, David, Perez-Salvia, Montserrat, Rosselló-Tortella, Margalida, Piqué, Laia, Bech-Serra, Joan J., De La Torre, Carolina, Vidal, August, Martínez-Iniesta, María, Martín-Tejera, Juan F., Villanueva, Alberto, Arias, Alexandra, Cuartas, Isabel, Aransay, Ana M., La Madrid, Andres Morales, Carcaboso, Angel M., Santa-Maria, Vicente, Mora, Jaume, Fernandez, Agustin F., Fraga, Mario F., Aldecoa, Iban, Pedrosa, Leire, Graus, Francesc, Vidal, Noemi, Martínez-Soler, Fina, Tortosa, Avelina, Carrato, Cristina, Balañá, Carme, Boudreau, Matthew W., Hergenrother, Paul J., Kötter, Peter, Entian, Karl-Dieter, Hench, Jürgen, Frank, Stephan, Mansouri, Sheila, Zadeh, Gelareh, Dans, Pablo D., Orozco, Modesto

المساهمون: Ministerio de Economía y Competitividad

المصدر: Acta Neuropathologica ; volume 138, issue 6, page 1053-1074 ; ISSN 0001-6322 1432-0533

الاتاحة: http://dx.doi.org/10.1007/s00401-019-02062-4
http://link.springer.com/content/pdf/10.1007/s00401-019-02062-4.pdf
http://link.springer.com/article/10.1007/s00401-019-02062-4/fulltext.html

المؤلفون: Foltman, Magdalena, Méndez, Iván, Bech-Serra, Joan J., de la Torre, Carolina, Brace, Jennifer L., Weiss, Eric L., Lucas, María, Queralt, Ethel, Sánchez-Díaz, Alberto

المساهمون: Queralt, Ethel, Sánchez-Díaz, Alberto, Foltman, Magdalena

مصطلحات موضوعية: Underlying molecular mechanisms, Signalling pathway plays, Cells following cytokinesis, Anaphase remain unknown, Cell cycle machinery, Since sec3 activates, Reproduces torc1 control, Promote membrane fusion, 2 tor complexes, Exocyst component sec3, Control cell separation, Cell separation, Snare component, Turn controls, Torc1 alters, Snare complex, Nuclear dbf2, Kinase family, Key substrate, Key role, First time, Data indicate, Cellular growth, Budding yeast, kinases, cell cycle

Relation: Publisher's version; Foltman M.; Mendez I.; Bech-Serra J.J.; de la Torre C.; Brace J.L.; Weiss E.L.; Lucas M.; Queralt E.; Sanchez-Diaz A. (2023). TOR complex 1 negatively regulates NDR kinase Cbk1 to control cell separation in budding yeast. PLoS Biol 21(8): e3002263. https//doi.org/10.1371/journal.pbio.3002263. http://hdl.handle.net/10261/334646; Sí; Foltman, Magdalena; Méndez, Iván; Bech-Serra, Joan J.; de la Torre, Carolina; Brace, Jennifer L.; Weiss, Eric L.; Lucas, María; Queralt, Ethel; Sánchez-Díaz, Alberto; 2023; Proposed model for how TORC1 might regulate Cbk1 activity in cdc15-2 cells [Dataset]; Figshare; https://doi.org/10.1371/journal.pbio.3002263.s010; http://hdl.handle.net/10261/363201

الاتاحة: http://hdl.handle.net/10261/363201
https://doi.org/10.1371/journal.pbio.3002263.s010

المؤلفون: Foltman, Magdalena, Méndez, Iván, Bech-Serra, Joan J., de la Torre, Carolina, Brace, Jennifer L., Weiss, Eric L., Lucas, María, Queralt, Ethel, Sánchez-Díaz, Alberto

المساهمون: Queralt, Ethel, Sánchez-Díaz, Alberto, Foltman, Magdalena, Lucas, María

مصطلحات موضوعية: Underlying molecular mechanisms, Signalling pathway plays, Cells following cytokinesis, Anaphase remain unknown, Cell cycle machinery, Since sec3 activates, Reproduces torc1 control, Promote membrane fusion, 2 tor complexes, Exocyst component sec3, Control cell separation, Cell separation, Snare component, Turn controls, Torc1 alters, Snare complex, Nuclear dbf2, Kinase family, Key substrate, Key role, First time, Data indicate, Cellular growth, Budding yeast, kinases, cell cycle

Relation: Publisher's version; Foltman M.; Mendez I.; Bech-Serra J.J.; de la Torre C.; Brace J.L.; Weiss E.L.; Lucas M.; Queralt E.; Sanchez-Diaz A. (2023). TOR complex 1 negatively regulates NDR kinase Cbk1 to control cell separation in budding yeast. PLoS Biol 21(8): e3002263. https//doi.org/10.1371/journal.pbio.3002263. http://hdl.handle.net/10261/334646; https://dx.doi.org/10.1371/journal.pbio.3002263.s009; Sí; Foltman, Magdalena; Méndez, Iván; Bech-Serra, Joan J.; de la Torre, Carolina; Brace, Jennifer L.; Weiss, Eric L.; Lucas, María; Queralt, Ethel; Sánchez-Díaz, Alberto; 2023; Sec3 contains conserved NDR/LATS phosphorylation consensus sites [Dataset]; Figshare; https://doi.org/10.1371/journal.pbio.3002263.s009; http://hdl.handle.net/10261/362601

الاتاحة: http://hdl.handle.net/10261/362601
https://doi.org/10.1371/journal.pbio.3002263.s009

المؤلفون: Foltman, Magdalena, Méndez, Iván, Bech-Serra, Joan J., de la Torre, Carolina, Brace, Jennifer L., Weiss, Eric L., Lucas, María, Queralt, Ethel, Sánchez-Díaz, Alberto

المساهمون: Queralt, Ethel, Sánchez-Díaz, Alberto, Foltman, Magdalena, Lucas, María

مصطلحات موضوعية: Underlying molecular mechanisms, Signalling pathway plays, Cells following cytokinesis, Anaphase remain unknown, Cell cycle machinery, Since sec3 activates, Reproduces torc1 control, Promote membrane fusion, 2 tor complexes, Exocyst component sec3, Control cell separation, Cell separation, Snare component, Turn controls, Torc1 alters, Snare complex, Nuclear dbf2, Kinase family, Key substrate, Key role, First time, Data indicate, Cellular growth, Budding yeast, kinases, cell cycle

Relation: Publisher's version; Foltman M.; Mendez I.; Bech-Serra J.J.; de la Torre C.; Brace J.L.; Weiss E.L.; Lucas M.; Queralt E.; Sanchez-Diaz A. (2023). TOR complex 1 negatively regulates NDR kinase Cbk1 to control cell separation in budding yeast. PLoS Biol 21(8): e3002263. https//doi.org/10.1371/journal.pbio.3002263. http://hdl.handle.net/10261/334646; https://doi.org/10.1371/journal.pbio.3002263.s008; Sí; Foltman, Magdalena; Méndez, Iván; Bech-Serra, Joan J.; de la Torre, Carolina; Brace, Jennifer L.; Weiss, Eric L.; Lucas, María; Queralt, Ethel; Sánchez-Díaz, Alberto; 2023; Most of known Cbk1 interactions were maintained in all 3 conditions, independently of TORC1 activity [Dataset]; Figshare; https://doi.org/10.1371/journal.pbio.3002263.s008; http://hdl.handle.net/10261/362574

الاتاحة: http://hdl.handle.net/10261/362574
https://doi.org/10.1371/journal.pbio.3002263.s008

المؤلفون: Foltman, Magdalena, Méndez, Iván, Bech-Serra, Joan J., de la Torre, Carolina, Brace, Jennifer L., Weiss, Eric L., Lucas, María, Queralt, Ethel, Sánchez-Díaz, Alberto

المساهمون: Queralt, Ethel, Sánchez-Díaz, Alberto, Foltman, Magdalena, Lucas, María

مصطلحات موضوعية: Underlying molecular mechanisms, Signalling pathway plays, Cells following cytokinesis, Anaphase remain unknown, Cell cycle machinery, Since sec3 activates, Reproduces torc1 control, Promote membrane fusion, 2 tor complexes, Exocyst component sec3, Control cell separation, Cell separation, Snare component, Turn controls, Torc1 alters, Snare complex, Nuclear dbf2, Kinase family, Key substrate, Key role, First time, Data indicate, Cellular growth, Budding yeast, kinases, cell cycle

Relation: Publisher's version; Foltman M.; Mendez I.; Bech-Serra J.J.; de la Torre C.; Brace J.L.; Weiss E.L.; Lucas M.; Queralt E.; Sanchez-Diaz A. (2023). TOR complex 1 negatively regulates NDR kinase Cbk1 to control cell separation in budding yeast. PLoS Biol 21(8): e3002263. https//doi.org/10.1371/journal.pbio.3002263. http://hdl.handle.net/10261/334646; https://doi.org/10.1371/journal.pbio.3002263.s007; Sí; Foltman, Magdalena; Méndez, Iván; Bech-Serra, Joan J.; de la Torre, Carolina; Brace, Jennifer L.; Weiss, Eric L.; Lucas, María; Queralt, Ethel; Sánchez-Díaz, Alberto; 2023; Chitinase Cts1 is unable to be localised at the site of division in cdc14-1 cells [Dataset]; Figshare; https://doi.org/10.1371/journal.pbio.3002263.s007; http://hdl.handle.net/10261/362427

الاتاحة: http://hdl.handle.net/10261/362427
https://doi.org/10.1371/journal.pbio.3002263.s007

المؤلفون: Foltman, Magdalena, Méndez, Iván, Bech-Serra, Joan J., de la Torre, Carolina, Brace, Jennifer L., Weiss, Eric L., Lucas, María, Queralt, Ethel, Sánchez-Díaz, Alberto

المساهمون: Queralt, Ethel, Sánchez-Díaz, Alberto, Foltman, Magdalena, Lucas, María

مصطلحات موضوعية: Underlying molecular mechanisms, Signalling pathway plays, Cells following cytokinesis, Anaphase remain unknown, Cell cycle machinery, Since sec3 activates, Reproduces torc1 control, Promote membrane fusion, 2 tor complexes, Exocyst component sec3, Control cell separation, Cell separation, Snare component, Turn controls, Torc1 alters, Snare complex, Nuclear dbf2, Kinase family, Key substrate, Key role, First time, Data indicate, Cellular growth, Budding yeast, kinases, cell cycle

Relation: Publisher's version; Foltman M.; Mendez I.; Bech-Serra J.J.; de la Torre C.; Brace J.L.; Weiss E.L.; Lucas M.; Queralt E.; Sanchez-Diaz A. (2023). TOR complex 1 negatively regulates NDR kinase Cbk1 to control cell separation in budding yeast. PLoS Biol 21(8): e3002263. https//doi.org/10.1371/journal.pbio.3002263. http://hdl.handle.net/10261/334646; https://doi.org/10.1371/journal.pbio.3002263.s006; Sí; Foltman, Magdalena; Méndez, Iván; Bech-Serra, Joan J.; de la Torre, Carolina; Brace, Jennifer L.; Weiss, Eric L.; Lucas, María; Queralt, Ethel; Sánchez-Díaz, Alberto; 2023; Analysis of CBK1 mutants in cbk1-deleted cells [Dataset]; Figshare; https://doi.org/10.1371/journal.pbio.3002263.s006; http://hdl.handle.net/10261/362411

الاتاحة: http://hdl.handle.net/10261/362411
https://doi.org/10.1371/journal.pbio.3002263.s006

المؤلفون: Foltman, Magdalena, Méndez, Iván, Bech-Serra, Joan J., de la Torre, Carolina, Brace, Jennifer L., Weiss, Eric L., Lucas, María, Queralt, Ethel, Sánchez-Díaz, Alberto

المساهمون: Queralt, Ethel, Sánchez-Díaz, Alberto, Foltman, Magdalena, Lucas, María

مصطلحات موضوعية: Underlying molecular mechanisms, Signalling pathway plays, Cells following cytokinesis, Anaphase remain unknown, Cell cycle machinery, Since sec3 activates, Reproduces torc1 control, Promote membrane fusion, 2 tor complexes, Exocyst component sec3, Control cell separation, Cell separation, Snare component, Turn controls, Torc1 alters, Snare complex, Nuclear dbf2, Kinase family, Key substrate, Key role, First time, Data indicate, Cellular growth, Budding yeast, kinases, cell cycle

Relation: Publisher's version; Foltman M.; Mendez I.; Bech-Serra J.J.; de la Torre C.; Brace J.L.; Weiss E.L.; Lucas M.; Queralt E.; Sanchez-Diaz A. (2023). TOR complex 1 negatively regulates NDR kinase Cbk1 to control cell separation in budding yeast. PLoS Biol 21(8): e3002263. https//doi.org/10.1371/journal.pbio.3002263. http://hdl.handle.net/10261/334646; https://dx.doi.org/10.1371/journal.pbio.3002263.s005; Sí; Foltman, Magdalena; Méndez, Iván; Bech-Serra, Joan J.; de la Torre, Carolina; Brace, Jennifer L.; Weiss, Eric L.; Lucas, María; Queralt, Ethel; Sánchez-Díaz, Alberto; 2023; Analysis of the importance of each serine or threonine followed by proline in Cbk1-6E [Dataset]; Figshare; https://doi.org/10.1371/journal.pbio.3002263.s005; http://hdl.handle.net/10261/361053

الاتاحة: http://hdl.handle.net/10261/361053
https://doi.org/10.1371/journal.pbio.3002263.s005

المؤلفون: Foltman, Magdalena, Méndez, Iván, Bech-Serra, Joan J., de la Torre, Carolina, Brace, Jennifer L., Weiss, Eric L., Lucas, María, Queralt, Ethel, Sánchez-Díaz, Alberto

المساهمون: Queralt, Ethel, Sánchez-Díaz, Alberto, Foltman, Magdalena, Lucas, María

مصطلحات موضوعية: Underlying molecular mechanisms, Signalling pathway plays, Cells following cytokinesis, Anaphase remain unknown, Cell cycle machinery, Since sec3 activates, Reproduces torc1 control, Promote membrane fusion, 2 tor complexes, Exocyst component sec3, Control cell separation, Cell separation, Snare component, Turn controls, Torc1 alters, Snare complex, Nuclear dbf2, Kinase family, Key substrate, Key role, First time, Data indicate, Cellular growth, Budding yeast, Kinases, cell cycle

Relation: Publisher's version; Foltman M.; Mendez I.; Bech-Serra J.J.; de la Torre C.; Brace J.L.; Weiss E.L.; Lucas M.; Queralt E.; Sanchez-Diaz A. (2023). TOR complex 1 negatively regulates NDR kinase Cbk1 to control cell separation in budding yeast. PLoS Biol 21(8): e3002263. https//doi.org/10.1371/journal.pbio.3002263. http://hdl.handle.net/10261/334646; https://dx.doi.org/10.1371/journal.pbio.3002263.s004; Sí; Foltman, Magdalena; Méndez, Iván; Bech-Serra, Joan J.; de la Torre, Carolina; Brace, Jennifer L.; Weiss, Eric L.; Lucas, María; Queralt, Ethel; Sánchez-Díaz, Alberto; 2023; Conserved phosphorylation sites in Cbk1 [Dataset]; Figshare; https://doi.org/10.1371/journal.pbio.3002263.s004; http://hdl.handle.net/10261/360960

الاتاحة: http://hdl.handle.net/10261/360960
https://doi.org/10.1371/journal.pbio.3002263.s004

المؤلفون: Foltman, Magdalena, Méndez, Iván, Bech-Serra, Joan J., de la Torre, Carolina, Brace, Jennifer L., Weiss, Eric L., Lucas, María, Queralt, Ethel, Sánchez-Díaz, Alberto

المساهمون: Queralt, Ethel, Sánchez-Díaz, Alberto, Foltman, Magdalena, Lucas, María

مصطلحات موضوعية: Underlying molecular mechanisms, Signalling pathway plays, Cells following cytokinesis, Anaphase remain unknown, Cell cycle machinery, Since sec3 activates, Reproduces torc1 control, Promote membrane fusion, 2 tor complexes, Exocyst component sec3, Control cell separation, Cell separation, Snare component, Turn controls, Torc1 alters, Snare complex, Nuclear dbf2, Kinase family, Key substrate, Key role, First time, Data indicate, Cellular growth, Budding yeast, Kinases, cell cycle

Relation: Publisher's version; Foltman M.; Mendez I.; Bech-Serra J.J.; de la Torre C.; Brace J.L.; Weiss E.L.; Lucas M.; Queralt E.; Sanchez-Diaz A. (2023). TOR complex 1 negatively regulates NDR kinase Cbk1 to control cell separation in budding yeast. PLoS Biol 21(8): e3002263. https//doi.org/10.1371/journal.pbio.3002263. http://hdl.handle.net/10261/334646; https://dx.doi.org/10.1371/journal.pbio.3002263.s003; Sí; Foltman, Magdalena; Méndez, Iván; Bech-Serra, Joan J.; de la Torre, Carolina; Brace, Jennifer L.; Weiss, Eric L.; Lucas, María; Queralt, Ethel; Sánchez-Díaz, Alberto; 2023; Function of known Cbk1 regulators is independent of TORC1 [Dataset]; Figshare; https://doi.org/10.1371/journal.pbio.3002263.s003; http://hdl.handle.net/10261/360882

الاتاحة: http://hdl.handle.net/10261/360882
https://doi.org/10.1371/journal.pbio.3002263.s003

المؤلفون: Foltman, Magdalena, Méndez, Iván, Bech-Serra, Joan J., de la Torre, Carolina, Brace, Jennifer L., Weiss, Eric L., Lucas, María, Queralt, Ethel, Sánchez-Díaz, Alberto

المساهمون: Queralt, Ethel, Sánchez-Díaz, Alberto, Foltman, Magdalena, Lucas, María, Consejo Superior de Investigaciones Científicas https://ror.org/02gfc7t72

مصطلحات موضوعية: Underlying molecular mechanisms, Signalling pathway plays, Cells following cytokinesis, Anaphase remain unknown, Cell cycle machinery, Since sec3 activates, Reproduces torc1 control, Promote membrane fusion, 2 tor complexes, Exocyst component sec3, Control cell separation, Cell separation, Snare component, Turn controls, Torc1 alters, Snare complex, Nuclear dbf2, Kinase family, Key substrate, Key role, First time, Data indicate, Cellular growth, Budding yeast, Kinases, cell cycle

Relation: Publisher's version; Foltman M.; Mendez I.; Bech-Serra J.J.; de la Torre C.; Brace J.L.; Weiss E.L.; Lucas M.; Queralt E.; Sanchez-Diaz A. (2023). TOR complex 1 negatively regulates NDR kinase Cbk1 to control cell separation in budding yeast. PLoS Biol 21(8): e3002263. https//doi.org/10.1371/journal.pbio.3002263. http://hdl.handle.net/10261/334646; https://dx.doi.org/10.1371/journal.pbio.3002263.s002; Sí; Foltman, Magdalena; Méndez, Iván; Bech-Serra, Joan J.; de la Torre, Carolina; Brace, Jennifer L.; Weiss, Eric L.; Lucas, María; Queralt, Ethel; Sánchez-Díaz, Alberto; 2023; Depletion of Cbk1 prevents cdc15-2 cell-separation rescue after TORC1 inactivation [Dataset]; Figshare; https://doi.org/10.1371/journal.pbio.3002263.s002; http://hdl.handle.net/10261/360848

الاتاحة: http://hdl.handle.net/10261/360848
https://doi.org/10.1371/journal.pbio.3002263.s002