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المؤلفون: Jiri Mayer, Hans Carl Hasselbalch, Zdenek Racil, Lijuan Han, Angela Maurer, Anders Lindholm Sørensen, Deniz Gezer, Claudia Schubert, Julia Czech, Ivan G. Costa, Sabrina Cordua, Tim H. Brümmendorf, Barbora Weinbergerova, Nicolas Chatain, Morten Orebo Holmström, Lasse Kjær, Trine Alma Knudsen, Bernd Denecke, Thomas Stauffer Larsen, Vibe Skov, Julian Baumeister, Steffen Koschmieder, Kristina Feldberg, Blanka Kubešová, Tiago Maié, Gerhard Müller-Newen, Assja Crepcia
المصدر: Czech, J, Cordua, S, Weinbergerova, B, Baumeister, J, Crepcia, A, Han, L, Maié, T, Costa, I G, Denecke, B, Maurer, A, Schubert, C, Feldberg, K, Gezer, D, Brümmendorf, T H, Müller-Newen, G, Mayer, J, Racil, Z, Kubesova, B, Knudsen, T, Sørensen, A L, Holmström, M, Kjær, L, Skov, V, Larsen, T S, Hasselbalch, H C, Chatain, N & Koschmieder, S 2019, ' JAK2V617F but not CALR mutations confer increased molecular responses to interferon-α via JAK1/STAT1 activation ', Leukemia, vol. 33, no. 4, pp. 995-1010 . https://doi.org/10.1038/s41375-018-0295-6
مصطلحات موضوعية: Adult, Male, 0301 basic medicine, Cancer Research, CD34, Alpha interferon, Apoptosis, Antiviral Agents, Peripheral blood mononuclear cell, Mice, 03 medical and health sciences, 0302 clinical medicine, Downregulation and upregulation, Pegylated interferon, Biomarkers, Tumor, Tumor Cells, Cultured, medicine, Animals, Humans, Aged, Cell Proliferation, Retrospective Studies, Myeloproliferative Disorders, biology, business.industry, Interferon-alpha, Janus Kinase 1, Hematology, Janus Kinase 2, Middle Aged, Prognosis, Haematopoiesis, STAT1 Transcription Factor, 030104 developmental biology, Oncology, 030220 oncology & carcinogenesis, Mutation, biology.protein, Cancer research, Female, Stem cell, Calreticulin, business, Follow-Up Studies, medicine.drug